Ramon Hernandez

Desarrollador de videojuegos en desarrollo (también hagos videos de deportes de motor)

[Ramon Hernandez @Ramonidous]

#IsleofManTT is back this week 🇮🇲, the best motorsport event in the world.
My first video about it has reached 800k views, check it out, you might become a fan of the sport! 🏍️

[Ramon Hernandez @Ramonidous]

Besides working on my game I've found time to make a cool video about the Isle of Man TT 🇮🇲 #IsleofManTT
https://t.co/V2K5rFKqcy

[Ramon Hernandez @Ramonidous]

@snpoehlm 😰

[Harry Spoelstra @HarrySpoelstra]

Beyond brain fog: viral proteins as convergent drivers of neuroinflammation and proteinopathy

🚨“COVID-19 never really leaves your brain.”

New science review proposes SARSCoV2 viral proteins stay behind as long-lived toxins, triggering chronic neuroinflammation and planting the seeds of Alzheimer’s and Parkinson’s, even after mild infection.

This very interesting and eye-catching GERMAN review reframes post-viral neurological syndromes( L0ngC0vid) as driven by persistent viral proteins acting as long-term toxins ("protein-as-pathogen" model), not just the active infection!

➡️Core mechanisms:
- SARSCoV2 Spike and OTHER viral proteins activate glial TLR4/TLR2 receptors, triggering chronic neuroinflammatory cascades via NLRP3 inflammasome,
- They also disrupt autophagy, allowing toxic protein aggregates (tau, amyloid-beta, α-synuclein) to accumulate and seed neurodegeneration,

➡️SARSCoV2 specific evidence:
- Animal studies show Spike protein alone (without live virus) induces TLR4-mediated cognitive deficits, memory impairment, synaptic loss, and sustained neuroinflammation, recapitulating post-COVID syndrome,
- Spike binds α-synuclein, accelerating Parkinson-like clumps,

➡️Human data evidence:
- Millions experience "brain fog,"
- Post-COVID patients exhibit measurable brain damage: cortical thinning, hippocampal iron accumulation, and biomarkers of ongoing neuronal injury,

➡️Broader risks:
- Even mild infections leave lingering proteins that promote Alzheimer’s and Parkinson’s-like pathology via shared pathways,
- Same pathways seen in influenza, dengue, West Nile etc,
- Mild infection = no protection,

‼️So, according to this review, the “protein-as-pathogen” model makes it crystal clear: every new SARSCoV2 infection (even mild or asymptomatic) deposits more of these long-lived toxic viral proteins into the brain. They don’t fully clear. They accumulate.

Each reinfection reloads the TLR4/TLR2 → NLRP3 inflammasome trigger and further collapses autophagy, speeding up the tau/amyloid/α-synuclein proteinopathy and neurodegeneration.

SARS-CoV-2 does not just infect.

It weaponizes its own proteins as long-lived intracellular saboteurs.

Millions are probably already carrying this hidden payload.

This is not brain fog.

This is a silent, population-scale reprogramming of human brains toward dementia-like decline.

The long-term neurological cost will probably dwarf the acute pandemic itself!
#AvoidSars2 #AvoidReinfections
https://t.co/x0oxacaNwl

[Ramon Hernandez @Ramonidous]

@ttracesofficial Dean would probably have won it but declaring a winner with just one lap completed seems excessive.

[Ramon Hernandez @Ramonidous]

@bmarci7

[Graham @Tachyon100]

Maria is paralysed from the chest down after her sidecar crash at the TT.
Please support this GoFundMe by donating or sharing – every bit of help makes a difference. https://t.co/Chd3kSAcZx

[Manuel Ruiz @manruipa]

⚠️‼️ I came across this news in Spain, and this discourse keeps doing enormous harm.

The post literally says:
“A study from Hospital del Mar reveals a worrying connection between childhood trauma and physical diseases in adult life. From headaches to diabetes, adverse childhood experiences could leave marks on our health. Early prevention has never been so crucial.”
“A study from Hospital del Mar details numerous physical diseases linked to childhood trauma.”

Not because they literally said that most organic diseases are caused by childhood trauma, but because in practice, many of the diseases they attribute to or reduce to trauma end up being organic diseases whose biomarkers have not yet been discovered, or whose proper medical workup was never even done.

And that is the real problem.

For years, many patients are first filtered through the lens of trauma, psychosomatic illness, or somatization, when in reality what they have is an organic disease that has simply not yet been diagnosed. I would even go further and say that this also happens with diseases we now understand well: before reaching the right specialist and before the appropriate tests are performed, many patients are treated as if the origin of their symptoms were psychological or trauma-related, simply because nobody has investigated their case properly.

That is not precision medicine.
That is a way of covering diagnostic ignorance with a psychological narrative.

The history of medicine shows this again and again: many diseases that in their early stages were treated as “nervous,” “hysterical,” or “psychosomatic” later turned out to be well-defined organic diseases. This happened with multiple autoimmune, neurological, and inflammatory diseases. And it is still happening today.

That is why this discourse is so dangerous. Because it not only delays the real diagnosis, but also blames the patient, isolates them, sends them into the wrong clinical pathways, and steals years of serious medical investigation from them.

Most of these patients do not need their illness to be explained through trauma.
They need the right tests, the right specialist, and serious investigation of their real biology instead of convenient theories being projected onto them.

Enough of turning the absence of known biomarkers into a psychological explanation.
What some people today call trauma or somatization too often ends up tomorrow having a name, a mechanism, and a biological marker.

[Ramon Hernandez @Ramonidous]

@7316 I believe that secretly they want us to hate EVs, so they design the ugliest cars they can think of.

[Ramon Hernandez @Ramonidous]

@wil88_lee Neither do I, hope they don't end up like MotoGP

[Ramon Hernandez @Ramonidous]

@RoadRacingEmp I love your videos, some of the best content about the TT online for those of us who cannot afford to watch it live 👍

[Ramon Hernandez @Ramonidous]

@clemmygames It will be great to see all the Steam sales games with zero hours played in 3D 💩

[Ramon Hernandez @Ramonidous]

@XorDev Yeah, the trigonometric functions must have a period which is a multiple of a common value, exponential/logarithmic values have to settle to a value... I actually did it for your shader and drew progress bars for each cycle to debug, but it was tricky.

[Ramon Hernandez @Ramonidous]

@AntonioRamosGP Tenía potencial para pelear por el título, además habría contado con el apoyo incondicional de las marcas japonesas. Una pena, me recuerda a Stefan Bellof en la F1, un talento fugaz que no tuvo tiempo para brillar.

[Ramon Hernandez @Ramonidous]

@gamefromscratch How does a game engine "comply with European rules and guidelines" ?

[Ramon Hernandez @Ramonidous]

@ttracesofficial Sad but expected, get well soon Davey, I'm sure we'll see you on the top spot of the podium next year.

[Ramon Hernandez @Ramonidous]

@FantasyGalaxies We need audiobooks with Jar Jar's voice, I'm sure there's a market for it.

[Ramon Hernandez @Ramonidous]

@GreyAlien Congrats!

[Ramon Hernandez @Ramonidous]

@TheVertlartnic I'm waiting for the "mildly dead" memes at this point.

[Ramon Hernandez @Ramonidous]

@TheMSHub Riding and skateboarding at 62 😎

[Minty Comedic Arts @MintComedicArts]

Me at The Met Gala...