New research published in Nature Communications has found that aging may damage mitochondria partly by reducing an important membrane lipid called phosphatidylcholine.
As this lipid declines, mitochondria become fragmented and less effective at producing energy. Researchers found that restoring phosphatidylcholine—or supplying choline, its precursor—helped stabilize mitochondrial networks and improve their ability to handle stress in aging worms and human cells.
The discovery could open new possibilities for treating age-related metabolic and neurological problems, although choline has not been proven to reverse aging in humans.
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