Abdinoor Senior Issack

Allah first ||A proud nomad🐪||Tea connoisseur☕||Books & sports||Gp🩺||OneManArmy|| La_TX

[Ezekiel @ezekiel_aleke]

Dear Data Analyst!!

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[Abdinoor Senior Issack @Issack7251]

@Manu_Sisti Guide

[Abdinoor Senior Issack @Issack7251]

Congratulations CITY.

Arsenal ain't winning anything anytime soon.
#ArtetaOUT

[Abdinoor Senior Issack @Issack7251]

@ezekiel_aleke DATA

[Abdinoor Senior Issack @Issack7251]

@KDanielkamande @Dr_AustinOmondi And "The frustrated MO" must be saying the bitter truth? What is the end game of the BSC Cos?
Where will they be counted!

If at All there is away of being made away with it, it should be. Those guys need to be rescued

[Abdinoor Senior Issack @Issack7251]

@Shahriar661731 Claude

[Mr Mark Gockelen @Shahriar661731]

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[Abdinoor Senior Issack @Issack7251]

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[Abdinoor Senior Issack @Issack7251]

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[Abdinoor Senior Issack @Issack7251]

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[Pushpendra Tripathi @pushpendratips]

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[Dr. Jasmeet Kaur (PhD) @jasmeet481]

Constipation is not just a gut issue. It’s a detox failure.

Stool isn’t only undigested food.
It carries used hormones, excess cholesterol, bile, toxins, and metabolic waste.

The liver processes waste.
The gut disposes it.

If bowel movements are sluggish:
• Toxins get reabsorbed
• Hormones (like estrogen) recycle back
• Cholesterol returns to the liver
• The liver is forced to detox the same waste again

This creates a toxic loop and definitely will lead to fatigue, hormonal imbalance, brain fog, skin issues, poor metabolism.

No daily bowel movement = detox not completed.

Constipation isn’t just about discomfort or bloating.
It’s about systemic stagnation.

Flow matters.

[Abdinoor Senior Issack @Issack7251]

@tech_crafters DATA

[William A. Wallace, Ph.D. @WilliamWallace]

Iodine, selenium, and tyrosine are non-negotiable for thyroid function.

• Iodine + tyrosine → build T4
• Selenium-dependent deiodinases → convert T4 → active T3
Without selenium, thyroid hormone activation stalls.

Daily targets (with biological sufficiency in mind):
Iodine ~150 mcg | Selenium ~55–100 mcg | Tyrosine ~500–1,000 mg

Balance matters. more ≠ better.

PMID: 18686295

[CME INDIA @CMEINDIA1]

SGPT (ALT) in Liver Function Tests: Clinical Significance

Serum glutamic-pyruvic transaminase (SGPT), now referred to as alanine aminotransferase (ALT), is a predominantly cytosolic enzyme localized within hepatocytes and serves as the most specific biochemical marker of hepatocellular injury among routine liver function tests.

ALT elevations reflect disruption of hepatocyte membrane integrity and correlate with active hepatocellular necrosis or inflammation rather than cholestasis.

Key Clinical Interpretations

• Mild elevation (≤2–3 × ULN)
Common in non-alcoholic fatty liver disease (MASLD), metabolic dysfunction, early viral hepatitis, drug-induced liver injury (DILI), hypothyroidism, and post-exercise states.

• Moderate elevation (3–10 × ULN)
Suggests acute viral hepatitis, autoimmune hepatitis, ischemic hepatitis (early), or significant DILI.

• Marked elevation (>10–20 × ULN)
Highly suggestive of acute viral hepatitis, ischemic (shock) liver, acetaminophen toxicity, severe DILI, or acute autoimmune hepatitis.

ALT vs AST (De Ritis Ratio)

• ALT > AST → Typical of MASLD / viral hepatitis
• AST > ALT (ratio >2) → Alcohol-associated liver disease
• Very high AST with modest ALT → Ischemic or muscle-related injury

Important Clinical Pearls

• ALT is more liver-specific than AST, as AST is also present in muscle, heart, and kidney.
• “Normal” ALT values may still be abnormal — current evidence supports lower upper limits (≈30 U/L in men, ≈19 U/L in women).
• Persistently elevated ALT in diabetes and obesity predicts progression to MASH and fibrosis.
• ALT may normalize in advanced cirrhosis despite worsening liver disease — do not equate normal ALT with benign liver status.
• ALT is the preferred marker to monitor treatment response in viral hepatitis and DILI.

Interpretation in the Present LFT Context

Reported SGPT (ALT): 29.5 U/L
This value lies within normal range, suggesting no active hepatocellular necrosis at present.

However, concomitant findings of:

Elevated bilirubin (Total & Indirect 4.7 mg/dL)

Raised AST (51.6 U/L)

suggest a pattern more consistent with predominantly unconjugated hyperbilirubinaemia with mild hepatocellular stress or non-hepatic causes (e.g., hemolysis, Gilbert’s syndrome, resolving hepatitis) rather than active ALT-dominant liver injury.

Take-Home Message

ALT is the most sensitive and specific routine marker of hepatocellular injury, but normal ALT does not exclude significant liver disease. Clinical context, AST/ALT ratio, bilirubin fractions, and fibrosis assessment are essential for meaningful interpretation.

[Abdinoor Senior Issack @Issack7251]

@ezekiel_aleke DATA

[Abdinoor Senior Issack @Issack7251]

@AndrewBolis Send

[Abdinoor Senior Issack @Issack7251]

@boye4christ2006 I haven’t registered yet but I would love to join the free class