Albert

[Albert @Albertdffg]

@SamaHoole That’s not a metabolic timeline; it’s carnivore fan fiction with timestamps. If every improvement proves the diet works and every problem means “wait longer and add butter,” you haven’t created a hypothesis—you’ve created a belief system that can’t fail.

[Albert @Albertdffg]

@NothingIsArt2 @NothingIsArt Disheartening 😢

[Albert @Albertdffg]

@NothingIsArt2 @NothingIsArt Just wow 🤯

[Albert @Albertdffg]

@PantelisKyrku @SamaHoole Try taking a nap 😴

[Albert @Albertdffg]

@LucasPremat @SamaHoole That’s a much more reasonable criticism—but it’s also a retreat from “medicine has exactly one move: addition.” If subtraction is routinely advised, then the original claim was simply false.

[Albert @Albertdffg]

@spotling @SamaHoole @grok Does that shed any light on it for ya?

[Albert @Albertdffg]

@ChrisHa12959414 @SamaHoole “You’re missing the point” isn’t a rebuttal. I addressed the point—you just don’t have an answer to it, Chris

[Albert @Albertdffg]

@SamaHoole There’s no solid epidemiological evidence that vegetable oil consumption explains the European melanoma gradient, nor does it account for the extreme rates in high-UV fair-skinned populations elsewhere. Phenotype and UV exposure pattern remain the dominant, well-supported drivers

[Albert @Albertdffg]

@SamaHoole Intralipid infusions do shut down insulin signaling via DAG/PKC-θ. That’s real.

High-dose IV oil ≠ normal dietary intake. Traditional high-carb societies without seed oils show starch isn’t the villain by itself — but they don’t prove seed oils are the main driver either.

[Albert @Albertdffg]

@SamaHoole That retreat doesn’t work. You didn’t just report observations—you supplied mechanisms: ketones spare protein, raw milk explains tolerance, fat maxxing explains leanness, and declining linoleic acid explains not burning. Once you explain why, those claims require evidence

[Albert @Albertdffg]

@PaulKendall1313 @SamaHoole “Some people read one post” is just a dodge.
Raw milk doesn’t restore lactase.
Energy balance still rules.
Sodium is essential.
Seed oils don’t cause sunburn.
Cite the data that overturns those, or stop pretending “do more reading” is an argument.

[Albert @Albertdffg]

@cancerclasses @SamaHoole The claim that dietary linoleic acid promotes atherosclerosis in people is not established by human outcome evidence and is contradicted by most of it. If you have a specific trial, meta-analysis, or biomarker study that you think settles the question the other way, cite it.

[Albert @Albertdffg]

@SamaHoole Absence of Paleolithic salt mines does not equal no sodium need

Pre-ag humans got it from blood/organs of whole prey, not pure muscle. Low insulin increases sodium loss.

Many on carnivore need more salt, not less. Your zero-salt success is valid for you—not a universal rule

[Albert @Albertdffg]

@SamaHoole A string of dates isn’t a mechanism. Heart disease predates Crisco, and randomized trials and modern evidence don’t show linoleic acid causes coronary disease. A carefully arranged timeline can make correlation look very dramatic; it still doesn’t make it causation.

[Albert @Albertdffg]

@LiveAncestral Humans synthesize creatine from glycine, arginine and methionine; dietary creatine is not classified as an essential nutrient.

Vegetarians generally do have lower muscle creatine stores. But studies do not consistently show that unsupplemented vegetarians are weaker

[Albert @Albertdffg]

@davesmith2468 @Ian_J_Tickle @ValerieAnne1970 You’re still quietly changing “transient troponin elevation” into “serious heart problems years later.” The study never demonstrated that. Calling a hypothetical outcome “off the scale” doesn’t turn it into evidence.

[Albert @Albertdffg]

@WhatMattersN0W @SamaHoole ApoB particles normally cross the endothelium; they don’t need a damaged artery to get in.

The problem is retention—they get in, but don’t all get back out. More circulating ApoB means more opportunities to get trapped.