Want to feel more confident during ENT history taking and physical exams?
Join us for a hands-on ENT workshop covering the basics every medical student and intern should know!
With Dr. Talal AlHarbi (R2 ENT Resident – KAMC-MNGHA)
📅 29 January
⏰ 1:00–3:00 PM
📍 Simulation Center, KSAU-HS
⚠️ Limited seats available, don't miss out!
https://t.co/sNpat1HJL8
الشؤون الصحية بوزارة الحرس الوطني تعلن عن نجاح إجراء أول عمليتين لترميم عيوب صيوان الأذن الخَلقية (Microtia)، إيذانًا بإطلاق برنامج ترميم عيوب صيوان الأذن الخَلقية باستخدام الغرسات الطبية، وذلك ضمن جهودها المستمرة لتقديم أحدث أنواع الجراحة الطبية المتقدمة.
ويهدف البرنامج إلى إتاحة هذا النوع المتطور من التدخلات الجراحية، والذي يتميّز بإمكانية إجرائه عبر عملية جراحية واحدة فقط، مع تمكين الأطفال دون سن السادسة من الاستفادة من هذا الإجراء، وفق تقييم طبي دقيق ومعايير إكلينيكية معتمدة.
#MNGHA
Emerging from RT/CRT based on pathology to ctDNA-guided risk stratification, considering de-escalation if -ve, and intensifying therapy if +ve, for which adjuvant therapy may be molecularly personalized as histology alone proven to be insufficient, which is an incredible finding!
As traditional paradigm defines surgical success by negative margins, pathological stage (TNM), and absence of adverse features reflecting definitive biological cure if anatomical clearance has been proven. However, recurrence still occurs despite these predictive factors!
Surprisingly, early stage (I–II) with ctDNA +ve has worse prognosis than advanced stage ctDNA -ve. Indicating that surgery removed local disease, yet systemic disease remains biologically active explaining the unexpected recurrences
They reduce tau phosphorylation by GSK-3 beta, they’re both antioxidants and reduce cytokines + microglial activation to reduce inflammation, and induce neurogenesis by supporting plasticity and BDNF (brain-derived neurotrophic factor), hoping for promising future clinical trials
Digging up more on this topic got me even with UWorld medical library as I noticed reduced melatonin levels in CSF could present way earlier than the onset of alzheimer’s symptoms
I read a study a while ago showing that higher trace lithium in drinking water was linked to lower dementia rates, suggesting lithium could help in preventing memory loss as it is known for its neuroprotective benefits in reducing suicidal risk, and protecting neurons from damage
Got me thinking of the possibility of making microdoses of lithium orotate and melatonin supplementation as a protective way to slow down the progression of Alzheimer's, mechanistically, they’re both targeting the amyloid beta accumulation by decreasing production and aggregation
🚨 Ready to expand your knowledge in ENT?
We are excited to invite you tomorrow to an exclusive and dynamic session:
👃 Inside Rhinology
Gain valuable insights into common rhinological diseases — from diagnosis to management. Perfect for sharpening your clinical expertise!
📅 Tomorrow Wednesday | 03/09/2025
⏰ 8:00 PM
👨⚕️ Speaker: Dr. Abdulaziz Alabdulkareem – R1
@abdulaziz_ma_
🔗 Join us on Teams by scanning the QR code
✅ Don’t miss this insightful session!
This is a novel finding that could change treatment for one of the most common neurological disorders alongside good sleep, a healthy diet, and physical activity, hoping one day with the reasoning of mine to suggest ideas for targeted treatments of various diseases
I read a study a while ago showing that higher trace lithium in drinking water was linked to lower dementia rates, suggesting lithium could help in preventing memory loss as it is known for its neuroprotective benefits in reducing suicidal risk, and protecting neurons from damage
It found out that neutral lithium orotate can increase free lithium levels in the brain by an experiment in rats with Alzheimer’s disease finding that physiologic (very low) doses of lithium orotate reversed dementia symptoms by 70% significantly