Daniel Plotkin, PhD

Interested in muscle growth and all things exercise.

[Daniel Plotkin, PhD @DanielPlotkin_]

@zbitter Hard to predict markets, but I’d be surprised if demand is big here. Current options are great, mix and match is tedious, you have to wait, and the end result may still be meh compared to stuff you already like.

[Mike Roberts @DrMikeRoberts]

Acute molecular and chronic adaptations to lengthened partial versus full range of motion resistance training. Awesome & hyper relevant dissertation work by @DanielPlotkin_ from @AuburnKINES with collab from @ @EXSS_UMHB. https://t.co/AZnBal6gIS

[Daniel Plotkin, PhD @DanielPlotkin_]

@dhood_york Hmm, review seems to make that conclusion with quality assessments that aren’t fair to the context; can we blind a participant to cuff condition? Given the volume of literature it’s pretty fair to say that hypertrophy is similar when looking at reasonable protocols, BFR vs Trad.

[Daniel Plotkin, PhD @DanielPlotkin_]

@BradSchoenfeld Nice, congrats! Good to see this out… there’s one particularly odd study in this area… 👀

[Daniel Plotkin, PhD @DanielPlotkin_]

@Brady_H

[Daniel Plotkin, PhD @DanielPlotkin_]

@gerdy_mac I’ll play!
1. Exercises aren’t always for one muscle so assumption all MU are recruited at ~3 RIR not true across exercises.
2. Granting reps after full MU recruitment are less hypertrophic they are still likely hypertrophic. Thus decision making would be in context of session

[Daniel Plotkin, PhD @DanielPlotkin_]

@rruetigger @AviBittMD @MattWalshBlog I think all that is being said is that you can run a trial and observe side effects over time without knowing or finding a mechanism.

[Experimental Physiology @ExpPhysiol]

@DanielPlotkin_ (@AuburnKINES) et al. look at the effects of exercise stressors and disuse, and how these impact skeletal muscle myosin heavy chain fragmentation!
📜https://t.co/edbGxEadgf

[Daniel Plotkin, PhD @DanielPlotkin_]

@DrAndyGalpin At first I thought you were underlining your disagreement by changing causal to casual 😂 would’ve been a savage move

[Daniel Plotkin, PhD @DanielPlotkin_]

@JuanSAMUDI57306 @RogerEnoka @avigotsky It’s possible but probably not given within subject design and untrained individuals who have a lot of room for type 1 growth.

[Daniel Plotkin, PhD @DanielPlotkin_]

@JuanSAMUDI57306 @RogerEnoka @avigotsky https://t.co/w6yewlvNh0 and https://t.co/eOg02pNKBk trained and untrained populations.

[Daniel Plotkin, PhD @DanielPlotkin_]

@JuanSAMUDI57306 @RogerEnoka @avigotsky For example one that comes readily to mind is why does soleus get same growth from seated and standing calf raise if there’s less contribution/growth in the gastroc in seated wouldnt we expect more from the soleus?

[Daniel Plotkin, PhD @DanielPlotkin_]

@JuanSAMUDI57306 @RogerEnoka @avigotsky Yeah to be clear I do think mechanical variables matter I just don’t like the reductionism. The extent to which each matters how they interact with hypertrophic stimulus I think will be unpacked with coming work(mine included). But there’s already head scratchers that cast doubt.

[Daniel Plotkin, PhD @DanielPlotkin_]

@JuanSAMUDI57306 @RogerEnoka @avigotsky More energy demanding would be dependent on the extent of diff. for whatever parameter right? Not sure how to ascertain that without some sort of modeling. Also do we know where a muscle would be on the length tension relationship in vivo with high force dynamic contractions?

[Daniel Plotkin, PhD @DanielPlotkin_]

@JuanSAMUDI57306 @RogerEnoka @avigotsky As you have seen there’s tons of debate about motor control and it’s far from a done deal 😂 so reducing things to one factor and then overlaying hypertrophy prediction on top I don’t think is appropriate. Particularly in light of some non-intuitive findings with hypertrophy.

[Daniel Plotkin, PhD @DanielPlotkin_]

@JuanSAMUDI57306 @RogerEnoka @avigotsky I think there’s a reason why biomechanists use optimization models and simulations because there’s a lot of factors that can be considered, muscle length(velocity of shortening), pCSA, length tension, if a muscle has multiple actions, history effects, etc.

[Daniel Plotkin, PhD @DanielPlotkin_]

@Nick_Christian2 @BradSchoenfeld Yeah it’s interesting cause it depends on the type, for example given that with training interventions of all types we see an exclusive shift away from IIx it would surprise me if there wasn’t a large genetic contribution/higher baseline amount in unique individuals.

[Daniel Plotkin, PhD @DanielPlotkin_]

@WardWriting Associated does not (always) imply causation 😎

[Daniel Plotkin, PhD @DanielPlotkin_]

@brady_h @WardWriting To my knowledge benefits were modest to non-existent in humans. I assume Shaun meant in humans 🤷🏻‍♂️