DermaPhotoGallery

Bank of dermatology images, clinical cases, results before and after, based on the best contributions from the most reputable dermatologists. Training

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Topical treatments may include keratolytics such as salicylic acid or urea, and depigmenting agents such as tretinoin or hydroquinone. In severe cases, physical procedures such as dermabrasion or laser may be considered, although effectiveness is variable and often temporary.

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ACANTOSIS NIGRICANS. VI. 1/4
Tratamiento: Se centra en abordar las comorbilidades asociadas. La pérdida de peso y el control glucémico mediante modificaciones en el estilo de vida y farmacoterapia, como metformina, son esenciales.

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ACANTHOSIS NIGRICANS. VI. 4/4
Treatment: Focuses on addressing associated comorbidities. Weight loss and glycemic control through lifestyle modifications and pharmacotherapy, such as metformin, are essential.

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ACANTHOSIS NIGRICANS BENIGN. V. 2/2
Given its association with metabolic disorders, it is essential to perform a comprehensive evaluation of the patient to detect and manage underlying conditions such as insulin resistance, dyslipidemias, and possible neoplasias.

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ACANTOSIS NIGRICANS BENIGNA. V. 1/2
Dada su asociación con trastornos metabólicos, es fundamental realizar una evaluación integral del paciente para detectar y manejar condiciones subyacentes como la resistencia a la insulina, dislipidemias y posibles neoplasias.

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ACANTHOSIS NIGRICANS BENIGN. IV. 2/2
Hyperinsulinemia plays a crucial role by stimulating these receptors, exacerbating epidermal hyperplasia.
The diagnosis is clinical, based on the observation of distinctive skin characteristics and their anatomical distribution.

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ACANTOSIS NIGRICANS BENIGNA. IV. 1/2
La hiperinsulinemia juega un papel crucial al estimular estos receptores, exacerbando la hiperplasia epidérmica.
El diagnóstico es clínico, basado en la observación de las características cutáneas distintivas y su distribución anatómica.

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ACANTHOSIS NIGRICANS BENIGN. III. 2/2.
The pathogenesis involves the activation of epidermal growth factor receptors (EGFR) and insulin receptor, resulting in the proliferation of keratinocytes and fibroblasts.

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ACANTOSIS NIGRICANS BENIGNA. III. 1/2.
La patogénesis implica la activación de los receptores del factor de crecimiento epidérmico (EGFR) y del receptor de insulina, lo que resulta en la proliferación de queratinocitos y fibroblastos.

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ACANTHOSIS NIGRICANS BENIGN. II. 2/2
Strong association with endocrine conditions such as type 2 diabetes mellitus and polycystic ovary syndrome. Histologically characterized by hyperkeratosis, acanthosis and papillomatosis, without evidence of significant inflammatory infiltrate

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ACANTOSIS NIGRICANS BENIGNA. II. 1/2
Fuerte asociación con condiciones endocrinas como diabetes mellitus tipo 2 y síndrome de ovario poliquístico. Histológica/ caracterizada por hiperqueratosis, acantosis y papilomatosis, sin evidencia de infiltrados inflamatorios significativos.

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ACANTHOSIS NIGRICANS BENIGN. I. 2/2
Hyperpigmentation and thickening of the skin, particularly in fold areas such as the neck, armpits, inguinal region and other flexural areas. Greater prevalence in individuals with obesity and insulin resistance.

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ACANTOSIS NIGRICANS BENIGNA. I. 1/2
Hiperpigmentación y engrosamiento de la piel, particularmente en áreas de pliegues como el cuello, las axilas, la región inguinal y otras áreas flexurales. Mayorprevalencia en individuos con obesidad y resistencia a la insulina.

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CUTANEOUS BLASTOMYCOSIS. 2/2
Deep mycosis caused by dimorphic fungus Blastomyces dermatitidis. Can affect both humans and other mammals. Characterized by geographic distribution in specific areas of North America, in riparian regions of rivers and lakes.

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BLASTOMICOSIS CUTANEA. 1/2
Micosis profunda causada por el hongo dimórfico Blastomyces dermatitidis. Puede afectar tanto a humanos como a otros mamíferos. Caracterizada por su distribución geográfica en áreas específicas de América del Norte, en regiones ribereñas de ríos y lagos

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CHALAZION. 2/2
Characterized by the obstruction and subsequent inflammation of a meibomian gland in the eyelid. It presents as a painless and persistent nodule on the tarsus of the eyelid, frequently associated with a previous history of blepharitis or previous chalazion.

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CHALAZION. 1/2
Caracterizada por la obstrucción y posterior inflamación de una glándula de Meibomio en el párpado. Se presenta como un nódulo indoloro y persistente en el tarso del párpado, frecuentemente asociado con una historia previa de blefaritis o anterior chalazión.