My kid saw the Dr for a URI. Likely viral but said that if it doesn’t improve she may need antibiotics. There’s an assumption here. The immune response differs for viruses and bacteria, but is there a reason for why the body would clear a virus but a bacterial infection persists?
My key takeaway from recent studying is that cancer physiology is extremely different from normal cells. Yet we use transformed cell lines all the time in research instead of primary cells. Surely this may skew some results? Why don’t more researchers discuss this limitation?
Connecting the dots is something that I've learned over the years about developing training and education materials. What are some tips that you have picked up?
I love medical writing. This weekend I was reading about kidney physiology and hyperkalemia for a grant and discovered instead why I get bloated during my cycle. :D
For #AMWA2021, we prepared a video that explores how well our loved ones understand what we do (spoiler alert: they don't!). Please enjoy! #medical writing @AmMedWriters
It's crucial for medical writers to become familiar with the structure and contents of PI (package inserts). In this blog, we explore four sections of the FPI (full prescribing information) and the information that can be found within.
#medcomm
https://t.co/Rju0OrLOZP
@AMAManual, how do you cite the search results of a
term from https://t.co/S6zQy44lj1? Eg, if you wanted to cite that there are 12 studies that appear in the results when you search for "breast cancer".
Very excited to announce the recently published review article on the conserved motifs in the flavivirus NS3 RNA helicase enzyme with @MartinMcCullag1 and @GeissLaboratory! Check it out https://t.co/PHkGectY9v
Update: After digging through the literature, I’ve found that a cytokine sink seems to refer to cell populations that sequester and degrade given cytokines so that those cytokines are not available to sustain the activity of relevant immune cells. Does this sound accurate?