If an emotionally healthy person is unhappy with something you do, they will address that specific behavior.
If an abusive person isn’t happy with something you do, they will target and attack your character as a whole.
Healthy person: “It bothers me that you always leave your clothes on the floor.” Abusive person: “You never pick up after yourself. You are so lazy and incompetent.”
Healthy person: "I feel hurt when you cancel our plans last minute." Abusive person: "You always ruin everything. You're so unreliable and selfish."
Healthy person: "I get frustrated when you don't listen to what I'm saying." Abusive person: "You never pay attention. You're completely incapable of understanding anything."
Healthy person: "I wish you would communicate with me more when you're upset." Abusive person: "You can't even have a discussion. No wonder no one can stand being around you."
#EmotionalAbuse #CoerciveControl
Some people ask why I keep repeating “no one is coming to save us” in my trauma recovery material. It’s not to bum anyone out. It’s because in my experience waiting for someone to save us is one of the most destructive patterns trauma survivors get tricked into.
Nobody’s coming. Our recovery is 100% on us. And that’s actually the good news.
1. Who’s allowed to make mistakes, and who isn’t?
In a toxic narcissistic family, most members can make mistakes and still be forgiven or even excused. The scapegoat’s mistakes—real or fabricated—are magnified, dissected, and made a stink about.
2. Who tells the same story, and who changes their story?
The scapegoat’s account of events tends to be consistent over time. Abusers often shift, embellish, or contradict their earlier versions depending on the audience.
3. Who uses specifics, and who speaks in vague accusations?
A scapegoat can usually describe concrete events with times, places, and details. Abusers tend to rely on broad, emotional claims like “they’re just difficult” or “you don’t know what they’re really like.”
4. Who is under constant surveillance and gossip?
If one person is the ongoing topic of criticism, yet you rarely hear them badmouthing anyone else, that’s a red flag.
5. Who stays calm vs. who needs a crowd?
The scapegoat may appear defensive in the moment (because they’re under attack) but generally seeks peace. The real bully needs witnesses, backup, and an audience to feel powerful.
6. Who gets isolated?
If an outsider notices one person consistently excluded, avoided, or treated as though they’re radioactive, that’s often the true target of the bullying.
If one person think SARS-CoV-2 means "common cold", and another person knows COVID-19 is a life-span altering vascular disease, we are living in two very different realities my friend
A new review pulls the neurobiology of Long COVID into a pretty strong map.
Neuroinflammation here is not treated as one isolated process. It’s the place where viral persistence, glia, BBB, blood vessels, mast cells, vagus nerve, metabolism, and unstable brain networks all meet🧵
Die bisher beste Long Covid Studie wurde nun veröffentlicht, mit überraschenden Ergebnissen die neue Therapieansätze ermöglichen. Vereinfacht gesagt: Long Covid wird (in den untersuchten Bereichen) von Autoantikörpern im Blut verursacht, die dann sehr wichtige Teile des Gehirns angreifen und eigentlich alle Symptome erklärbar machen.
Wichtige Erkenntnis: Long Covid Patienten dürfen keinesfalls Blut spenden denn es scheint übertragbar zu sein über jene Autoantikörper im Blut!
Toller Artikel, verständlich geschrieben:
https://t.co/GDxqontAb1
People in the comments saying it was 15 mins away, in the middle of the night and between gangs so there's nothing to worry about, just goes to show how accustomed they are to a constant threat of gun violence. It's normalised.
This is measurable and not a series of imagined symptoms.
My digestive system either works overtime so I feel empty and hungry most of the day or without warning I feel full, nauseous and bloated for a few days.
Your autonomic nervous system runs everything you do not think about. Heart rate, blood pressure, digestion, sleep, temperature.
In Long COVID, it runs all of it at emergency settings.
Beyond brain fog: viral proteins as convergent drivers of neuroinflammation and proteinopathy
🚨“COVID-19 never really leaves your brain.”
New science review proposes SARSCoV2 viral proteins stay behind as long-lived toxins, triggering chronic neuroinflammation and planting the seeds of Alzheimer’s and Parkinson’s, even after mild infection.
This very interesting and eye-catching GERMAN review reframes post-viral neurological syndromes( L0ngC0vid) as driven by persistent viral proteins acting as long-term toxins ("protein-as-pathogen" model), not just the active infection!
➡️Core mechanisms:
- SARSCoV2 Spike and OTHER viral proteins activate glial TLR4/TLR2 receptors, triggering chronic neuroinflammatory cascades via NLRP3 inflammasome,
- They also disrupt autophagy, allowing toxic protein aggregates (tau, amyloid-beta, α-synuclein) to accumulate and seed neurodegeneration,
➡️SARSCoV2 specific evidence:
- Animal studies show Spike protein alone (without live virus) induces TLR4-mediated cognitive deficits, memory impairment, synaptic loss, and sustained neuroinflammation, recapitulating post-COVID syndrome,
- Spike binds α-synuclein, accelerating Parkinson-like clumps,
➡️Human data evidence:
- Millions experience "brain fog,"
- Post-COVID patients exhibit measurable brain damage: cortical thinning, hippocampal iron accumulation, and biomarkers of ongoing neuronal injury,
➡️Broader risks:
- Even mild infections leave lingering proteins that promote Alzheimer’s and Parkinson’s-like pathology via shared pathways,
- Same pathways seen in influenza, dengue, West Nile etc,
- Mild infection = no protection,
‼️So, according to this review, the “protein-as-pathogen” model makes it crystal clear: every new SARSCoV2 infection (even mild or asymptomatic) deposits more of these long-lived toxic viral proteins into the brain. They don’t fully clear. They accumulate.
Each reinfection reloads the TLR4/TLR2 → NLRP3 inflammasome trigger and further collapses autophagy, speeding up the tau/amyloid/α-synuclein proteinopathy and neurodegeneration.
SARS-CoV-2 does not just infect.
It weaponizes its own proteins as long-lived intracellular saboteurs.
Millions are probably already carrying this hidden payload.
This is not brain fog.
This is a silent, population-scale reprogramming of human brains toward dementia-like decline.
The long-term neurological cost will probably dwarf the acute pandemic itself!
#AvoidSars2 #AvoidReinfections
https://t.co/x0oxacaNwl
I really don't think enough people fully comprehend the worlds that are about to collide here.
You already have people in geopolitical circles warning about the threat of famine based on surging prices / availability of fertilizer components, and you also have long-term weather modeling all converging on a worst case scenario for a building El Nino event, which will peak near the end of the year. These are two slow moving but entirely predictable disasters that when coupled together will each make the other orders of magnitude worse. (This will take months to fully unfold, but at this point, the die is cast.)
There's no event in our history books that combines the current global population with the impending fertilizer shortage and the strength of the El Nino that's coming. We are about to witness an unprecedented event that will push crops around the globe to their limit.
‘There's no event in our history books that combines the current global population with the impending fertilizer shortage and the strength of the El Nino that's coming. We are about to witness an unprecedented event that will push crops around the globe to their limit.’
I've always said that brain fog conjures up an image of slight absent mindedness, not the horrific abyss of being unable to remember a single word in a sentence, how to write numbers, if you had lunch, names of your family.
Crying with fear in case it lasted forever.
I prefer “altered brain” to “brain fog.”
“Brain fog” sounds harmless. Temporary. Like you just need a nap or another cup of coffee.
But altered brain function raises a different question:
What changed?
We have evidence of neuroinflammation. Evidence of vascular injury. Evidence of elevated inflammatory markers. Evidence of neuronal damage and structural changes in some patients.
Whether those changes are reversible, partially reversible, or permanent remains an active area of research.
But damage doesn’t become harmless simply because the brain is capable of adaptation.
An ACL tear is still an ACL tear, even if rehabilitation restores function.
A scar is still a scar, even if you learn to live with it.
The brain is no different.
Neuroplasticity is remarkable. People recover function after strokes, traumatic brain injuries, and other neurological insults every day.
But recovery is adaptation… not a time machine.
The brain doesn’t roll back the clock to the moment before the injury occurred.
Instead, it reroutes traffic around damaged roads.
That’s why I’ve said since the earliest neurological findings emerged that COVID had the potential to cause brain injury. The extent, prevalence, and permanence are still being worked out.
But the idea that a virus can injure the brain shouldn’t be controversial. Viruses have been doing that throughout human history.
The real question is not whether the brain can adapt.
The question is how much damage occurred before adaptation became necessary.
Oh, there's no doubt: trauma recovery is going to feel like an identity crisis-- and it is. The identity you were conditioned to accept will not fit into your new life, where self-respect & self-care are non-negotiables. And your new identity would terrify your bullies & abusers.
We must be crystal clear about a few points. Long Covid is not a "poorly understood neuroimmune disorder" with no biomarkers. It is a widely studied biological medical condition. Pathology spanning basically all body systems is well documented, with damage in situ across organs
@hannahspierMD 1/Hannah, we have now almost 500,000 peer reviewed published research into long covid now. “Fatigue”, “post exertional malaise (PEM)” “Brain Fog” “difficulty concentrating” are not psychiatric symptoms. They have a pathological basis and clear demonstrable abnormalities 1/
Another incredible photo of a beautiful bird.
As Robert Macfarlane said in The Lost Words:
Kingfisher: the colour-giver, fire-bringer, flame-flicker, river’s quiver.
So many of my followers are no longer seeing my photos. 😔
If you see this Kingfisher photo, please leave a comment! 🙏
Do you see my photos every day on your timeline or not? 🐦