Our comments on the future (and present!) of neuromodulation in psychiatry. Another call to action in pushing the field forward through education/training, collaboration, and infrastructure. Our patients deserve it.
https://t.co/bpfFNzw4Dd
@UCLAHealth@ClinicalTMS #neuromodulation #TMS #psychiatry
In today's Age of (Mis)Information, there's an overwhelmingly unfathomable attraction to people saying "I've figured something out that no one will tell you." Conspiracies, fringe medical treatments, etc. With unfiltered, copious amounts of information flying through all channels, it's as if it has become boring and mainstream to trust reputable information and science. We can and should be thoughtful and critical, but that also doesn't mean believing in ideas that have no merit and are simply against evidence/data.
Reassuring cohort study showing a lack of association between maternal peri-partum S(N/S)RI treatment and negative neuropsychiatric effects in offspring. Ultimately: more important to treat maternal depression and anxiety, which ARE associated with worse outcomes when untreated.
https://t.co/bFzQ2vM7GP
For all the anti-science/anti-vaxx/anti-psychiatry etc folks out there:
If we share a common ground - to be compassionate and helpful to our fellow human beings - then you may care about science more than you feel.
Science is simply the exploration of truth.
Over time, tools and methods were designed to allow us to rigorously explore ideas or thoughts about our world, or a medication, for example.
Yes, we should always be thoughtful - even skeptical - of the agenda of institutions and companies who pursue those explorations.
But the methods of testing something over and over, the method of being undying in collection of data objectively to pursue truth, should never be replaced by personal anecdotes.
Personal stories are vital to our personal world. But applying anecdotes to everyone else is dangerous, and not indicative of a compassionate desire to understand truths and help each other.
We both want and need science to help us in the pursuit of truth in a world full of chaos and entropy.
https://t.co/HX0GDEiMLB
The comments on this reckless article are reassuring and important. The vast majority of readers rightfully reject someone charging $$ by providing dangerous "services" developed without education and training, but rather by their personal history, folk psychology, dogmatically rigid antipsychiatry views, and self-aggrandizement masked as altruism. Dissuading all comers from taking all medication is ridiculously detrimental and ironically unempathic to the population meant to be "helped" here. Plus, perhaps it's evidence of her doing so well currently, of not only her social and financial supports, but perhaps some of her prior medication treatment during her personal dark times.
There is no one size fits all approach, and over-prescribing is not an insignificant problem in some cases, but please do not stop or taper medications without help from a trained psychiatrist.
Suthana et al. studied brain recordings from implanted electrodes in freely moving humans. They show that neural dynamics encoded actual and imagined routes similarly. @SuthanaLab@m_seeber
https://t.co/tKardIicUs
Augmenting TMS response in MDD: We know DLPFC-TMS works, and for non-responders that OFC-TMS adds some 20+% remissions, so what if we combine these two protocols? Large sample shows no advantage. Maybe it is about specificity of TMS?
Congrats @AmouriePrentice and @NikitaVinne with this work, and great collaboration with @JonathanDownar
https://t.co/C3tg5Gny5b
Another win for accelerated treatment! Now we need head-to-head trials. But great to see these responses in depression over 15 days without neuronavigated targeting.
https://t.co/uOoHUPZyGY
And even more so, it's a waste of time for her and others to double-down on semantics. If people are suffering and want help, we ought to do our best for them. She'd rather they suffer indefinitely I suppose. Is it really more important to emphasize and equate any sad mood state with months of refractory depression?
TMS is delivered to the DLPFC in depression, aiming to modulate SGC activity
But axons linking DLPFC and SGC are absent
How then does TMS stimulation modulate SGC activity?
Caio's work on TMS propagation in the absence of direct anatomical paths:
https://t.co/WIN8S7XYF0
DEI IS an initiative TO hire the best and brightest. Because historically industries haven't "broadened their search" to find typically OVERQUALIFIED (out of necessity) minority candidates for positions. Getting rid of these initiatives means going back towards mediocrity. It's not difficult to understand, and it's disappointing and despicable if you can't.