Top Tweets for #Askrenalpath
In primary amyloids DIF-F is the most helpful and cost effective tool! Isn't it??
#Renal #RenalPath #AskRenal #AskRenalPath #RenalCases
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Now see these two!
Primary amyloid can be extremely subtle, without DIF, you can easily miss it! PAS won't help. May miss on DIF-P!
F,58Y, recurrent skin purpura for 20yrs, 24hUP1-2g, MIg: IgAL, sFLCR0.002, MPGN with IN (mainly plasma cells); IF：C3+++Mes, L+++(Glo+BMC+TBM, no Arteriol deposits), no IgA/G/M; EDD without substructure. Diag: L-PGNMID with TBM-L deposits?#askrenalpath #renalpath #nephrology
22yrs, male, nephrotic syndrome. 1st RP 3yrs ago: IC-MPGN(full house). Very low C3 with C6 mutation (LP). Pred+MMF, then Ecub (C5b9 increased), UP decreased. 2nd RP: less Igs, similar C3, MPGN pattern, low dense deposits. IC-MPGN transformed to C3GN? #askrenalpath #renalpath
70yrs, Male, DM 20yrs, high urea acid, HTN, gouty, thyroid malignancy. RB revealed glomeruli ischemia and medulla fibrosis? surround the tubules, what's the diagnosis #askrenalpath #renalpath
Case 6 (2026): A 20 year old was biopsied for acute renal dysfunction. Urinalysis was bland. IF was negative. What’s your diagnosis? What are the common causes for this pathology? #askrenalpath
A consultation case: RB 3yrs ago (AKI after NSAID, didn't do MIg detection), but MM now. LM with only ATN features in several tubules (PAS negative casts), no typical MM casts but light chain was restricted. OK to diagnose LCCN-lambda? #renalpath #askrenalpath
C4 glomerulopathy is a rare disease with only a small case series. The glomerular lesion is mostly MPGN with negative Igs, C3, C1q and need C4d staining for diagnosis. Have anyone see one of these cases? #askrenalpath
M, 48y, increased Scr (123umol/L) for 3m. LM shows (a) an "onion-skin" collagen nodule, and (b) a granuloma with central dark spherules. What might be the cause? #askrenalpath #renalpath
Would you consider dysmorphic lymphoplasmacytic infiltration as suggestive of a lymphoproliferative disorder? #renalpath #askrenalpath
45y, F, CML 13yrs (stable, controlled by TKI), UP1.8g, URBC++, Scr 144umol/L. LM: IgAN(no active lesions) with slight TMA, mild IFTA. EM: several smuggy lysosomes in focal PTs. Should lysozyme nephropathy be considered? #renalpath #askrenalpath
#askrenalpath F, SCr increased from 0.8 to 4.3, proteinuria 3g/d, treating lung cancer with anti-VEGF, osimertinib, diphosphonate for several years. Recently adding heparin, diuretic. Microaneurysm(related to antiVEGF) and unique myelin bodies was seen.gene test-. Which drug?
60yMale, no DM/HTN/smoking, SLL/CLL for 5y, 24hUP0.6g, URBC486/ul, Scr-, RB: lymphoma cell infiltration and glo nodular sclerosis. Is the nodule pattern caused by SLL/CLL? #renalpath #askrenalpath
29y male，body builder, 1-1.7g proteinuria, Alb normal, Scr slightly increased (92.9-97.2-100.3) umol/L. LM: 2 glom with FSGS lesion, but what is the pie-like granular lesion in interstitium? #renalpath #askrenalpath
IgA subclasses showed IgA1 only. IFE could not be performed on limited cryoprecipitates. MS results showed amyloidosis (AK as well), which is more confusing! #renalpath #askrenalpath How many of you would believe it's amyloidosis!
#askrenalpath young female, NS, normal Scr, membranous pattern on LM. subepithelial EDD with spherules on EM. IgA was 3+, while IgG was trace(shown as follows). K and 入both positive, C1q was negative. No secondary factors was found. Serum complement was normal. Any thoughts?
68yrs, male, NS. LM: MN pattern; IF: IgAK+; GdIgA1+; EM: subepithelial fibrils. No MIg detected. Cryo (weak+). FGN? PGNMID? CryoGN? #renalpath #askrenalpath
#askrenalpath 2 weeks after RTX, Scr increased from 2.2-5.6, adenovirus + in blood. PRA-, C4d-, low C3. T2 and i3 in cortex. Ruptured tubules and tubular microabscess in a small part of medulla. Swollen endothelial cell and TMA lesion Can not do adenovirus IHC. Diagnosis?
26y, M, AKI, used NSAID+Contrast Agent；What do you think of those fibrin like materials?#renalpath #askrenalpath
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