Pleased to share our paper showing that suppression of lipid biosynthesis may potentially improve the efficacy of chemotherapy against human prostate cancer. @DukePathDept@DukeMedSchool@DukeCancer
https://t.co/QHc3fuI8sB
Tracking down metabolic vulnerabilities in CDK12‐mutant prostate cancer - https://t.co/y2liHY5veV We commented on an article by Zhang et al. showing mitochondrial ETC as a potential vulnerability for targeted therapies in CDK12-mutant prostate cancer; #Commentary
Excited to share our paper spearheaded by my postdoc Mu-En Wang, detailing how RB1 loss leaves cancer cells susceptible to ferroptosis. Big thank you to our awesome collaborators, reviewers, and editor. https://t.co/AmJ9xc14jc @DukePathDept@DukeCancer@gaziou@AarmstrongDuke
Pall of suspicion by
@jeffmervis: NIH’s secretive "China initiative” has upended hundreds of lives and destroyed scores of academic careers https://t.co/KolupTCCKI
A team led by @DukePathDept chair Jiaoti Huang, MD, PhD, found glypican-3, a protein on the surface of neuroendocrine #prostatecancer cells provides a better opportunity for targeting cells that are intrinsically resistant to hormonal therapy. @DukeCancer
https://t.co/AQNcbDn2Fb
Check out our PhD Graduate program, which gives students the unique ability to do research in the basic sciences as well as in the translational sciences. https://t.co/ca7jgnUdnX via @YouTube
Please RT 🎉🎉🎉
Join us to understand how the ribosome dictates cell identity, focusing on hematopoiesis and leukemia. We're studying cell identity in-vivo and developing new and amazing tools!
Fully funded @ERC_Research positions - postdocs and PhDs.
https://t.co/c3YeUCSEVt