Scientists are frantically searching for the cause of long-Covid, and are mystified by the myriad of symptoms in different long-covid patients. Long-covid is not a one size fits all disease, arising from a single cause; which makes understanding the cause particularly difficult
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Scientists are frantically searching for the cause of long-Covid, and are mystified by the myriad of symptoms in different long-covid patients. Long-covid is not a one size fits all disease, arising from a single cause; which makes understanding the cause particularly difficult
intracellular co-infections and combinations of co-infections can cause and direct the myriad of different symptoms in long-covid patients. Diagnosing underlying chronic intracellular infections and directing treatment to the underlying co-infections could aid in understanding
could help all students become better readers, and the class winners will also likely be the best readers in the class. Children should be encouraged to choose a dominant hand and helped to develop a fixed gaze.
Children who do not develop a strong hand dominance are more likely to have difficulty learning to read; and disorganization in the brain, which causes confusion in storing and retrieving information. It does not matter which hand is dominant, children should be encouraged to
choose a dominant hand. Similarly, children who fail to develop a fixed gaze by age seven have a greater difficulty learning to read. A fixed gaze can be developed by focus on a task and by old fashioned staring games, i.e. who blinks first? Staring contests in the early grades
the life cycle of the pathogen. Further investigation of underlying chronic infections and prior use of penicillin may add to the understanding of long-covid, and its myriad of symptoms.
An explanation for long-covid has been evasive. Underlying chronic intracellular infections of many types are the likely reason for long-covid. Covid enters the cell and becomes intracellular to allow reproduction, and can merge with to the underlying chronic intracellular
asked if they have been prescribed penicillin. Penicillin potentiates intracellular pathogens by destroying the cell wall but not the pathogen, causing the reticulate body to change shape and become larger, allowing the pathogen to make more elementary bodies, and lengthening
Science has now quantified the risk of a heart attack as six times greater after having influenza. We have no reason to believe an increased risk of a heart attack and other co-morbidities would be different after covid than after influenza. The best protection is to avoid
intracellular pathogen; thus, people with these chronic conditions are at higher risk when infected with covid19, because the same underlying chronic intracellular infection causing these conditions increases the risk when co-infected with covid19.
Most chronic diseases have one or more known co-morbid diseases. Often co-morbid diseases are not independent diseases, but rather represent the evolution of a chronic disease from a common originating pathogen, over time. The co-morbid named diseases are based on a description
disease increases the risk for Parkinson’s disease, because both are caused by the same intracellular pathogen, which evolves into another co-morbid chronic disease over time. Cardiovascular disease, macular degeneration, and Alzheimers, can be caused by the same chronic