๐จ DR. WILLIAM MAKIS SAYS THIS IS THE MOST IMPORTANT IVERMECTIN DOSING GRAPH HE'S CREATED.
Not a new chemotherapy drug.
Not a breakthrough immunotherapy.
Not a billion-dollar cancer treatment.
Just one simple dosing strategy.
According to Dr. William Makis, this is the dosing framework he believes everyone researching repurposed medicines for cancer should understand first.
And it all starts with one number.
๐ 1 mg/kg/day.
Before explaining why, here's the dosing framework Dr. Makis discusses.
๐ The Four Ivermectin Dosing Ranges
๐ข Low Dose: โค0.5 mg/kg/day
Used for cancers in remission, strong family history, genetic predisposition, prophylaxis, and lower-dose protocols discussed in published case reports.
๐ก Medium Dose: 1.0 mg/kg/day
Dr. Makis' recommended starting dose for most cancers.
๐ต High Dose: 2.0 mg/kg/day
Discussed for very aggressive cancers, including leukaemia, pancreatic cancer, and some brain cancers.
๐ด Very High Dose: โฅ2.5 mg/kg/day
Discussed in selected reported cases involving extensive metastatic disease or extremely poor prognosis, where temporary visual side effects have also been reported.
Now here's why Dr. Makis believes 1 mg/kg/day deserves the most attention.
After reviewing laboratory research, published studies, and numerous case reports, he says this is the starting dose he recommends for most cancers.
According to Dr. Makis, research has investigated Ivermectin across a surprisingly wide range of cancers, including:
โข Breast cancer
โข Colon cancer
โข Lung cancer
โข Pancreatic cancer
โข Kidney (renal cell) cancer
โข Gastric cancer
โข Leukemia
One exception stood out.
Lymphoma.
Dr. Makis says he has found very little published research involving Ivermectin and lymphoma.
That doesn't mean it doesn't work.
It simply means the evidence is currently limited.
โWhat does 1 mg/kg/day actually look like?
For someone weighing 60 kg (132 lbs)...
That's approximately 60 mg of Ivermectin every day.
Equivalent to:
๐ Five 12 mg tablets
OR
๐ง About 6 mL of liquid Ivermectin, roughly one teaspoon.
According to Dr. Makis, that's where he recommends starting for many cancers.
Can lower doses still have an effect?
โ He believes they can.
Dr. Makis discusses a published ovarian cancer case reported by Dr. Tess Lawrie.
The dosing chart lists the patient as receiving 12 mg of Ivermectin per day. During his discussion, Dr. Makis explains that this was approximately 0.2 mg/kg/day, placing it within the chart's Low Dose (โค0.5 mg/kg/day) range.
๐ According to the case report, the patient's CA125 tumor marker reportedly fell from 288 to 22 after two months.
That case is one reason Dr. Makis believes Ivermectin may have anticancer activity even at lower doses.
However...
He also believes the available evidence suggests the effect is dose dependent.
In other words...
Higher doses may produce greater effects, although more clinical research is needed.
What about the most aggressive cancers?
โข Pancreatic cancer
โข Leukemia
โข Brain cancer
For cancers like these, Dr. Makis discusses doses around 2 mg/kg/day in selected situations.
He notes that Ivermectin crosses the blood-brain barrier, although not very efficiently, which is one reason he says higher doses may be considered for some brain cancers.
He also discusses a case reported by Dr. Allan Landrito.
According to Dr. Makis, a physician with terminal gallbladder cancer reportedly took 2 mg/kg/day for 14 months.
He says her cancer eventually disappeared.
She experienced temporary visual side effects that resolved after a few days.
โHow high have reported doses gone?
According to Dr. Makis, he has seen reported doses as high as 2.5 mg/kg/day.
He references patients treated by Dr. Shankara Chetty who reportedly used those doses without long-term side effects, although some experienced temporary visual disturbances that later resolved.
One point Dr. Makis returns to repeatedly...
Long-term use.
He references the Castro study involving children with leukemia who reportedly received Ivermectin for six months without long-term side effects.
He also discusses anecdotal reports of adults taking it daily for more than a year without long-term toxicity.
So why aren't there larger cancer trials?
Dr. Makis believes one of the biggest obstacles is economics.
Because Ivermectin is an off-patent generic medicine, he argues there is little financial incentive to fund large clinical trials.
Whether future research ultimately confirms or challenges these ideas remains to be seen.
But Dr. Makis' message is remarkably simple.
One graph.
One dosing strategy.
One starting point.
๐ 1 mg/kg/day.
Because the conversation isn't just about Ivermectin anymore.
It's about whether repurposed drugs deserve the same level of scientific investigation as newer therapies.
๐จ๏ธ Do you think inexpensive repurposed medicines like Ivermectin deserve larger, well-designed clinical trials for cancer?
๐ Save this post so you'll always have Dr. Makis' dosing framework handy, and repost it to help others discover the ongoing research into repurposed medicines.
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Many patients have asked whether they need to use ivermectin, fenbendazole, and mebendazole simultaneously. This post aims to clarify the situation. According to this description, each of these substances operates through distinct mechanisms and targets cancer in different ways.
๐@ivermectincure
Ivermectin vs. Fenbendazole vs. Mebendazole
๐ฌ Ivermectin vs. Fenbendazole vs. Mebendazole: 12 Proposed Anti-Cancer Mechanisms Each Uses to Attack Tumors
These repurposed medications were originally designed to fight parasites, but research and real-world use have suggested they may do much more. Each one is claimed to attack cancer on multiple biological fronts, helping to shut down tumor growth, starve cancer cells, enhance the immune system, and more.
Here's a breakdown of how each is claimed to work:
โ IVERMECTIN โ 12 Proposed Anti-Cancer Actions
1. Inhibits the WNT/ฮฒ-catenin pathway โ may stop cancer cell proliferation.
2. Induces apoptosis โ triggers programmed cancer cell death.
3. Blocks importin ฮฑ/ฮฒ transport proteins โ may prevent cancer cell replication.
4. Inhibits the PAK1 enzyme โ may reduce inflammation and tumor progression.
5. Anti-angiogenic โ may inhibit the formation of new blood vessels in tumors.
6. Modulates the immune system โ may enhance recognition of cancer cells.
7. Disrupts autophagy โ may interfere with cancer cell survival strategies.
8. Targets glioblastoma stem cells โ proposed activity in brain cancers.
9. Inhibits mitochondrial respiration โ may reduce the energy supply to tumors.
10. Disrupts mTOR signaling โ may slow cell growth.
11. May help overcome chemotherapy resistance โ potentially making chemotherapy more effective.
12. Has antiviral properties โ potentially relevant for some virus-associated cancers (such as HPV-related cancers).
โ FENBENDAZOLE โ 12 Proposed Anti-Cancer Actions
1. Disrupts microtubules โ may prevent cancer cells from dividing.
2. Inhibits glucose uptake โ may starve cancer cells of energy.
3. Activates the p53 tumor suppressor gene โ may help eliminate damaged cells.
4. Triggers apoptosis โ proposed activity in lung, colon, and prostate cancers.
5. Inhibits metastasis โ may reduce the spread of cancer.
6. Increases oxidative stress in cancer cells โ may make them more vulnerable.
7. Modulates the immune system โ may help the immune system target tumors.
8. Blocks angiogenesis โ may prevent tumors from developing a blood supply.
9. Depletes glutathione in tumors โ may weaken tumor defenses.
10. Suppresses the AKT signaling pathway โ involved in cancer cell survival.
11. May help restore normal cell-cycle regulation โ potentially limiting uncontrolled growth.
12. May work synergistically with other agents (such as CBD, curcumin, and vitamin D).
โ MEBENDAZOLE โ 12 Proposed Anti-Cancer Actions
1. Destabilizes microtubules โ similar to fenbendazole.
2. Inhibits angiogenesis โ may block new blood vessel growth.
3. Triggers apoptosis โ promotes cancer cell death.
4. Inhibits VEGF signaling โ may reduce tumor blood supply signals.
5. Crosses the blood-brain barrier โ proposed usefulness in brain cancers.
6. Activates caspase-3/7 enzymes โ involved in programmed cell death.
7. Reduces MYC oncogene expression โ may slow tumor growth.
8. Inhibits the Bcl-2 protein โ may decrease cancer cell survival.
9. Has anti-metastatic activity โ may reduce cancer spread.
10. Disrupts mitochondrial function โ may impair tumor energy production.
11. May improve chemotherapy sensitivity โ potentially helping standard treatments work more effectively.
12. Low toxicity and a long safety record โ has been used in humans for decades.
๐ฑ Each of these medications is claimed to target cancer through different biological pathways.
Visit:-๐ https://t.co/REwGZCcJqB
#Ivermectin #Fenbendazole #Mebendazole #CancerCure
๐จ I just did a podcast on cancer remedies that the AMA doesnโt want you to know about:
โข Chlorine Dioxide
โข Ivermectin / Fenbendazole
โข Baking Soda & Vitamin C
โข Royal Rife frequency machines
โข IP6 & Inositol
โข Laetrile (B17 / Apricot Seeds)
A physician in Salt Lake City once shared a Stage 4 cancer protocol with a friend of mine. He had them leave their phones outside and write the instructions down, saying he was risking his medical license by revealing it.
The Rockefeller-influenced AMA is corrupt.
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