@strokeaction_RW joined fellow OPDs and partners in commemorating #Kwibuka32, honoring victims of the 1994 Genocide against the Tutsi and reaffirming our commitment to inclusion, unity, peace, and fighting genocide ideology. #NeverAgain
You’re young. Your body is not invincible. Every cigarette damages your lungs, blood vessels, brain, and heart long before symptoms appear. Don’t let tobacco steal your future. Quit smoking today and protect your life tomorrow. #StrokePrevention#QuitSmoking
Fascicular Blocks: Small Conduction Delays, Big ECG Clues
The left bundle branch divides into fascicles, and even modest conduction delay in one fascicle can significantly alter the sequence of LV activation, producing characteristic ECG patterns without substantially prolonging QRS duration.
Left Anterior Fascicular Block (LAFB):
Delay in the left anterior fascicle causes early activation of the inferoposterior LV, followed by late activation of the anterosuperior LV.
ECG findings:
🟢Marked left axis deviation (typically −45° to −90°)
🟢qR pattern in aVL
🟢rS pattern in leads II, III, and aVF
🟢QRS <120 ms
🟢R-wave peak time in aVL ≥45 ms
🟢Precordial leads may show delayed transition and deeper S waves in V4–V6.
🟢Often benign in otherwise healthy individuals, but in patients with coronary or structural heart disease, LAFB has been associated with increased mortality risk.
Left Posterior Fascicular Block (LPFB): Delay in the left posterior fascicle causes early activation of the anterosuperior LV and late activation of the inferoposterior LV.
ECG findings:
🔵Right axis deviation (often >+90°, with >+110° improving specificity)
🔵rS pattern in leads I and aVL
🔵qR pattern in leads III and aVF
🔵QRS <120 ms
🔵Before diagnosing LPFB, exclude more common causes of right axis deviation such as RV overload, normal variants, and extensive infarction.
LPFB is uncommon because the posterior fascicle is thicker and better protected; when present, it often reflects significant underlying cardiac disease and may coexist with RBBB.
Key Concept Fascicular blocks change the sequence of ventricular activation rather than the total duration of activation. As a result, the QRS axis shifts dramatically while QRS duration usually remains normal.
Follow @TrackYourHeart for daily cardiovascular insights.
Reference: Braunwald's Heart Disease: A Textbook of Cardiovascular Medicine. Chapter
A stroke changed his life, but not his determination. Robert’s recovery journey shows that hope, rehabilitation, and support can help people rebuild their lives. 💙 What inspires you to keep going through difficult times?
Read more: https://t.co/IwUMGF944A
Today, on May 31st, the world joins together to mark #WorldNoTobaccoDay2026 🚭
Many people think smoking is a way to relax or have fun, yet tobacco smoke causes serious diseases including lung cancer, heart disease, and respiratory illnesses.
Even non-smokers, especially children, can be harmed by secondhand smoke. Choosing not to smoke is choosing life and protecting the people we love.
@RBCRwanda@RwandaHealth
#WorldNoTobaccoDay #TobaccoExposed #NoTobacco
@strokeaction_RW yifatanije n’imiryango y’aba bana mu kwishimira iki gikorwa cy’indashyikirwa cy’ubuyobozi. Ubufasha nk’ubu ni ingenzi mu gufasha ababana n’ubumuga kugira ubuzima bwiza no kwigira.
Atrial fibrillation with the Wolff-Parkinson-White (WPW) syndrome.
The clues to this diagnosis are the extremely rapid wide-complex tachycardia (about 300 beats/min at times) with a very irregular rate.
This rhythm constitutes a medical emergency since it may spontaneously degenerate into ventricular fibrillation.
Key movement patterns seen in basal ganglia lesions
High-yield review: chorea, athetosis, ballismus, tremor & parkinsonism
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Angioedema is more than an allergic swelling.
It is a localized, non-pitting swelling involving the deeper layers of the skin or mucosa, commonly affecting the lips, face, tongue, airway, and gastrointestinal tract. The most critical distinction is understanding why it happens, because treatment depends on the mechanism.
Histamine-mediated angioedema is the classic allergic form. It is often triggered by foods, medications, insect stings, or latex. Patients may also have urticaria, itching, flushing, bronchospasm, or even anaphylaxis. Symptoms usually develop rapidly, often within an hour of exposure, and standard therapy with antihistamines, corticosteroids, and epinephrine is effective.
Bradykinin-mediated angioedema is a different disease process entirely. It is not driven by allergy, so antihistamines and steroids may fail. Common causes include ACE inhibitor use, hereditary C1 esterase inhibitor deficiency, and acquired C1 inhibitor deficiency. These patients typically present without urticaria or itching, often with more prolonged and severe swelling, and may have prominent abdominal pain that mimics an acute surgical abdomen.
The immediate priority in any angioedema patient is airway assessment. Tongue, pharyngeal, or laryngeal involvement can rapidly become life-threatening.
A practical bedside clue:
Itching plus hives =think histamine.
No hives, recurrent swelling or ACE inhibitor exposure = think bradykinin.
Correct diagnosis matters, because not every swollen lip is an allergy.
Read more:
https://t.co/ypNklfg5po