Torill M.F F

Sykepleier, gift , mor til 3

[Torill M.F F @torill_fr]

Norges håndtering av covid 19 kommer til å bli tidenes skandale .@Folkehelseinst hevder at covid 19 er som en forkjølelse, Dette er løgn. De hevder at gjentatte infeksjoner er bra for oss, Dette er løgn. Anbefalingene fra FHI er at testing er unødvendig. WHO anbefaler testing Det er ingen tiltak iverksatt for å begrense smitte.All forskning viser at vaksinering beskytter noe mot long covid, og bedre ved flere vaksiner, da beskyttelsen går ut etter noe tid. Anbefalingene for vaksinering uforandret Kiurgiske munnbind blir anbefalt i helsvesnet ( ved smitte) i stedefor FF2 åndedrettsvern, covid 19 er en luftbåren smitte Arbeidsmiljøloven blir brutt hver eneste dag De kan ikke si at de ikke viste, for forsknigen ligger der , 1000vis av forkningsartikler med biomedisinske funn. tallene ligger der. WHO snakker om 400 millioner med /eller har hatt long covid , allikevel er long covid et ikke tema. Mange har advart, uten å bli hørt. Folket blir ført bak lyset, knapt noen tar dette på alvor, (dette gjelder også endel leger) Folk flest ser på dette som en forkjølelse, da dette er inprentet fra FHI,

[Torill M.F F @torill_fr]

Nå er det mange studier som viser at Covid-vaksinene beskytter mot hjertekarsykdom i tiden etter covid-19, og at de vaksinerte har lavere risiko for å dø. I denne studien ser man at det er de med gjennomgått covid-19 som har økt nivå av troponin mens de andre som er vaksinert men ikke har hatt covid ikke har det.

[Sally Bourliakas 💙 @SallyBourliakas]

From @Sunny_Rae1
https://t.co/KivMs4maqm
@hannahspierMD 1/Hannah, we have now almost 500,000 peer reviewed published research into long covid now. “Fatigue”, “post exertional malaise (PEM)” “Brain Fog” “difficulty concentrating” are not psychiatric symptoms. They have a pathological basis and clear demonstrable abnormalities 1/
@hannahspierMD 2/PEM - isn’t tiredness is impaired metabolic recovery with clear demonstrable abnormalities on 48hr submaximal CPET testing. I’m a member of the executive committee of the International Society of Long Covid & PAIS (ISLC). My research colleagues from the same group have 2/
@hannahspierMD 3/ already clearly demonstrated abnormal findings on muscle biopsies in LC w/PEM (my colleague Rob Wusts team) and my colleagues Resia Pretorius and Mark Faghy have clearly shown microclot fragmentation and cytokines driven inflammation flares post exertion in long Covid 3/
@hannahspierMD 4/my colleague Dr Danielle Beckman, Covid Neuroscientist has also shown that “brain fog” is also not psychological or psychiatric. She’s clearly demonstrated SARS2 virus in the brain infecting neurons and causing microbial mediated (among other causes) neuroinflammation 4/
@hannahspierMD 5/the PolyBio researchers, headed by Dr Amy Proal, also part of our extended committee, have demonstrated abnormalities on TSPO-PET scans in the brains of a significant number of long covid patients. TSPO tracer helps identify glial activation and demonstrates that at least 5/
@hannahspierMD 6/part of the pathology is linked to microglial mediated neuroinflammation, other groups have clearly shown the degree of neuroinflammation in the Brian’s of long covid correlates with the degree of vascular inflammation in the blood vessels and Brain MRI with ASL sequences 6/
@hannahspierMD 7/also show clear microstructures and microperfusion abnormalities in the brain.
In addition to this, Covid damages the autonomic nervous system of about 50% of individuals with long covid. We have demonstrated a drop in cerebral perfusion to the brain in the upright position 7/
@hannahspierMD 8/in patients with this phenotype. This is not a psychiatric condition it is a measurable set of pathobiological abnormalities

Coming onto the “difficulties concentrating” again not primarily psychiatric or vague. Again work by Dani Beckman & others has shown a predilection 8/
@hannahspierMD 9/ for the pre-frontal cortex in areas governing higher executive functioning which would explain the very clear dysexecutive syndrome we see clinically in our long Covid patient clinics. Also likely explaining why those with ADHD/Au may be more susceptible to developing LC 9/
@hannahspierMD 10/AND the fact that LC worsens the already preexisting higher executive function issues in ADHD

Moving on to other pathological contributing factors - SARS2 causes endothelial dysfunction and inflammation , platelet hyperactivation, and fibinaloid microclotting - this all 10/
@hannahspierMD 11/contributes to the microvascular disease we see in long Covid. This has been clearly demonstrated in multiple research

[Torill M.F F @torill_fr]

‼️‼️

[Harry Spoelstra @HarrySpoelstra]

Unraveling the cardiovascular burden of long COVID: symptom profiles, underlying mechanisms, and clinical management insights

🚨A peer-reviewed cardiac bombshell just AGAIN exposed the brutal reality of Long COVID HEART DAMAGE!

➡️This Chinees review synthesizes current evidence on the symptom profiles, underlying mechanisms, and clinical management of Long COVID-related cardiovascular complications.

➡️Symptom profile:
- Long COVID cardiovascular sequelae affect 10–20% of survivors, with persistent symptoms (≥2 months post-3-month mark) including palpitations (68%), chest pain (53%), fatigue (63%), and dyspnoea,
- POTS occurs in ~31% of cases,
- Functional deficits persist up to at least 12 months: 20% drop in 6-minute walk test distance, reduced cardiac index, stroke volume, and ejection fraction (18–29%).

➡️Structural findings:
- Cardiac MRI shows involvement in 78% and myocardial inflammation in 60%,
- Autopsies reveal myocarditis (14%) and macrophage infiltration (86%).

➡️Mechanisms:
→Multifactorial and overlapping:
- Viral persistence (spike protein detectable in 60% at 12 months),
- Chronic immune dysregulation (elevated IL-1/IL-6/IFN-γ/TNF-α),
- Endothelial dysfunction,
- Microvascular injury with microthrombi (80% in some autopsies),
- Hypercoagulability, and
- Autonomic dysregulation.

➡️Blood markers / biomarkers:
- Persistent spike protein is detectable in blood plasma (and tissues) in ~60% of Long COVID patients at 12 months, but absent in recovered controls. This further dismantles any vaccine-causation narrative,
- Inflammatory cytokines are elevated (IL-1, IL-16, IL-17, IL-22, IFN-γ, TNF-α), linked to endothelial dysfunction, platelet activation, hypercoagulability, and myocardial injury,
- Cardiac troponin is often elevated as a marker of ongoing ischemia/injury.

➡️Risk modifiers:
- Severe acute infection drives more structural damage,
- mild cases more autonomic/functional,
- Vaccination may cut risk: 30–50%,
- Delta-era cases worse than Omicron.

➡️Management:
→Currently symptom-driven:
- Graded rehabilitation,
- Lifestyle measures,
- Beta-blockers,
- Targeted anticoagulation,
→Multidisciplinary phenotyping recommended,
→No approved mechanism-specific therapies exist.

➡️Conclusions:
“Based on these findings, the following clinical recommendations are proposed:
(1) cardiovascular evaluation should be considered in patients with persistent post-COVID-19 symptoms, including autonomic function testing and imaging where indicated,
(2) management should be phenotype-driven and multidisciplinary, incorporating tailored exercise rehabilitation, pharmacological control of heart rate and symptoms, and anticoagulation only when thromboembolic disease is confirmed,
(3) vaccination should be encouraged as a preventive measure to reduce Long COVID risk; and
(4) future research should prioritize mechanism-based subtyping and randomized trials of targeted interventions.”

‼️So, AGAIN, Cardiovascular Long COVID is a real, heterogeneous, and enduring burden fuelled by unresolved viral–immune–vascular damage that current symptomatic care cannot fix. Without urgent, mechanism/phenotype-targeted treatments, millions will face chronic heart impairment for years to come!
PREVENTION NEEDS PRIORITY !
#AvoidSars2 #AvoidReinfections #CleanAir
https://t.co/OisgNHrgtR

[Torill M.F F @torill_fr]

En ny norsk studie viser at covid-19 er koblet til økt risiko for en umerkelig, kronisk skade på hjertet. Forskerne har funnet et urovekkende signal i blodet til tusenvis av nordmenn etter pandemien.

Nok en gang bagatelliserer FHI funnene. De har en arrogant overbevisning om at de selv vet best, og overgår de fremste forskerne på feltet

Fagdirektør Hanne Gulseth i FHI mener studien interessant, men at kunnskapsgrunnlaget foreløpig ikke er tilstrekkelig til å konkludere med at covid-19 gir økt risiko for hjertesykdom på sikt.
Våkn opp!!!! Vi snakker om liv og helse er

https://t.co/GsBGXstCAh

[Torill M.F F @torill_fr]

Dette kommer frem i studien

SARS-CoV-2, the vaccine elicits spike protein IgG and not nucleocapsid IgG, a serological profile separating immunization from infection. Accordingly, we did not use values of spike protein IgG to evaluate infection status, as these values would be disturbed by vaccination.

[Torill M.F F @torill_fr]

De har brukt en metode som skiller mellom vaksinen og smitte
De fleste i studien var vaksinerte study reports that 98.9% of participants were vaccinated and explain(in methods) why they used nucleocapsid igg (not spike) to avoid vaccine confounding,«

SARS-CoV-2, the vaccine elicits spike protein IgG and not nucleocapsid IgG, a serological profile separating immunization from infection. Accordingly, we did not use values of spike protein IgG to evaluate infection status, as these values would be disturbed by vaccination

[Torill M.F F @torill_fr]

Dette kommer frem om du leser studien De har brukt en metode som skiller mellom vaksinen og smitte
De fleste i studien var vaksinerte study reports that 98.9% of participants were vaccinated and explain(in methods) why they used nucleocapsid igg (not spike) to avoid vaccine confounding,«

SARS-CoV-2, the vaccine elicits spike protein IgG and not nucleocapsid IgG, a serological profile separating immunization from infection. Accordingly, we did not use values of spike protein IgG to evaluate infection status, as these values would be disturbed by vaccination

[Torill M.F F @torill_fr]

Viktig! Jeg har tatt vaksinen. Vaksinen er dyr , burde vært mye rimeligere. Forebygging er lønnsomt, også samfunnsøkonomisk.
Dette er interessant ikke bare når det gjelder Herpes Zoster , men også andre virus og postinfeksiøs senvirkninger, som feks etter covid og kyssesyke.og man ser også sammenhengen med hjerte karsykdommer her
Denne studien er interessant Forskere i Bergen ønsker å se på om virus er årsak til alle de nevrologiske sykdommene de forsker på, inkludert både MS, demens, Parkinson og ALS. Hvis virus viser seg å spille en rolle, gjør det sykdommene lettere å angripe.

https://t.co/JP75im4cXZ

[Torill M.F F @torill_fr]

@awgaffney @Prebens @AlanLevinovitz @WIRED https://t.co/2gO7Una3Hk

[Dr Elisa Perego @elisaperego78]

Failure to acknowledge the broader, well-documented impact of SARS-CoV-2 infection on the human body is detrimental to advocacy and scientific accuracy. Denying there is vast evidence of biological damage visible through imaging, biopsy and autopsy is pseudoscience.

[Torill M.F F @torill_fr]

@awgaffney @Prebens @AlanLevinovitz @AlanLevinovitz @WIRED This is not something that can be easily fixed by changing your mindset. If it were that easy, we would not have several hundred million
with long covid.
This ⬇️‼️

[Dr Elisa Perego @elisaperego78]

We must be crystal clear about a few points. Long Covid is not a "poorly understood neuroimmune disorder" with no biomarkers. It is a widely studied biological medical condition. Pathology spanning basically all body systems is well documented, with damage in situ across organs

[davidtuller @davidtuller1]

Alan, you say it would be "great to know" more about these approaches. Then why did you fail to mention the fraudulent trial of the Lightning Process, with a 3,000-word correction, that was published in a Lancet journal? And the Gupta trial in Scandinavia with null early results?

[Danielle Beckman @DaniBeckman]

Hey @WIRED, how about interviewing neuroscientists who are actually studying the brains of people with #LongCovid? I'm available, and so are many others in the field.

"When the brain gets stuck in a feedback loop of fight or flight" What does that even mean? #Pseudoscience

[Mike Hoerger, PhD MSCR MBA @michael_hoerger]

As a clinical health psychologist who has written >20 papers on COVID, I would emphasize 4 facts:
1) Long COVID is not a psychological diagnosis nor manifestation of a psychological condition
2) Billions of dollars need to be invested in biomedical treatments and preventives, and that money is not being invested because of wealthy short-term interests, which prop up various narratives, including in the media
3) Behavioral interventions can help with infection/reinfection prevention (e.g., COVI-CAN pilot) and stress/coping support (gaslighting/ostracism as huge issues), but these are not cures, and the same interventions are relevant to people with cancer, organ failure, immunocompromising conditions, etc.
4) Many psychological/behavioral "treatments" for Long COVID are directly harmful to patients and are indirectly harmful to society by incorrectly framing the issues

I would consider these issues obvious in summer 2020.

Articles like this should not be written in 2026, but it is a consequences of cultural evolution, or organizational selection by consequences. The organizations that write puff pieces propping up pseudoscience get the gold, while truth tellers do not. It would be useful to examine the organizational practices at WIRED that led to the incentive systems that allowed this piece to manifest.

[Torill M.F F @torill_fr]

There is always someone who will get well on their own, or miraculously get better. You can believe in angels or a medallion from @ShamanDurek that will cure long covid, but this is not science. Studies with change your mindsets methods have also made many sicker. With over half a million peer-reviewed scientific articles, the evidence base is very large, so you can put this nonsense away.

[Torill M.F F @torill_fr]

There is always someone who will get well on their own, or miraculously get better. You can believe in angels or a medallion from @ShamanDurek that will cure long covid, but this is not science. Studies with change your mindsets methods have also made many sicker. With over half a million peer-reviewed scientific articles, the evidence base is very large, and you can put this nonsense away.

[S Blitshteyn MD, FAAN, FANA, Dysautonomia Clinic @dysclinic]

There are always going to be people "with whatever" who miraculously recovered "from whatever" -- this is not science, it's anecdotes. We have scientific method to help us sort things out and inform the media and the public on what works, why and for whom. 🧠🩺

[Dr Elisa Perego @elisaperego78]

The concept of Long Covid covers a broad range of biological abnormalities not because it's "vague", as some erroneously claim. It does so because SARS-CoV-2 infection can affect the whole body from "head to toe". Multi-system pathology is an hallmark of the disease

[Torill M.F F @torill_fr]

Når vi gjør ingen ting er dette satt på spissen . Vi tester ikke. Vi teller ikke , vi mangler ICD kode i primærhelsetjenesten. For long covid Kunnskapsministeren snakker om at foreldre er for ettergivende. Her er nok mørketallene veldig store, og disse barna lider i det stille. Folk flest anser covid som ufarlig for barn og unge. Vi bør oppfordre til å teste, òg luftrensere inn i skole og offentlige bygg! Det er en god start

[Torill M.F F @torill_fr]

Ja det er jeg klar over, men her hjemme er vi tyst når det kommer til langtidsvirkninger.

- Vi anerkjenner ikke pediatrisk long covid
- Vi har ingen nasjonal plan eller strategi for long covid
- Vi har ingen tverrfaglig tilnærming til long covid
- Vi har ikke et dedikert helsebudsjett til long covid
- Vi har ikke øremerket midler til forskning på long covidI stedet bevilges enorme summer til forskning på "tenk-deg-frisk"
- Vi er et av kun tre land som ikke har noen kliniske retningslinjer for behandling av long covid ( i følge rapporten fra OECD
Sykefraværet øker . Regjeringen skylder på holdninger, og @jcvestre ber fastlegene sykemelde mindre Fraværet i skolen øker, Kari Nessa Nortun , snakker om at foreldrene er for ettergivende .
FHI snakket om at gjentatte infeksjoner er bra for oss , og at det ikke er gode holdepunkt for å si at covid skader hjertet og blodomløpet, Vi har personer her hjemme som gjør veldig en god jobb , som feks @ArSoraas @GANyborg @raubreywhite det er riktig, men fra helsemyndighetene er det ikke mye å hente