@thyroiddriven WH Bates
He basically proposed that the the changes is visual axis length which produce refractive error are driven by the extra ocular muscles squeezing the eyeball
Also don't get lost between all the social media bs around his ideas - go read his book first
@thyroiddriven ... get rid of the strain
The retina is also one of the most mitochondria dense tissues- although I think that's less of an issue
A large part of strain is mental in origin and driven by lack of energy in the brain. The other portion is the easily fatigued muscles
@thyroiddriven If we combine Peat's metabolic ideas with Bates ideas on strain/extraoccular muscles there'd definitely an eyemaxxing protocol
Bates primary hypothesis was that strain in the extraoccular muscles cause refractive error
Peat ideas about metabolism & relaxation fill in how to...
@CowsEatGrassBlg Diet is otherwise nutritious
Actually inspired by the sugar for therapy book - this is the easiest way for me to get that much sugar in - only issue is that it seems to be too much water
And it got me wondering with the whole prl /hyponatremia thing
@aestheticprimal I've been screaming this from the rooftops for a while now
Same like how tsh rises w a bad thyroid
Low glucose/ fasting negatively signal igf1 , and so with a drop in negative feedback the gh rises
@metabolic_print I'd also add t if you're constantly running on Cortisol & adrenaline- carbs switch those systems off
What you perceived as energy is tired and wired, but not having enough energy to rest - so as soon as there's some fuel the body wants to rest
@AlphaGels When you say 5-10 mg dose this means you apply 5mg total on the skin, or apply enough for 5-10mg to get into blood(what % penetrative on average do topical stuff have? 5%?)
@doctormorphh Low insulin/hypoglycemia are direct inhibitors of igf-1 and stimulants of hgh which plays a role in bring bs back up
Additionally, a large part of the bone growth depends on autocrine/paracrine igf-1 and metabolic efficiency in those cells
@doctormorphh This is probably because gh doesn't directly mediate growth
Just like tsh doesn't mediate the thyroid response, or acth for cortisol.
The actual active factor is igf-1
Also - the rise in hgh during fasting is the same as tsh rising in hypothyroidism.