I’ve been quiet for the past two years for a reason. I found a fatal mistake in one of the foundational pillars of modern medicine—autoimmunity. When it fully collapsed, it rapidly took down every other supporting pillar that has propped up our understanding of health, the consequences directly affecting each of us. The rising sickness swamping society today has a straightforward explanation. So after 25 months, I’m back—and angry.
Autoimmune diseases have been an enigma ever since scientists hypothesized their existence. Why do they largely impact women? Why in the prime of their lives? Why?
The very concept of the body attacking itself is a cascade of contradictions, defying Darwin. Instead of rates falling, they’ve been increasing steadily with every recent generation. Instead of symptoms fading, inflammation and disability rage. Mental illness, metabolic disorders, and cancer are frequent partners. Autoimmunity would appear to be an unsolvable mystery of the ages, destined to vex scientists for eternity.
Or not.
Seventy years ago, when frustrated scientists kept finding unusual antibodies accompanying a swath of diseases—yet never finding an attacker—they came to the fateful conclusion that the immune system must be turning on itself. Fateful, because there is a telling maxim in science: Absence of evidence is not evidence of absence. There was a deviously hidden invader—and it still lurks within most of us.
The evidence is clear, overwhelming, indisputable. But blink, and you’d miss it. Everyone blinked. Everyone missed it.
There is only one organism on the planet that can produce the unique, signature characteristics of “autoimmunity.” A bacterial infection today known as Lyme disease—vindication for everyone who has been gaslit and disbelieved. The evidence lies in a trace mineral required for the spirochete to survive and cause disease: manganese.
The damning clue is in the distinctive interplay between iron and manganese. As manganese levels rise, iron levels fall and vice versa. In childhood, boys and girls have similar levels of manganese. But once young women begin their monthly period, and start routinely losing blood, iron levels dip and manganese levels spike. The enduring mystery—why women, why in their prime—cleanly answered.
We've unknowingly been trying to treat Lyme for decades. The bacteria derive energy from glucose, essentially carbohydrates. Every intervention that works across an inexplicable range of conditions—from GLP-1s to carb-limiting diets to exercise—works because it restricts the bacteria’s food supply. They all effectively lower blood sugar. That's why exercise helps prevent the recurrence of cancer and why ketogenic diets aid mental health conditions. The bewildering success of the newest weight-loss drugs—improving 175 health conditions and counting—isn't bewildering at all. It’s starving out the bacterial infection.
Borrelia's appetite for manganese is the elusive missing puzzle piece. The body absorbs manganese from food, but several common substances block that absorption: iron, fiber, calcium, tetracycline antibiotics, and tannins. What’s rich in tannins? Coffee, tea, dark chocolate, and red wine—their inexplicable health benefits now fully explainable. (Now it makes sense why cranberry juice, chock-full of tannins, is often recommended for preventing UTIs and why a new study found that drinking coffee reduces the risk of MS by nearly 50%.)
Once you realize that virtually everything we have been doing has been to control Lyme disease—our drugs primarily tamp down inflammation brought on by our own immune system desperately trying to fight the bacterial infection—everything crystallizes.
On May 29, the HHS issued a national call to action to solve the chronic disease crisis disproportionately affecting women. I’ve written an open letter to the global scientific community—merely correcting a single mistake fully rewrites modern medicine. I recommend you sit down before reading.
So many health issues share the same symptoms. What are the odds this is all just one massive coincidence?
*Sit Down Before Reading*, a memoir of misdiagnosis that unravels medicine’s greatest mysteries, continues with Chapter 47: Little Sparks. https://t.co/yTZAHkED1Q
A vexing medical mystery affecting 65 million+ finally has an answer.
*Sit Down Before Reading*, a memoir of misdiagnosis that unravels medicine’s greatest mysteries, continues with Chapter 46: The Smoking Flamethrower.
https://t.co/40inTeuAGW
The entire theory of autoimmune disease threatens to implode for a spectacularly mundane reason.
*Sit Down Before Reading*, a memoir of misdiagnosis that unravels medicine’s greatest mysteries, continues with Chapter 36, Part 9: The Immaculate Storm.
https://t.co/in0tbwCCFN
Mental illness is in the crosshairs as a pillar of medicine unexpectedly sways. *Sit Down Before Reading*, a memoir of misdiagnosis that unravels medicine’s greatest mysteries, continues with Chapter 36, Part 8: Brian. https://t.co/otXihyscTm
Something is decidedly amiss in the theory of autoimmune disease. *Sit Down Before Reading*, a memoir of misdiagnosis that unravels medicine’s greatest mysteries, continues.
https://t.co/mBADYnrDze
What if Lyme disease really is as widespread and calamitous as sky-is-falling alarmists have warned? *Sit Down Before Reading*, a memoir of misdiagnosis that unravels medicine’s greatest mysteries, continues. https://t.co/zQZpOJ57Kk
Treatments for Lyme disease may lurk in plain sight. But you'd never guess where. *Sit Down Before Reading*, a memoir of misdiagnosis that unravels medicine’s greatest mysteries, continues with Chapter 36, Part 5: Antidotes.
https://t.co/RW09s80xL8
Could overlooked diagnostic markers reveal an MS misdiagnosis? If the devil is in the details, we take the deepest of dives into potential clues that just might be able to separate Lyme disease from multiple sclerosis and other similar conditions.
https://t.co/g56caUptDa
One of the most accepted theories in multiple sclerosis implodes under the crushing weight of logic. Will any neurologist still defend it?
https://t.co/Y62kmFKRy4
Was the most famous disabled American misdiagnosed? Scientists now believe #FDR never had polio, that he had Guillain-Barré syndrome. Just one problem: he didn't have GBS, either. The case study: https://t.co/z9AFhruUNt
Everyone knows that antibiotics aren’t supposed to change the trajectory of multiple sclerosis. So what happens when they shockingly do? *Sit Down Before Reading*, a memoir of misdiagnosis that unravels medicine’s greatest mysteries, continues.
https://t.co/KOtpH5GysS
The final chapter of *Sit Down Before Reading*, a memoir of misdiagnosis that unravels medicine's greatest mysteries—begins releasing 1/24/24. I recommend revisiting the warning I gave readers many months ago. THIS IS GOING TO BE HARD.
https://t.co/HTOH1OsmXX
Could you be misdiagnosed with #MultipleSclerosis? See if you have one of these telltale red flags in your journey with the chronic disease. *Sit Down Before Reading*, a memoir of misdiagnosis, continues.
https://t.co/7XogJOpUta
Everything that could go wrong went wrong. Safeguards catastrophically failed as faulty #multiplesclerosis research was crowned settled science. *Sit Down Before Reading*, a memoir of misdiagnosis, continues with Chapter 34: A Perfect Storm.
https://t.co/N7rm3sy47I
For researchers studying multiple sclerosis, they’ve unwittingly fallen into a trap of their own making, admitting that they “cannot exclude the possibility that MS is an infectious disease." *Sit Down Before Reading*, a memoir of misdiagnosis, continues.
https://t.co/LpUNa5de75
The groundbreaking speech in Miami had every potential to change the course of multiple sclerosis… if only a single researcher had managed to put the puzzle pieces together. *Sit Down Before Reading*, a memoir of misdiagnosis, continues. https://t.co/zPouFnwrIo
Crap. Proving that Lyme disease was getting frequently misdiagnosed as multiple sclerosis required math. Phew, good thing I can add. *Sit Down Before Reading*, a memoir of misdiagnosis that unravels MS’s greatest mysteries, continues with Chapter 31: Math.
https://t.co/X1AGflr8kL