A. SASSI (Ph.D., MSc, Eng.)

I'm a #Doctor / #Renal_Physiology / #Electrophysiologist

[A. SASSI (Ph.D., MSc, Eng.) @Ali_SASSI]

12 Qualities of Leaders Who Lead with Heart & Mind
https://t.co/IRRIDEL2fy 🐸

[A. SASSI (Ph.D., MSc, Eng.) @Ali_SASSI]

Happy to share our latest publication in Frontiers in Physiology. @FrontPhysiol
#Nephrology #KidneyResearch #RenalPhysiology #Claudin #Macula_densa #Frontiers
#Frontiers_in_physiology
https://t.co/uGxXWpamCJ…
https://t.co/16krD507WO

[Frontiers - Physiology @FrontPhysiol]

New Research: Claudin-3 is expressed at the macula densa and is associated with altered glomerular filtration in claudin-3 knockout mice https://t.co/24Zj9aZ1a0 #FrontiersIn #Physiology

[A. SASSI (Ph.D., MSc, Eng.) @Ali_SASSI]

Our new review explores how claudins regulate paracellular transport along the nephron, enabling energy-efficient ion & water reabsorption and shaping kidney pathophysiology:
#Current_Opinion_in_Nephrology_and_Hypertension https://t.co/sffxHjcXoq

[A. SASSI (Ph.D., MSc, Eng.) @Ali_SASSI]

Excited to share our new paper in Acta Physiologica!
We reveal how claudin-3 contributes to epithelial barrier function and tissue organization. @Acta_Physiol @UNIGEnews https://t.co/B8lQDZEfa2

[biorxiv_physio @biorxiv_physio]

Low-salt diet induces claudin-3 expression and drives adaptive changes in collecting duct of claudin-3-deficient mice https://t.co/Xq1ryOLKQE #biorxiv_physio

[A. SASSI (Ph.D., MSc, Eng.) @Ali_SASSI]

Proud to share an article I co-authored with my undergraduate biomedical students at the Faculty of Medicine of Geneva. @revmedsuisse
https://t.co/aDrBzQQtyG

[A. SASSI (Ph.D., MSc, Eng.) @Ali_SASSI]

Please find our latest commentary published in Pflügers Archiv - European Journal of Physiology:
https://t.co/OtlfePzJWS

[A. SASSI (Ph.D., MSc, Eng.) @Ali_SASSI]

Magnificent!

https://t.co/tW29gd0Q6X

[A. SASSI (Ph.D., MSc, Eng.) @Ali_SASSI]

Knock out of CGN and CGNL1 in MDCK Cells Affects Claudin-2 but Has a Minor Impact on Tight Junction Barrier Function #cingulin #paracingulin #sodium #claudin-2 https://t.co/Zy4jqvLDzv #mdpicells via @Cells_MDPI

[Alan Yu🇭🇰 @AlanYuNeph]

It's not all about transcellular transport!

🔥Come hear what's new in paracellular transport from leaders in the field: @todd2_todd @h_dimke @Ali_SASSI #DorotheeGünzel

@ASNKidney Friday 10:30 am W230👇

@BhallaResearch & I have honor to chair session

[A. SASSI (Ph.D., MSc, Eng.) @Ali_SASSI]

@LucileFigueres @JASN_News @et_rein @CR2TI_Nantes Very nice paper Lucile Congrats!!!

[bioRxiv @biorxivpreprint]

Increased severity of chronic kidney disease in response to high potassium intake is dependent on mineralocorticoid receptor activation https://t.co/AWsFGJ4ZL0 #bioRxiv

[Pflugers Archiv - European Journal of Physiology @PflugersArchiv]

Working on #kidney physiology? Then this #review about #renal water transport in health and disease will catch your interest. @Ali_SASSI @Hopitaux_unige #aquaporin #openaccess https://t.co/CxmJpV44tL

[A. SASSI (Ph.D., MSc, Eng.) @Ali_SASSI]

Increased severity of chronic kidney disease in response to high potassium intake is dependent on mineralocorticoid receptor activation https://t.co/0nQXwAOyJd

[biorxiv_physio @biorxiv_physio]

Increased severity of chronic kidney disease in response to high potassium intake is dependent on mineralocorticoid receptor activation https://t.co/Yhy8LtfUlv #biorxiv_physio

[A. SASSI (Ph.D., MSc, Eng.) @Ali_SASSI]

@NahidTabibzadeh Thank you Nahid.

[A. SASSI (Ph.D., MSc, Eng.) @Ali_SASSI]

Please find our last publication about renal water transport in health and disease. #Renal_physiology #Aquaporin #Renal_transport #Kidney
https://t.co/5HXripomGv

[A. SASSI (Ph.D., MSc, Eng.) @Ali_SASSI]

@audard15 @et_rein Data are exclusively from FSGS cases.
If needed, we can perform similar IF experiment on MCD and membranous nephropathy. Thank you for your question.

[A. SASSI (Ph.D., MSc, Eng.) @Ali_SASSI]

Claudin-4, a core component of the tight-junctional complex along the collecting system, is induced in nephrotic syndrome https://t.co/fxlaY0dc4O

[bioRxiv @biorxivpreprint]

Claudin-4, a core component of the tight-junctional complex along the collecting system, is induced in nephrotic syndrome https://t.co/XJZsfwQOy5 #bioRxiv