New publication🔥💃
Really grateful to had the chance to participate in this study in collaboration with the Heinrich Lab and led by @DebbieYK1 .
https://t.co/Nbk5REcfwj
We thank @FrontiersIn, the reviewers and editor for their valuable comments on improving the manuscript. I'm very proud to have now published the final chapter of my PhD thesis!
#neuroprotection#Epo#CRLF3#humanipsc
Check out our newest study on the functions of the cytokine receptor CRLF3 in protection of hiPSC-derived neurons! We identified CRLF3 as Epo and EV-3 responsive, eliciting neuroprotection via differential expression of pro- and anti apoptotic genes. https://t.co/a3v1IexSQz
Besides the fact, that this is the first receptor identified to elicit neuroprotective functions of EV-3, this work will hopefully also contribute to the generation of Epo derivates, which can safely be applied in clinics for treatment of neurodegeneration diseases.
We're happy to announce that NeuroDoWo is back next year, this time in in beautiful Konstanz!🥳🥳Save the dates: 2nd-5th May 2023, for another wonderful doctoral students conference!🤓And watch this space for more information! 👀🧐
On my way to #SfN22 with a short stop in San Francisco first! Hoping to meet many amazing people from the #neuroscience community! Come visit me Sunday between 3 and 4 pm at poster 201.11 if you are interested in cytokine-mediated neuroprotection of human iPSC-derived neurons!!
New paper on the regulation of cytokine-mediated #neuroprotection out now! We teamed up with @BartGeurten for live/dead analysis via AI and show the regulation of pro-apoptotic AChE by Epo (human cytokine) and CRLF3 (endo. receptor) in beetles and locusts! https://t.co/qft0YwaFYv
Dear Drosophila Community,
Our lab has a permanent position open for a German speaking scientist interested in Drosophila sensory biology and teaching (very important!!). Full pay and a great lab in beautiful Göttingen await you!
Really excited to share our preprint together with @PoloAug on neuroprotection in human iPSC-derived neurons: https://t.co/S8lrRCk7aT
We identified CRLF3 as a neuroprotective cytokine receptor activated by an Epo variant. We also characterized the activated intracellular pathways
Super excited to share our latest preprint on the regulation of pro-apoptotic acetylcholinesterase by Epo/CRLF3 interaction. This is how neurons are saved (partially)! https://t.co/J5Fhg07ANW
Verschuldigung, ich war die ganze Zeit der Auffassung, dass die angesprochenen Bevölkerungsgruppen keine Wahl haben, während die neue "marginalisierte Gruppe" sehr wohl eine hat. Könnte das der Grund sein, weshalb sie als Gefahr gesehen und betitelt wird? Just asking..