@ecgandrhythmRoe You r right, these are probably ectopic beats from right upper PV.
The ECG is somehow strange: sinus bradycardia of 35/min but QT is about 400ms! ectopic beats are very rapid (200-240ms) but have a relatively long (400ms) coupling interval to the sinus beat...
@MaruanCarlos Most probably continuous prolongation of AVp interval due to an algorithm such as VIP, search AV+,... to promote intrinsic AV conduction. Due to lack of intrinsic conduction the device switches back to a shorter AVp interval.
@Hiren_PatelMD@willyhfrick@syamkumarmd I thought obout TWOS as well, especially because there is no obvious trigger for the tachy (such as a PVC,...), but TWOS classically shows the tram track appearance. without EGMs, I prefered just to give an evidence based explanation😁.
@willyhfrick@syamkumarmd the tachy falls from the beginning in the VF zone, there is an unsuccessful(?) ATP while charging and then comes the successful shock.
@ecgandrhythmRoe Most Probably slow VT terminated through a vent. Burst.
2 another things: first the episode Nr. 2840 ist somehow strange!
secondly I think ATP with 220ms(280/min) for almost 20 beats seems very aggressiv for this slow VT. Interestingly we see capture by every burst beat!
@IrmaMDv@ecgandrhythmRoe Interpreting PM ECGs can be very challenging, especially if the pacemaker cofigs are unknown. Other explanations are probably possible too. by the way, the sweep rate of the ECG must be 25 mm/s?
@ecgandrhythmRoe@IrmaMDv It is most probably not a VT. I think The PVC induces an antegrade AV conduction prolongation/block, the PM begin to stimulate in RV and most probably induces a PMT. PMT is terminated due to retrograde AV conduction prolongation. Anyway i think it is PM-induced.
Deep dive on #EARLYTAVR from ##TCT2024
Lots already said & discussed
It takes time to read the paper, read the supplementary appendix, analyse the results, think about them etc!
Some thoughts...🧵