Optimized 3-T MR neurography can be diagnostic even near metal. Check out persistent radial nerve palsy after humeral shaft fixation: 1.5-T MRI was nondiagnostic, while 3-T MR neurography revealed nerve deviation and entrapment, confirmed surgically. https://t.co/EpSn1Io69H
1/Wish that your knowledge of autoimmune encephalitis was automatic?
Do you feel in limbo when it comes to the causes of limbic encephalitis?
Do you know the patterns of autoimmune encephalitis?
Here’s a thread with some hints to help you figure it all out!
@OGdukeneurosurg The following are common Neuroradiological signs of Progressive Supranuclear Palsy (PSP) and related disorders, most often Parkinsonian Disorders due to pathological changes in the Basal Ganglia & Structural Abnormalities of the Midbrain & Cerebral Peduncles⬇️
Time to go for the jugular when it comes to carotid stenosis!
Degree of stenosis isn’t all that matters
Here’s cheat sheet you NEED to remember the risky features of carotid plaques
Read on for the newest @theAJNR SCANtatistic on carotid stenosis:
https://t.co/SieEdSWFrc
NASCET isn’t the end of what you need to know—it’s just the beginning.
In addition to degree of stenosis, there are features of plaques on imaging that suggest they are prone to rupture & cause strokes.
You can remember the high-risk features by remembering that atherosclerosis is influenced by a bad diet—so the features you get when you have a bad diet are the same features vulnerable plaques will show on imaging!
High risk features:
🔸Size
You get increased size w/outward remodeling of your belly w/a bad diet. Same w/vulnerable plaques—larger plaques with outward remodeling are riskier
🔸Irregularity
If you eat a poor diet, you will get constipated & irregular. Same w/vulnerable plaques—irregular plaques are at higher risk
🔸Ulceration
Obesity is associated with ulcers. Same w/vulnerable plaques—ulcerated plaques are more vulnerable
🔸Lipid necrotic core
If you eat a lot, you get more belly fat or lipid in your core. Same w/vulnerable plaques—plaques with fatty deposits in their core are more vulnerable
🔸Loss of fibrous cap
If you eat a lot, your belt can’t hold your fat in anymore & will break. Same w/vulnerable plaques—loss of the fibrous cap holding in the lipid core is a high-risk feature.
🔸Neovascularity
Poor diet leads to constipation & hemorrhoids, a sort of neovascularity. Same w/vulnerable plaques—increased vasculature in the plaque & enhancement is a high-risk feature.
🔸Hemorrhage
Poor diet & hemorrhoids leads to bleeding. Same w/vulnerable plaques—intraplaque hemorrhage is associated with increased chance of rupture
More and more research like the research in this month’s @theAJNR have shown that looking at only degree of stenosis & symptomatology are not enough
Now you know all the imaging features that suggest a high-risk plaque beyond the degree of stenosis. Don’t narrow your focus when it comes to carotid narrowing!
1/Don’t be narrow minded when it comes to carotid narrowing!
Everyone know the NASCET criteria for carotid stenosis
But are you stuck in the 1990s when it comes to what you know??
Get up to date w/the latest @theAJNR SCANtastic on carotid stenosis:
https://t.co/RlJa9J1VYQ
Just because it’s called SMALL vessel disease doesn’t mean it doesn’t have a BIG impact!
Small vessel disease (SVD) is a BIG contributor to vascular dementia, along w/large vessel cortical infarcts
Do YOU know all the faces of small vessel disease?
Common imaging markers of SVD include:
--White matter hyperintensities (WMHs)
--Lacunes
--Enlarged perivascular spaces
--Microbleeds
--Small subcortical infarcts
--Brain atrophy
But what you see isn’t always what you get!
Even in patients w/similar degrees of SVD, clinical symptoms can be very variable.
Variability exists because of:
--Perilesional penumbra
--Remote effects from disruption of brain connectivity
--Differing brain reserve & compensatory mechanisms
So remember, imaging findings of SMALL vessel disease are just the tip of a very BIG iceberg
This rPoster from day 1 #Radiopaedia2024 by Dr Khan was another favourite from the many high quality submissions, on imaging of the hippocampus. Very pragmatic approach of dividing pathologies into non-nodular and nodular, before subdividing further.