Hello Twitter, starting the 2nd of January i am starting to offer Psychiatric consultations at my clinic in Zalka, Saint Joseph 49 tour, 3rd floor, behind Al Arez Hospital. For appointments, please call 70/496819 #Psychiatry#mentalhealth
We had a patient who came for psychotic symptoms. Was labelled psychotic, started on antipsychotics, his state got worse and he became catatonic. After days of meticulous history taking, we discovered cognitive deficits at the beginning of his suffering, epileptic seizures (1).
“We are not claiming that we are trying to fix some medical causes behind these syndroms but we are making a practical and pragmatic claim about being able to help with certain medical interventions in order to alleviate suffering”.
“When we encounter states of suffering… and they are amenable to be described in medical clinical terms and we have clinical ways of helping some people with that then that becomes bona fide medical condition. The practical approach is helping with available tools”.
I am on the Thinking Mind podcast with Anya Borissova, talking about various conceptual and critical issues around psychiatric diagnosis and classification.
https://t.co/9tnlU9wugd
By shifting emotional processing, we mean not attributing negative value to neutral stimuli like in depression. Neuroplasticity improves when BDNF does. In addition, Serotonine is also linked to less neuroticism and impulsive behaviors.
Anti-depressants work by shifting emotional processing, improving neuro plasticity and reducing neurotic features. Serotonine as a NT is the cornerstone of these processes. The reductionist theory of “correcting a chemical imbalance” by SSRIs has not a scientific base.
It was a pleasure to have a conversation with @DavidPuder as a guest on his top-rated Psychiatry & Psychotherapy Podcast.
Ep 235: The Serotonin Hypothesis: Controversies and Nuance with Awais Aftab, MD
(Thanks to @NTFabiano for connecting us!)
https://t.co/DHKYMqQrxV
“ If you start lithium early in the course of bipolar your patient has a chance of staying well longer, living longer, and – at a biological level, you might undo some of the mechanisms that cause bipolar disorder and prevent some of the neurodegeneration that worsens its course”
In conclusion, Dr.Phelps says : “With the spectrum approach, the diagnostic question changes from “could this depression be bipolar?” to “how bipolar is it?”. If bipolarity is suspected , we should avoid AD (Partial or no response and may cause mixed features/rapid cycling). (7)
This discussion emphasizes on the importance of considering Bipolar Spectrum disorder as a differential diagnosis for every depressive episode. Ruling out mania/hypomania is not sufficient to confirm that Bipolar Disorder is not the diagnosis. (1)
The 2ndG antipsychotic induced metabolic syndrome in mood disorders should not be neglected especially with the overprescription of antipsychotics in this category of patients. Antidepressants can also cause weight gain, sexual SE and in certain cases severe withdrawal signs. (6)
Up to 90% of patients with ADHD experience at least one comorbidity—ranging from anxiety to substance use disorders.
These overlaps complicate diagnosis and treatment, making clinicians need to recognise and address them effectively.
Here are 10 key ADHD comorbidities and evidence-based strategies to navigate them 👇🧵
We had a patient who came for psychotic symptoms. Was labelled psychotic, started on antipsychotics, his state got worse and he became catatonic. After days of meticulous history taking, we discovered cognitive deficits at the beginning of his suffering, epileptic seizures (1).
@kristinaEBP Step 1: Find the true diagnosis in a record time.
Step 2: Plasmapheresis and corticosteroids.
Step 3: The patient who was suffering from the ENCEPHALITIS and not medications, gets better and his life is saved.
:)