Dr. Nocopes

Whatever is required.

[Dr. Nocopes @NoCopesX]

penis peptides already exists

• vesugen
• klow
• andractim
• tadalafil
• lapump

[BowTiedYukon @BowTiedYukon]

We’re 3 months out from 2027 & there’s no penis peptide?

Enough of fucking hair, nails, skin, focus, energy & mitochondria bullshit. We want bigger dicks

We don’t need peptides for boners or bigger loads. We got those already. I’m talking 10mg vials of 10 units a day for bigger dicks

It’s time for China, The Gray Market & Big Pharma to come together & make the DLP-1

2027 could be our year

[Dr. Nocopes @NoCopesX]

@butcher_xbt every single side effect you listed is already countered in my post.

[Dr. Nocopes @NoCopesX]

best first roid cycle with zero side effects

• test
• tren
• nebivolol + telmisartan
• fluvoxamine
• zone 2 + clean diet
• lemborexant
• reta and exemestane

that's it

what you will get

• chud to chad in 6 months
• better mood, cognition, alrtness, motivation, ambition, reduced anxiety and confidence
• whole life would change
• and even after coming off brain and baseline personality will change permanently especially because of fluvoxamine plasticity
• can mog 90% of people
• will be easier to get rich in future
• and many other things

50mg testosterone eod (~175mg/week) saturates skeletal muscle androgen receptors (ar) to drive satellite cell proliferation, myonuclear addition, and mtor-mediated protein synthesis while maintaining physiological estradiol and dht conversion without serum peaks and valleys.

5mg trenbolone acetate eod (~17.5mg/week) binds the nuclear ar with 3x the affinity of testosterone while competitively blocking the glucocorticoid receptor (gr) to halt cortisol-induced catabolism. centrally, it downregulates dat and net clearance while sensitizing post-synaptic d1/d2 receptors and orexin neurons for relentless executive drive and aggression.

25–50mg fluvoxamine agonizes intracellular sigma-1 receptors (sig-1r, ki = 36 nm) to shield striatal dopamine neurons from oxidative stress while surging bdnf, ngf, and allopregnanolone (via 3α-hsd). this rewires prefrontal dendritic spine architecture and silences amygdala anxiety, locking in permanent structural neuroplasticity and high-status personality traits that persist long after discontinuing the androgens.

telmisartan blocks at1 receptors and agonizes ppar-γ while nebivolol selectively antagonizes β1-receptors and activates endothelial enos to surge nitric oxide. paired with daily fasted zone 2 cardio (ampk/pgc-1α activation) and lemborexant (dual ox1r/ox2r antagonism), resting heart rate, blood pressure, and slow-wave glymphatic sleep remain completely locked down.

if starting at high body fat, retatrutide (triple glp-1/gip/gcgr agonist) forces hepatic beta-oxidation and crushes visceral fat, while exemestane irreversibly inactivates excess adipose aromatase (cyp19a1) to prevent estrogenic water retention and gynecomastia.

the variable is pairing micro-dosed ar/gr synergy with sigma-1 neuroplasticity and endothelial shielding to permanently upgrade both myonuclear density and prefrontal executive circuitry with zero systemic toxicity.

[Dr. Nocopes @NoCopesX]

here's how to use tren with zero side effects

• 5mg mwf - 15mg/week
• 25mg daily fluvoxamine with dinner

that's it, what you get in return ?

• more orexin
• better mood
• better dopamine sensitivity
• more motivation
• almost zero catabolism if on reta and heavy cut
• better work capacity
• insane fat oxidation and nitrogen retention
• would only need 5-6 hour sleep
• so much more

• pair with daily 30 min zone 2 fasted cardio and 90% clean diet

• baseline vitamins, minerals, antioxidants should be maxxed out.

5mg trenbolone mwf (15mg/week) saturates skeletal muscle androgen receptors (ar) and competitively blocks glucocorticoid receptors (gr) without crossing the systemic toxicity threshold.

this halts ubiquitin-proteasome muscle breakdown on a heavy retatrutide cut while forcing positive nitrogen retention and beta-adrenergic lipolysis.

central ar activation downregulates dat and net clearance while upregulating lateral hypothalamic orexin-a/b neurons and post-synaptic d1/d2 receptor sensitivity. this locks in relentless catecholaminergic drive, baseline mood elevation, and high-output wakefulness on just 5–6 hours of sleep.

taking 25mg fluvoxamine with dinner agonizes intracellular sigma-1 receptors (sig-1r, ki = 36 nm) and inhibits acid sphingomyelinase (asm) to shield striatal dopamine neurons from oxidative stress, surge bdnf, and lower ceramide-induced inflammation.

fluvoxamine inhibits hepatic cyp1a2 and cyp2c19 to block endogenous melatonin breakdown, multiplying nocturnal pineal and mitochondrial melatonin via pept1/2 transporters.

as the body's apex terminal antioxidant, this elevated melatonin directly scavenges hydroxyl and peroxynitrite radicals while upregulating sod and glutathione peroxidase, neutralizing tren-induced mitochondrial ros, lipid peroxidation, and neuro-cardiorenal toxicity while compressing your sleep requirement into 5–6 hours of hyper-efficient slow-wave glymphatic clearance.

30 minutes of daily fasted zone 2 cardio alongside maxed micronutrient and antioxidant stores activates ampk and pgc-1α, clearing mobilized fatty acids and scavenging trace mitochondrial ros before they accumulate.

[Dr. Nocopes @NoCopesX]

@pawgmaxi maybe low 5ar

[Dr. Nocopes @NoCopesX]

@Biomaxxer0 nope

[Dr. Nocopes @NoCopesX]

"Rosuvastatin" in 2026🤣

[Biomaxxer @Biomaxxer0]

Top 10 steroid ancillaries:
10. Nebivolol
9. TUDCA
8. Rosuvastatin
7. NACET
6. Astaxanthin
5. Ezetimibe
4. Fish Oil EPA/DHA
3. Pentoxifylline
2. SS-31
1. Telmisartan

[Dr. Nocopes @NoCopesX]

@grok @xexexxexexexe @grok what if tyramine foods or drug interactions are taken care of.

[Dr. Nocopes @NoCopesX]

parnate mogs d-meth, adderall or any other stimulant

tranylcypromine (parnate) irreversibly knocks out >90% of mao-a and mao-b, permanently expanding intracellular monoamine pools instead of draining them. at 30mg, it saturates mao inhibition and blocks net reuptake—elevating baseline brain serotonin by ~200–300%, norepinephrine by ~200–300%, and dopamine by ~150–200% with zero oxidative crash.

at 60mg, parnate crosses the concentration threshold into direct dat/net/sert blockade while multiplying endogenous beta-phenylethylamine (β-pea) by over 1000% and trace tryptamines by 500%+. this surges intracellular and synaptic serotonin by 400–500% and dopamine and norepinephrine by 300–400% via clean taar1-mediated efflux.

pure dopamine and norepinephrine without serotonin is a trap that leads to manic hyper-fixation, anxiety, and impulsive task-switching. high serotonergic tone in the orbitofrontal and prefrontal cortex (via 5-ht1a and 5-ht2a) is the master regulator of cognitive flexibility, emotional patience, and delayed gratification—locking down the amygdala so high dopamine translates into calm, methodical, high-output execution instead of tweaker spinning.

total mao-a blockade also prevents the breakdown of endogenous n,n-dmt and inhibits epigenetic lysine-specific demethylase 1 (lsd1/kdm1a). this activates intracellular golgi 5-ht2a receptors and unlocks h3k4 histone methylation to drive continuous bdnf transcription and prefrontal synaptogenesis.

[Dr. Nocopes @NoCopesX]

limitless pill isn’t fiction it’s parnate (tranylcypromine). think adderall, transdermal selegiline and dmt working together in one molecule.

adderall like focus, emsam transdermal selegeline like mao a and b destruction so your nuerotransmitter pool is always full and literal DMT so all the benefits of microdosing intracellular 5ht2a agonism, neuroplasticyty etc.

it reverses net, dat and sert at the same time because of reduction of b-pea and tryptamine breakdown at 60mg dose, if dont want serotonergic effects that much then 30mg is enough

tranylcypromine is a cyclized amphetamine derivative that irreversibly inhibits both MAO-A and MAO-B. this stops the enzymatic degradation of dopamine, norepinephrine, and serotonin to keep vesicular monoamine stores permanently maxed out.

halting MAO-B prevents the rapid breakdown of endogenous trace amines, surging brain levels of beta-phenylethylamine (β-PEA) and tryptamine by over 10-fold. high-density β-PEA and tryptamine activates TAAR1 to phosphorylatedly reverse DAT, NET, and SERT, actively pumping neurotransmitters directly into the synapse.

total MAO-A blockade preserves endogenous N,N-DMT and trace tryptamines from first-pass destruction. these lipophilic molecules cross the neuronal membrane to bind intracellular 5-HT2A receptors on the Golgi apparatus, driving rapid TrkB/mTOR-mediated synaptogenesis and prefrontal dendritic spine growth.

at 30mg, it saturates MAO inhibition while primarily inhibiting NET reuptake for clean catecholaminergic focus without heavy serotonergic saturation. at 60mg, the concentration threshold crosses into direct DAT/SERT blockade and massive trace-amine efflux for full triple-transporter reversal.

the variable is dose-dependent trace amine amplification to combine amphetamine-grade efflux with intracellular 5-HT2A neuroplasticity.

but you will need trazodone, clonidine and lemborexant to sleep because the effects are 24/7.

[Dr. Nocopes @NoCopesX]

@Rands_cr yup

[Dr. Nocopes @NoCopesX]

@BitwisePanic do it

[Dr. Nocopes @NoCopesX]

@BasedBiohacker @gaezpeat @BasedBiohacker tell this pussy to stop lying, those were just two things out of a 10+ compound ancillary list. mf is demonic, also unblock me so i can directly dismantle your copes.

[Dr. Nocopes @NoCopesX]

@BasedBiohacker @gaezpeat @BasedBiohacker tell this pussy to stop lying, those were just two things out of a 10+ compound ancillary list. mf is demonic, also unblock me so i can directly dismantle your copes.

[Dr. Nocopes @NoCopesX]

@BasedBiohacker @gaezpeat tell this pussy if he's going to talk shit about me, unblock me first. i will dismantle him scientifically.

[Dr. Nocopes @NoCopesX]

@Acclivouslight my way

[Dr. Nocopes @NoCopesX]

@anonprofit2 serotonin is good, just use any 5ht2b antagonist if want to be extra safe

[Dr. Nocopes @NoCopesX]

@BasedBiohacker @Biomaxxer0 what if someone told you "do the world a favor and stop writing" when you first started and you actually listened?

[Dr. Nocopes @NoCopesX]

@BasedBiohacker @Biomaxxer0 "we'll see" was just the reply to his "total fucking larp" allegation.

[Dr. Nocopes @NoCopesX]

@Biomaxxer0 @BasedBiohacker we'll see

[Dr. Nocopes @NoCopesX]

@donthefifth yes, but make sure not to push past the 40% sert occupancy dose.

[Dr. Nocopes @NoCopesX]

@milkfluencer what does the anecdotal reports says ?

[Dr. Nocopes @NoCopesX]

kx826 is way better than finasteride and without any side effects

kx-826 (pyrilutamide) is a non-steroidal topical selective androgen receptor antagonist with an extreme binding affinity (ic50 = 0.28 nm)—roughly 4x stronger than dht itself. it competitively locks onto the ligand-binding domain of the dermal papilla ar, blocking dht, free testosterone, and synthetic dht derivatives from translocating into the nucleus to trigger tgf-β2 and dickkopf-1 (dkk-1) hair apoptosis.

unlike systemic 5ar inhibitors, kx-826 acts strictly at the follicular receptor and rapidly metabolizes upon hitting peripheral blood into inactive metabolites with negligible systemic accumulation. serum dht, 5α-reductase activity, hepatic clearance, and central neurosteroid synthesis remain 100% untouched.

finasteride irreversibly inhibits 5α-reductase (5ar), blocking the systemic conversion of testosterone into dht alongside progesterone and deoxycorticosterone into 5α-dhp and 5α-dhdoc. this starves 3α-hsd of its substrates, crashing brain allopregnanolone and thdoc to destroy gabaa tone, nitric oxide synthase (enos/nnos), and dopamine signaling.

blocking 5ar triggers a compensatory feedback surge in follicular scalp testosterone while leaving the androgen receptor (ar) wide open. because testosterone itself binds and activates the follicular ar, finasteride fails to stop miniaturization from high local testosterone or exogenous dht derivatives (masteron, primobolan, etc.).

[Dr. Nocopes @NoCopesX]

just use these two no need for all this extra jestermaxxing

[Dr. Nocopes @NoCopesX]

@milkfluencer what they say