@DrSuneelDhand Cows didn't just fall out of the sky. We domesticated them over 10K years ago. So we didn't have to hunt anymore and have a more steady supply. So eating meat is not unnatural.
And you're right, everyone should make their own choices.
@BenBikmanPhD@iowahawkblog@TuckerGoodrich It also seems that PhDs can be obtained by remembering large lists of facts. Shouldn't it be more about showing understanding?
@CoachDanGo Is that what the modern man considers a vulnerability?
Being in a relationship?
Being in a relationship is a strength, not a vulnerability.
And at some point, the system notices the tension between what sustains and what drags. It stays there for a moment. Long enough for the question beneath the title to echo in a different register.
9/9
Why YOU,.... can't Lose Weight
A number on a scale rises, yet the day didn’t feel different. That mismatch is the first hint that something doesn't make sense. The problem isn’t character. It’s physiology that keeps its own score.
1/9
Reward pathways resist abandoning a pattern that once worked. Even a small shift can feel like breaking an implicit pact between body and brain.
That resistance isn’t loud. It’s subtle and steady, like a hand on the brake.
8/9
@gregmushen I'm trying to figure out the cases where LDL is high, yet no or little plaque, and the cases where LDL is super low, yet still significant plaque. This was an attempt to explain that.
I know there are some issues with this hipothesis, but the core idea I can't refute (yet?)
@gregmushen Grek,
I like your logical approach. You mentioned LDL particles getting stuck, but didn't get into the why of it.
A while back I posted a hypothesis. Could you share your opinion on it?
https://t.co/uFHuF1tXOD
Hypothesis: Mechanical Stress–Driven Intimal Remodeling as a Primary Initiator of Atherosclerosis
Core idea
Atherosclerosis begins not simply from lipid infiltration, but from mechanically driven smooth muscle cell (SMC) migration and extracellular ...
1/10
Pre existing plaque did. The vessels reacted to what they were, not to what circulated.
The rise in LDL was loud. The plaque listened to something else.
13/13
An interpretation of the LMHR study at https://t.co/057pSCFWyo
A look at how extreme LDL behaves when the metabolic context is lean, insulin sensitive, and fat adapted. And why plaque progression in this group did not follow the signal everyone expected.
1/13
The Insight That Sits Beneath the Numbers
A risk marker only predicts well when the system generating it stays the same. Change the terrain and the marker behaves differently. High LDL here did not dictate the path.
12/13