Super happy to see the final version of our study online. What started as a summer project in 2023 culminated into this, wherein we assessed the basis of directional α-Synuclein transfer from neuronal cells to microglia
https://t.co/alYSSxeSvu
Super happy to see the final version of our study online. What started as a summer project in 2023 culminated into this, wherein we assessed the basis of directional α-Synuclein transfer from neuronal cells to microglia
https://t.co/alYSSxeSvu
Happy to share our work (final aspect of my thesis). We tried to understand why neuronal and microglial cells upregulate TNTs in the presence of α-Synuclein aggregates? Turns out inflammation has a major role to play in the process! https://t.co/Vyr3igP6bY
Delighted to share a second part of my Ph.D. work. As a follow-up of our previous observations, we wanted to understand the reason behind directionality of aggregate transfer between neuronal cells and microglia. https://t.co/XFTJWNHnT5
And it's out! The BIC lab presents our new perspective article on "Immune Control of Brain Physiology" - fresh off the press at @NatRevImmunol https://t.co/WgTcbrUsOF
wonderful synergy @_marikovacs@Ali_MoussaSamir and twitterless Amaia Dominguez-Belloso.
Delighted to share a second part of my Ph.D. work. As a follow-up of our previous observations, we wanted to understand the reason behind directionality of aggregate transfer between neuronal cells and microglia. https://t.co/XFTJWNHnT5
Consequently, autophagy flux was inhibited in neuronal cells (and not in microglia). We hypothesized that autophagy dysfunction could be one of the reasons for directional aggregate transfer, and it indeed was.