Top Tweets for #DLL4
Wang et al identify monocyte-derived #Dll4 as a key contributor to chronic #inflammation & a potential biomarker & therapeutic target for #HIV-associated #CV risk. Learn more at https://t.co/39YseXDvHj
@CraigMorrell5
🌟 In new angiogenesis research, Dr. Christina Mitchell and team report that PI(4,5)P2 is critical for β-catenin/#DLL4/NOTCH signaling, which governs tip cell specification during #angiogenesis 🩸.
📓 Available in @ScienceAdvances
📌 https://t.co/dZLb01YXXr
Under the leadership of Drs. Anne Bouloumié and Anaïs Briot, a team of scientists 🧑🔬 conclude that endothelial #DLL4 is an #adipose depot–specific fasting sensor 📡regulating fatty acid fluxes
Learn more in this @atvbahajournals study:
🔎 https://t.co/uAE5mGFkSf
Role of #DLL4 in the adaptation of #adipose tissue #endothelium to regulate #lipid fluxes in response to energy transition #metabolism #vascular #endocytosis #obesity @i2mcT @alexaupetit @ChapoulyC https://t.co/6dphU9fXH5
#Dll4-#SREBP1 signaling in the #retina controls #transcytosis independently of #TightJunction permeability.
Dll4 dysregulation may underlie #hypertension-induced pathological leakage of the #CNS vascular barrier. https://t.co/NEL57VS5MG
#ArticlesInPress Activation of Notch signalling by soluble Dll4 decreases vascular permeability via a cAMP/PKA-dependent pathway https://t.co/3urinT9PB3
@UniofNottingham
#DLL4 #notch #endothelialcells
Using #QuantitativeSingleCellImaging, Nandagopal et al. show that cells use dynamics to discriminate signaling by the #ligands #Dll1 and #Dll4 that induce opposite cell fates during embryonic myogenesis through the same #Notch receptor. Free article: https://t.co/VHiM21XFav
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