Top Tweets for #PathophysiologyPearls
TRAKI: Trauma related AKI ca. 2020 from @NatRevNeph
#Nephpearls #PathophysiologyPearls
π https://t.co/2o86kGUghe
Dialysis Dysequilibrium Syndrome #Nephpearls #PathophysiologyPearls
According to the "reverse urea hypothesis," urea is unable to move freely between the extra- and intracellular spaces in neuronal tissue because of the presence of tight junctions and other components of the blood-brain barrier (BBB).
When urea is rapidly removed from the plasma water during intermittent dialysis, urea molecules are slow to move out of cells, including neurons, and CSF which results in a temporary increase in intracellular urea concentration relative to plasma (it can take up to 12-24 hours for the urea in the brain to equilibrate with blood).
Given that the water movement through aquaporins is about 20 times faster than urea transport, water will move along the concentration gradient generated by the difference in urea concentration, passing into the brain and leading to the development of brain swelling and eventual increase in the intracranial pressure (ICP).
ππΌ https://t.co/F1MzoMXO2c
Dialysis Dysequilibrium Syndrome #Nephpearls #PathophysiologyPearls
According to the "reverse urea hypothesis," urea is unable to move freely between the extra- and intracellular spaces in neuronal tissue because of the presence of tight junctions and other components of the blood-brain barrier (BBB).
When urea is rapidly removed from the plasma water during intermittent dialysis, urea molecules are slow to move out of cells, including neurons, and CSF which results in a temporary increase in intracellular urea concentration relative to plasma (it can take up to 12-24 hours for the urea in the brain to equilibrate with blood).
Given that the water movement through aquaporins is about 20 times faster than urea transport, water will move along the concentration gradient generated by the difference in urea concentration, passing into the brain and leading to the development of brain swelling and eventual increase in the intracranial pressure (ICP).
https://t.co/F1MzoMXO2c
Obesity, Hypertension and CKD from @AlpIkizler #Nephpearls #PathophysiologyPearls
https://t.co/jwU8HLFBas
Nice comprehensive review of the complement pathway (9th Edition) #PathophysiologyPearls #Nephpearls
Nice comprehensive review of the complement pathway (9th Edition) #PathophysiologyPearls #Nephpearls
Nice comprehensive review of the complement pathway #PathophysiologyPearls #Nephpearls
https://t.co/R03Vnr0QSE
The βFour Hitβ IgA Nephropathy (IgAN) Pathogenesis Theory #Nephpearls #PathophysiologyPearls
https://t.co/zsm8WFDie9
Hypertension in patients on maintenance hemodialysis: Pathophysiology ca. 2023 from @American_Heart @KidneyInCVD @NishaKidneyDoc @BakrisGeorge @taraichang @jordy_bc @gchertow @DrWanpen
#Nephpearls #PathophysiologyPearls
https://t.co/tta2iTFc4r
APOL1 associated kidney disease ca. 2022 from @NatRevNeph #Nephpearls #PathophysiologyPearls #NephJC
ππΌ https://t.co/M0nQVfqznn
APOL1 associated kidney disease ca. 2022 from @NatRevNeph #Nephpearls #PathophysiologyPearls #NephJC
ππΌ https://t.co/M0nQVfqznn
Renal Physiology 101: Albumin and its ligands induce expression of inflammatory and fibrogenic mediators resulting in inflammation and fibrosis resulting in loss of renal function as a result of tubular proteinuria #Nephpearls #PathophysiologyPearls
ππΌ https://t.co/FbV9wNsibM
The βFour Hitβ IgA Nephropathy (IgAN) Pathogenesis Theory from @NephUCommunity
#Nephpearls #PathophysiologyPearls #NephJC
π https://t.co/YSTEjulMri
The βFour Hitβ IgA Nephropathy (IgAN) Pathogenesis Theory from @NephUCommunity
#Nephpearls #PathophysiologyPearls #NephJC
π https://t.co/YSTEjumkgQ
The βFour Hitβ IgA Nephropathy (IgAN) Pathogenesis Theory #Nephpearls #PathophysiologyPearls
π https://t.co/YSTEjumkgQ
ELE_PATHO: Case Discussion 117 β Hyporeninemic Hypoaldosteronism
A case about a 64 year old male with Hyporeninemic hypoaldosteronism (type IV renal tubular acidosis). Learn more about the disease through this visual graphic.
#PathophysiologyPearls
@edgarvlermamd
Check out the Visual Graphics created by @USTScience students for our elective class on Pathophysiology
Topic: Thyroid Disease
#PathophysiologyPearls
Check out the Visual Graphics created by @USTScience students for our elective class on Pathophysiology
Topic: Disorders of the Hypothalamus and Pituitary Gland
#PathophysiologyPearls
TTP: ADAMTS 13 deficiency ca. 2002 from @NEJM #Nephpearls #PathophysiologyPearls
ππΌ https://t.co/tMN5viP1R5
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