Top Tweets for #trpv4
Too Much Salt? TRPV4 May Be the Culprit Behind Cardiac Hypertrophy
https://t.co/iloGVbldCn
@tianlang1120
#CardiacHypertrophy #TRPV4 #HighSaltDiet #Autophagy #HeartFailure
虚血性脳卒中後の脳浮腫形成に於るTRPV4-glymphatic系の役割
TRPV4活性化がRhoA/MMP9経路を介しβ-dystroglycanを分解しAQP4脱極性/glymphatics障害
TRPV4阻害薬HC-067047がAQP4極性回復/glymphatic機能改善/脳浮腫軽減/神経予後改善
OA
#glymphatics
#TRPV4
#papers
https://t.co/3LWCjOf08J
TRPV4 on Schwann cells is a potential mediator of mechanically induced pain in oral cancer, check out this article! #TRPV4 #oralcancerpain #Schwanncells https://t.co/rsZaiThTJj
Congrats to Gabi Marushack and @AliSavadipour & team for our latest @Guilak_Lab paper showing that polyunsaturated fatty acids alter cell membrane properties & inhibit chondrocyte #mechanotransduction by #Piezo1 and #TRPV4. https://t.co/1SQrx7qjZl
Congrats to our postdoc Yu Seon Kim & team for developing a new way to tune mechanogenetic gene circuits using mutations in the mechanosensory channel #TRPV4!
Part of the Special Issue of @TissueEngin in honor of Dr. Kyriacos Athanasiou @KA_Athanasiou! https://t.co/GpBYGpQoR6
Studying mice with #TRPV4 mutations, scientists show that the mutations lead to progressive neurological symptoms linked to breakdown of the blood-spinal cord barrier, a hallmark of many #neurodegenerative disorders. @ScienceTM https://t.co/XQmCJnTTMq
Studying mice with #TRPV4 mutations, scientists show that the mutations lead to progressive neurological symptoms linked to breakdown of the blood-spinal cord barrier, a hallmark of many #neurodegenerative disorders. @ScienceTM https://t.co/PUJaRGCWNU
@DavidPatrickLab et al at @VUMCClinPharm, @VUMC_Heart, & @VAResearch show that endothelial cell stretch & #TRPV4 drive #NETosis in hypertension. NETosis deficient mice have lower blood pressure. Find out how NETosis contributes to #hypertension at https://t.co/7kTDdPGpXO
Studying mice with #TRPV4 mutations, scientists show that the mutations lead to progressive neurological symptoms linked to breakdown of the blood-spinal cord barrier, a hallmark of many #neurodegenerative disorders. @SumnerLab @hopkinsneurons https://t.co/8v9HRpktQX
#ArticlesInPress: Myoendothelial feedback in mouse mesenteric resistance arteries is similar between the sexes, dependent on nitric oxide synthase, and independent of TPRV4
Looft-Wilson
https://t.co/YWnXR6rTEU
@williamandmary
#myoendothelialfeedback #eNOS #TRPV4
What underlies #IschemiaReperfusion injury?
The @sonkusarelab group reveals that IRI causes lung endothelial cells to release ATP through #Pannexin1, activating purinergic receptors, stimulating #TRPV4 and leading to inflammation and edema. @UVACVRC https://t.co/Stb0eQs6nx
New @JORSPine article out: Exploring new insights into #TRPV4, the ion channel critical for #mechanotransduction in IVDs, under dynamic/static loading and role in cytokine regulation. ⚙️
>>> Find all here: https://t.co/kxvcfpr170
@OrsSpineSection
What a treat to get a visit from the amazing @amandarose_615 and @andygsdaniel today! (Yes one more #TRPV4 paper in the works!)
What underlies #IschemiaReperfusion injury?
The @sonkusarelab group reveals that IRI causes lung endothelial cells to release ATP through #Pannexin1, activating purinergic receptors, stimulating #TRPV4 and leading to inflammation and edema. @UVACVRC https://t.co/T6dtKrNOGk
Warmest congratulations @Nathan_NguyenTN. We’re so proud of you and all you achieved @INMLAB @CreekLab . I can’t wait for the science world to see the data in published form! It was a risky project, but with your persistence it has changed the direction of our #TRPV4 program🥳👏
Graduation ✅
Thanks @Dan_Poolio, @NikVeldhuis,@G__Siddiqui and everyone in @INMLAB, @CreekLab for your support!
Congratulations to all @MIPS_DDB PhD graduates today!
@ChemJ_Tsao @7anessa Liudi Zhang
#TRPV4 expression in the renal tubule is necessary for maintaining whole body K+ #homeostasis (Stavniichuk et al.) - new in @AJPRenal!
https://t.co/tpJWIv7irn
@dkrizaj @McGovernMed @UofUMedicine #hyperkalemia #hypokalemia #kaliuresis
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