Check out our new preprint:🧠Neuronal 🧬DNA 🧬repair mediates systemic mitochondrial maintenance through acetylcholine signaling. Neuronal repair is sufficient for the overall health amid DNA damage! https://t.co/cKVmwntJFZ
We have a postdoc/lab manager position available in our lab. The position is initially until December 2025 with the possibility to be extended until December 2032. If interested, please contact me or apply here: https://t.co/CwM3LOfm6A
We are looking for a motivated postdoc to study the links between aging and amyotrophic lateral sclerosis. The project combines disease modeling using patient-derived iPSCs and C. elegans models. Please apply here or contact me: https://t.co/eZCDghDS23
📢Thrilled to share our latest work @NAR_Open w/@OMathioso and @schumacherbj lab's on the DNA repair 🧬🛠 pathways that protect an animal from the environmental and metabolic carcinogen formaldehyde☠️ @CECAD_@CarrerasIJC https://t.co/gHp3svXoc8
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Although I moved in my career from basic research to corporate research and technology at #teamZeiss, my passion and support for the aging and age-related diseases research fields remain strong!
Our work, based on a genetic screen in C. elegans, investigates how various DNA repair mechanisms prevent FA toxicity during development, reproduction, and aging, and suggest therapeutic interventions to revert FA-pathogenic consequences.
Excited to announce our paper in @NAR_Open: "Distinct #DNARepair mechanisms prevent #Formaldehyde toxicity during development, reproduction, and aging." https://t.co/RE3P7XPgSZ
Our new preprint SIRT6 as a key initiating point in the loss of proteostasis in aging brains and neurodegeneration.
During aging, and particularly in several neurodegenerative diseases, brain proteostasis is affected. In familial cases, mutated proteins form aggregates.
🔥🔥🔥Neuron-type specific aging-rate reveals age decelerating interventions preventing neurodegeneration. 🧠🧠🧠Read here our latest super exciting #preprint on @researchsquare Congrats to @Meyer_DH and @CGallrein
https://t.co/e4WMFCUJa9
🔥🔥🔥my 💯 paper is out, congrats 🎈🎉🍾 @Meyer_DH for this amazing story. Aging clocks tick by the noise: we can build aging clocks 🕰️ on accumulating stochastic variation! @CECAD_@UniCologne@CGA_age@UKKoeln follow 🧵retweet & read now open access: https://t.co/x3sCZoYkKP
Preprint alert 🚨🚨. It is known that mitochondrial impairments cause premature aging. But "natural causes" of mitochondrial demise during normal aging are obscure. Here we identify membrane lipid phosphatidylcholine as a mendable culprit of mito-aging 🧵
https://t.co/sR3ddc4Gyp
📢 Thrilled to announce that I just joined @ZEISS_Group as Senior Innovation Manager for Health & Lab! Excited to explore new markets and technologies and help shaping the future of #ZEISS in the amazing #team at Corporate Research and Technology! https://t.co/Rp26JrdPJx