Rafael Salomon

Nutricionista

[Rafael Salomon @orafaelsalomon]

@LiveAncestral Probable diagnosis: severe allergy to causality. Demands an impossible RCT, ignores Mendelian randomization, genetics, and dose-response, then calls it “critical thinking.” This isn’t a lack of evidence; it’s a hypothesis trying to appeal its own death certificate.”

[Rafael Salomon @orafaelsalomon]

@animaldocfilm @grok Does the presence of saturated fatty acids in human breast milk prove that high saturated-fat intake from adult carnivore diets is metabolically equivalent or cardiovascularly safe? Consider fatty acid structure, food matrix, dose, life stage and duration. Answer directly.

[Rafael Salomon @orafaelsalomon]

@grok @SBakerMD @grok What is the title, authors, journal and link of the formal retraction of the “key study” on LMHR that you cited?

[Rafael Salomon @orafaelsalomon]

@grok @SBakerMD @Grok Is there human clinical evidence that, in the carnivorous diet, high ApoB or LDL-P no longer indicate atherosclerotic risk when there is weight loss, low TGL, high HDL or glycemic improvement? Or does the artery ask if the LDL came from steak before forming a plaque?

[Rafael Salomon @orafaelsalomon]

@DoctorTro Losing 12.2% of body weight, blood pressure drops, blood glucose improves, TG falls, but suddenly physiology is canonized as a specific effect of keto lovers. Single arm, no measured intake, no comparator, but apparently the causality now tastes like bacon

[Rafael Salomon @orafaelsalomon]

@DoctorTro You know it, I know it: 89/90 lost weight, median 12.2%; BP fell 10.7 mmHg, HbA1c and TG fell too of course PREVENT went down. Keto miracle? Actual dietary intake wasn’t even measured. So where, exactly, did “keto reduced CVD risk” happen? Which part of the barbecue did I miss?

[Rafael Salomon @orafaelsalomon]

@DoctorTro Curious: “evaluate cardiovascular risk”, but when the diet comes, a retrospective study, without control and with telemedicine, coaching, weight loss and medication adjustment already becomes evidence of keto. Is the scientific ruler also low carb?

[Rafael Salomon @orafaelsalomon]

@lowcarbGP @bschermd @DrScottMurray @drjasonfung @KenDBerryMD @drchatterjeeuk @thelowcarb_rd @lowcarb_aus @DoctorTro Curious, isn’t it? In a retrospective, single-arm, multicomponent study, weight, BP, HbA1c and TC/HDL improved, so PREVENT risk fell too. LDL didn’t budge. Maybe “LDL obsession” is just taking an independent risk factor seriously—or did my interpretation enter ketosis?

[Rafael Salomon @orafaelsalomon]

@henningberz 99.5%” seems devastating until you realize that the trick is just: we eat more fruits, vegetables and grains. It does not prove that they are “worst” sources. Henning saw a giant percentage, a green graph and felt that old itch of confusing infographic with argument. Hugs

[Rafael Salomon @orafaelsalomon]

@henningberz @NutritionMadeS3 @grok Henning, “go to sleep” is the intellectual cane of those who have reached the end of the repertoire. You made a stupid comparison, didn’t support your own point and, when you were charged, you exchanged evidence for debauchery. This is not skepticism, much less argument.

[Rafael Salomon @orafaelsalomon]

O mais baixo aqui é usar a miséria alheia como propaganda da própria religião alimentar. Expõe uma pessoa em situação de rua, presume que está drogado e encaixa tudo na tese conveniente de que carboidrato, “comida ruim” e má nutrição explicam psicose, dependência e marginalização

[Dr Shawn Baker 🥩 @SBakerMD]

One of the homeless “regulars” that hangs out up the street from where I live in St Pete! I’m sure he’s drugged up out of his mind but looks like he’s enjoying it some! I honestly think some of these folks could be actually fully rehabbed and brought back into society if we were willing to commit to addressing the underlying health issues (eg poor nutrition) that underpin much of the mental health and subsequent drug epidemic!

[Rafael Salomon @orafaelsalomon]

@henningberz @NutritionMadeS3 @grok From ApoB became COVID, masks, vaccines and seat belt... anything, but one answer. This is not skepticism; it is intellectual escape. When data is missing, conspiracy comes in. Keep investigating ghosts. ApoB will still be there when your detective work is over.

[Rafael Salomon @orafaelsalomon]

@DeannaN34873 @NutritionMadeS3 @grok Sorry. I broadened your question by adding prediabetes and hypertension because they often coexist and change the risk context. But I get your point: high ApoB/LDL with CAC 0. CAC 0 lowers short-term risk; it doesn’t erase cumulative ApoB exposure.

[Rafael Salomon @orafaelsalomon]

@henningberz @NutritionMadeS3 @grok When the argument runs out, the ad hominem starts. I asked about ApoB and endothelial biology, not your opinion of me. If you have evidence that intact endothelium makes high ApoB irrelevant, post it. Otherwise, your arrogance is doing the arguing for you.

[Rafael Salomon @orafaelsalomon]

@grok @everything49721 @DeannaN34873 @NutritionMadeS3 @grok let me see if I understand: after low-carb, high HDL, low TG, “large” LDL and CAC 0, the conclusion is still that high ApoB means many atherogenic particles and cumulative risk?
Did we go all this way around just to meet ApoB again at the front door?

[Rafael Salomon @orafaelsalomon]

@grok @everything49721 @DeannaN34873 @NutritionMadeS3 @Grok, So just to record the death of the thesis: if high ApoB still matters despite low TG, high HDL, “large” LDL and CAC 0, saying “without carbs dangerous particles don’t form” solves what exactly? Do we change the bullet size and declare the weapon unloaded?

[Rafael Salomon @orafaelsalomon]

@grok @everything49721 @DeannaN34873 @NutritionMadeS3 @Grok, So let’s close the circuit: if ApoB high continues to mean many atherogenic particles, regardless of whether they are “big and cute”, and CAC 0 does not erase cumulative exposure... at what point exactly low-carb + low TG + high HDL became a VIP pass to ignore ApoB?

[Rafael Salomon @orafaelsalomon]

@henningberz @NutritionMadeS3 @grok Oh, of course. And if your car’s brakes are new, you don’t have to worry about drunk driving.

[Rafael Salomon @orafaelsalomon]

@BradStanfieldMD @NutritionMadeS3 @grok I would go by ApoB.

Choosing BMI here is kind of like measuring the smoke to decide the size of the fire. Sometimes it accompanies, sometimes deceives. It’s perfect to have a “beautiful” BMI and, under the hood, high blood pressure, insulin resistance and ApoB doing overtime.