I’m Hanna, a disabled mother of three, medical enthusiast and dissectologist. Search shadowbanned. Now having to ask grok for help to search through my posts
📌 Hello and welcome to my twitter! Every tweet within this tweet is about the relationships between viruses, bacterium, and protozoans on the human/animal body and hopefully working treatments
The government literally just told us that:
-AI was responsible for the mistake of targeting a school in Iran and killing over 100 school girls.
-Almost caused a conflict with China due to a hallucination about transporting nuclear material
And they want it to run air traffic…
The cryptocurrency industry, furious at Democrats for blocking the passage of a major business-friendly bill last week, is beginning to wage war on the party’s midterm candidates.
The industry’s main super PAC, Fairshake, which is backed by some of the richest people in Silicon Valley, is starting to unspool a planned $30 million advertising campaign against former Senator Sherrod Brown of Ohio, a Democrat running again for the chamber, the group told The New York Times.
This week is World Mitochondrial Disease Awareness Week.
A few years ago, work by @AmyV91 in our group showed that skeletal muscle mitochondria in people with mitochondrial diseases tended to be more fragmented, but also had more of those long sub-micron size tubular connections called nanotunnels. https://t.co/3kYgIck6wc
We interpreted this finding as an attempt of defective mitochondria to "reach out for help."
That interpretation aligns with a defining feature of mitochondria: they're social.
Mitochondria don't operate in isolation within our cells. They build networks, form connections, and communicate with one another. In many ways, if we apply traditional features of sociality, mitochondria are a coordinated social collective.
This short video gives us a glimpse of the mitochondrial social network. It features interconnected mitochondrial communities within a cell, and the tiny nanotunnels that help them communicate.
Want to further explore how mitochondria move and connect? Visit MitoLife: https://t.co/UOW33CTrzi
I’ve covered Trump’s arch for nearly a year. I’ve watched officials reveal their plans to federal commissions and tracked them in court.
Nobody, until today, made any suggestion it would be a “military complex.”
But Trump has used a similar strategy to defend his ballroom.
Pete Hegseth went to Texas A&M & did the Holy War song & dance while quoting Bible verses & buzz words at the youth. He’s calling it the “Send Me Tour.”
As in, “Please SEND ME to war so I can die for someone else’s country.”
Tell it, Fat Bastard!
How about @MarkMeadows, who voted in 2020 swearing that this old single-wide mobile home on Scaly Mountain NC where he’d never set foot was his place of residence?
Mitch McConnell has 40 aids
Collectively taxpayers are paying nearly $4 million dollars per year for McConnell’s aids
What exactly do American taxpayers get in return for paying $4 million dollars per year for Mitch McConnell?? We need Term Limits for Congress
🚨📡 3 Stories Getting Buried in the Noise
1. Iran war cost: $43.6B and counting
2. Six senators demand probe into US strike on Iranian wedding (5 dead, 68 injured)
3. United Airlines caught transporting military weapons systems to Israel on a passenger flight
@atrupar “Our enemies are innovative and resourceful, and so are we. They never stop thinking about new ways to harm our country and our people, and neither do we.”
SARS-CoV-2 has a protein that can turn immune cells into accomplices
➡️ Researchers found that the viral protein #ORF8 reprograms #macrophages, increasing #ACE2 on their surface and making them easier for SARS-CoV-2 to infect.
➡️ The infected macrophages then lose antiviral function and release inflammatory signals that amplify infection and lung damage in experimental models. 1/
University of Amsterdam researchers analyzed 394 samples and found a 7-gene Long COVID signature across 5 datasets.
ECHS1 was repeatedly reduced, pointing to altered mitochondrial fatty acid oxidation alongside immune dysregulation.
https://t.co/wpmY0WSsYy
SARS-CoV-2 impairs VDR signaling in monocytes/macrophages, downregulating VDR/target genes (CAMP/LL-37, IRF5) and amplifying IL-6 via NF-κB (Valdés-López et al., PLoS One 2026; George et al., Sci Rep 2021). Reduced antimicrobial peptides and anti-inflammatory effects weaken control of latent EBV. EBV EBNA-3 binds VDR, blocking its genes (Yenamandra et al., Cell Mol Life Sci 2010). COVID inflammation/NLRP3 and lymphopenia further drive EBV reactivation; low 25(OH)D correlates with higher EBV load (Pérez-Pérez et al., Eur J Neurol 2018).