Could aging and #LongCOVID weaken the body’s ability to keep “sleeping” viruses asleep?
➡️ A provocative framework suggests that aging and LongCOVID may represent states of declining biological resilience, where chronic inflammation, immune exhaustion, mitochondrial dysfunction and metabolic stress gradually erode the systems that keep latent viruses like CMV under control. 1/
"Covid-19: 40% of affected healthcare workers had long covid, and a quarter still had symptoms after a year"
Published: 20 August 2026
Source: https://t.co/GcrujOLLje
🧵 1/2 For 10 years, Project Lyme has brought patients, researchers, clinicians, advocates, and supporters together to help move Lyme research forward. Dr. Michal Tal @ImmunoFever from @MIT will join us at our upcoming Gala to celebrate what collaboration can make possible.
🇮🇳India: "Study reveals alarming 2.5x surge in cardiac emergencies post-COVID among youngsters"
Before the pandemic in 2018–19, patients under 40 made up over 8% of cardiac emergencies. That figure has since escalated more than 2.5x to nearly 21% of cases.
Autopsies of 18 foetuses showed that "SARS-CoV-2 can be widely distributed in fetal organs through vertical transmission". In addition immune and DNA damage were found in the infected organs
https://t.co/2HCRw66ziI
This piece was hard to write. It's hard to read. Most importantly, it's hardest to live.
@CODA_research and our partners won't accept 400M+ people losing their lives to chronic illness. We've built a model to bring real answers.
Please read my latest blog here: 👇
https://t.co/hVXvZudeMv
I'm excited to share that our recent paper has been featured in Nature Reviews Neurology.
Our study provides evidence that autoantibodies can play a causal role in the neurological manifestations of #LongCOVID.
https://t.co/3SNJIKZfpo
#Autoimmunity#Neuroimmunology#Immunology
A recent YSM study published in @CellCellPress links #LongCOVID symptoms in some patients to specific autoantibodies, suggesting #autoimmunity as one cause of post-infection illness.
First author @keylas3 and senior author @VirusesImmunity share how their findings could lead to new diagnostic tools and #treatment options.
Learn more: https://t.co/LZwc6mRioF
A new philanthropic effort — backed by money from some of the biggest names in AI — is taking aim at eliminating respiratory viruses, starting with the common cold and influenza.
Intercept is starting with $500 million in seed funding from Anthropic, OpenAI Foundation, and the payment company Stripe, along with venture capitalists at Jane Street, and from Bill Gates, through a philanthropic entity.
The organization is going after cold and flu viruses first, citing the huge economic and health burden these common infections cause each year. And anything that targets these viruses is likely to “meaningfully reduce pandemic risk,” according to an online post by the fund that explains its mission. https://t.co/2nB99LSzzN
“Wearing a well-fitting N95 or KN95.. can reduce the risk that you get infected — and I think for most people, that’s the goal at this point” https://t.co/lCcb1zURCw
Far-UVC is rapidly being recognised as an effective way to reduce the amount of infectious pathogens in indoor air
Given the importance of player's health when it comes to their performance, it's not surprising to see NFL teams utilising this new tech
https://t.co/ecf0UCLnPf
PMC COVlD Update, Week of Aug 24, 2026
(posted 2 days early)
The US is experiencing COVlD outbreaks in 23 states/territories (see Alt text).
▪️Largest outbreaks: TX, CA, HI, AK
▪️1 in 136 Americans estimated actively infectious
▪️Highest levels since March 20th
🧵1/11
IBSAL researchers studied 304 people with Long COVID.
The high multisystem burden group had higher ICAM-1, a marker of endothelial activation, than low burden and affective-mental groups.
Vascular measures did not differ.
https://t.co/LlNBdbQTrM
Exactly—but if you want to erase the demolition of 20+ million American lives—go ahead & complain about school closures without acknowldeging how many more children, teachers & healthcare workers would be living with this utterly ignored & devastating medical quagmire. #LongCovid
How does gut dysbiosis ('sick' gut microbiome) lead to neuroinflammation?
Why is treating 'whole body pathogen load' and lowering LPS so crucial?
This is a beautifully simple diagram.
1. In dysbiosis, pro-inflammatory bacterial strains predominate, producing larger amounts of endotoxins such as Gram-negative lipopolysaccharides (LPS).
2. The translocation of LPS through the intestinal lining can activate the HPA axis
3. LPS escaping through the gut lining also triggers an immune response with increased cytokine production, mediated by Toll-like receptors (TLR) 4 and TLR2 through NF-κB signaling.
4. Additionally, dysbiosis activates mast cells, leading to the release of tryptase, TNF-α, IL-1β, IL-4, IL-13, and NF-κB, further stimulating the immune response and causing damage to the intestinal mucosa.
5. Note the feedback loop here where damage begets damage in the gut and then the gut issues start to impact to brain (neuroinflammation).
Photobiomodulation of gut microbiota with low-level laser therapy: a light for treating neuroinflammation
https://t.co/ZcQF6jCmrb
"the COVID-19 pandemic continues. Long COVID is still making people sick, long after infection, damaging immune systems and triggering various illnesses." https://t.co/fZmD9n7uSE
One of the ways our researchers determine what could be causing fatigue in complex chronic conditions is through metabolomics.
In our study, we measure 2,000+ metabolites that can give participants insights on their biology.
More Info: https://t.co/vvYZYECJhw