d

[Dr. Khaled @kh505043]

What is the correct site for an intramuscular injection IM?

[Oren Gottfried, MD @OGdukeneurosurg]

Bacteria

[Dr Ihab Suliman @IhabFathiSulima]

What is the connection used for?

[AbdAllah Othman @CardioBeat_]

https://t.co/MC9dQhaIo5
💥 In-Depth Visual Guide to ECG rhythms🫀📊
#Cardiology #HeartHealth #Arrhythmia #ECG
#ecginterpretation

[マル太 @luchunan21]

Salamat sa 23k ffs!😘
Eto na maagang pamasko

UNCENSORED PART TWO (17min)
Inaya ako ng isang nearby edger kaya dinala ko sya sa labas at doon nya ko hinubaran, nilamas at ginatasan hanggang lumabas lahat ng naipon na katas 💦🥵

Bookmark and share! Follow for more!

[Jameel Bunu @JamiluBunu3465]

@Doctors_GUILD A simple way to remember it:

Heart attack = circulation blockage (a “plumbing” problem)

Cardiac arrest = electrical shutdown (a “power” problem)

[Jameel Bunu @JamiluBunu3465]

A heart attack and a cardiac arrest are related but very different events:

A heart attack (myocardial infarction) is a circulation problem. It happens when a coronary artery becomes blocked, so part of the heart muscle doesn’t get enough oxygen and starts to die. The person is usually conscious at first and may have chest pain, sweating, nausea, or shortness of breath. The heart is still beating, but damaged.

A cardiac arrest is an electrical problem. The heart suddenly stops pumping effectively due to a rhythm failure (like ventricular fibrillation or asystole). The person collapses, becomes unconscious, and has no pulse or normal breathing. This is immediately life-threatening and requires CPR and defibrillation.

A simple way to remember it:

Heart attack = circulation blockage (a “plumbing” problem)

Cardiac arrest = electrical shutdown (a “power” problem)

Important point: a heart attack can lead to cardiac arrest, but they are not the same thing.

[Mednomics @mednomics]

Most preferred antihypertensive drug to use during PREECLAMPSIA 🤔

[Dr PARVEEN YOGRAJ @dr_yograj]

The Silent Superbug Rising Again: Acinetobacter baumannii in Recurrent UTIs
—————————————————
An unusual but worrying trend is resurfacing in clinical practice. Today in my evening clinic, the third patient with recurrent UTI showed growth of #Acinetobacter baumannii — a Gram-negative bacillus increasingly emerging as a difficult-to-treat urinary pathogen. Once predominantly confined to ICUs and hospital-acquired infections, this organism is now appearing more frequently in OPD practice, particularly among elderly, diabetic, catheterized, immunocompromised, or repeatedly antibiotic-exposed patients.
Acinetobacter baumannii is considered one of the most resilient bacteria in modern medicine. It survives for prolonged periods on hospital surfaces, medical instruments, sinks, bed rails, and even dry environments. Its resurgence is largely attributed to irrational antibiotic use, repeated empirical therapies, prolonged hospitalizations, invasive procedures, urinary catheterization, and excessive broad-spectrum antibiotic exposure during the post-COVID era.
Clinically, it can present with recurrent burning micturition, pyuria, fever, persistent bacteriuria, catheter-associated UTI, prostatitis, sepsis, pneumonia, and wound infections. Microbiologically, it is a Gram-negative coccobacillus with a frightening ability to rapidly acquire multidrug resistance genes through plasmids and biofilm formation.
The major concern is its growing resistance to antibiotics. Many strains are now resistant to penicillins, cephalosporins, fluoroquinolones, aminoglycosides, tetracyclines, and even carbapenems. Resistance mechanisms include beta-lactamase production, efflux pumps, porin loss, and biofilm-mediated protection. Carbapenem-resistant Acinetobacter baumannii (CRAB) is now globally recognized among the most dangerous antimicrobial-resistant organisms.
Yet there is hope — culture-guided therapy remains the key. Sensitivity-based antibiotics such as meropenem, cefoperazone-sulbactam, piperacillin-tazobactam, aminoglycosides, fluoroquinolones, or cotrimoxazole may still be effective depending upon local antibiograms. The rising incidence clearly emphasizes one message: recurrent UTI should never be treated blindly without urine culture and antibiotic sensitivity testing.
#RecurrentUTI #AntibioticResistance #AMR #UTI #Microbiology #MedEd #InfectionControl #MedTwitter #AntibioticStewardship #MedX
@IhabFathiSulima @DocPriyamMD @docakx @drkeithsiau

[Milking Muscle @MilkingMuscle]

🩷🩷🩷 PISS VIDEO 🩷🩷🩷

MUSCLE DADDY @man999xxx
DELICIOUS HOT PISS

https://t.co/FgQtpSrW8U

🐽 30 MINUTE VIDEO 🐽
❌❗️ NO MOSAIC ❗️❌
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[Dr RP Singh, MD @rpsingh1894]

🔥 Which hormone decreases appetite? 🔥

⸻

🎯 Correct Answer:
👉 💥 Leptin 💥

⸻

🧠 Quick Concept
•Leptin = Satiety hormone 🛑🍽️
•Secreted by adipose tissue
•Acts on hypothalamus → ↓ appetite + ↑ energy expenditure

⸻

⚡ Why NOT others?

❌ Ghrelin → 😋 Hunger hormone (↑ appetite)
❌ Cortisol → ↑ appetite + weight gain
❌ Neuropeptide Y (NPY) → Strong appetite stimulator

⸻

🖼️ Appetite Regulation

⸻

💡 Memory Trick
👉 “LEP-tin = LET’s STOP eating”

⸻

⚡ Exam Pearl
•Leptin resistance → obesity
•Opposes Ghrelin & NPY

⸻

#Endocrinology #Physiology #NEETPG #PLAB2 #MedTwitter 💊🔥

[Summaiyat | PharmWise⚕️📚 @PharmWise]

Why is Ceftriaxone avoided in Lactated Ringer's ?

[Safiullah @ChShafiullah15]

The TRIAD is called ?

[Dr. Muhammad Moor @MoarSahitoPTI]

The drug of choice for treating methotrexate toxicity is.? 💊

A. Folic acid
B. Leucovorin
C. Vitamin B12
D. Pyridoxine

[Dau⚕️ @daoo100]

🚨Possible diagnosis ❓

A. Rheumatoid Arthritis
B. Carpal Tunnel Syndrome
C. Dupuytren's contracture
D. Volkmann ischemic contracture
#MedX ?

[Dr. Medica🩺 @DrMedica_13]

Did u know?

🥀☘️🥀

[Doctor Med @DoctorMed0]

The TRIAD is called....

[The Medics🩺💊💉🏥 @DrArslan480]

Answer???

[el novio de Mexico 🇲🇽 @alex_uri13]

Chudai

[Dr. Muhammad Moor @MoarSahitoPTI]

What are the main clinical uses of Semaglutide injection.? 🤔