各位觀眾朋友,大家好。我是何頻。今天我們《中國財經台》要聊的這個話題,可以說是切中了當前中美戰略競爭的「命門」。《華爾街日報》最近披露了一個極為關鍵的動向:白宮正計劃調整策略,將原本撥給大學的科研經費,大規模地轉向人工智能(AI)領域。在我看來,特朗普這一次是真正抓對了方向。
為什麼我會這麼說?
長期以來,大家習慣了傳統的軍備競賽,拼坦克、拼航母。但大家必須清醒地認識到,在今天這個時代,AI 的重要性早已超越了傳統武器。這不再僅僅是技術之爭,而是國力底層邏輯的重構。誰能掌握最強大的算力和演算法,誰就掌握了未來世界的話語權。特朗普政府意識到,與其把經費分散在那些進度緩慢、甚至可能被滲透的傳統大學科研項目中,不如集中火力,在 AI 這個決定性的戰場上實現絕對領先。
這就是我一直強調的觀點:**AI 比傳統軍備競賽更關鍵**。如果美國不能在 AI 領域保持領先,那麼傳統軍事的優勢將會迅速瓦解。
與此同時,我們也要看到全球地緣政治的聯動。當前最迫切的需求是什麼?是結束那些無休止的、消耗巨大的戰爭。不管是俄烏衝突還是其他地區的動盪,這些戰爭都在極大地拖累全球經濟,特別是消耗了原本可以用於科技創新、用於 AI 研發的寶貴資源。
所以,我始終主張,我們應該推動結束戰爭,並對北約進行徹底的改革。北約不能再維持冷戰時期的僵化思維,而必須適應新時代的挑戰。如果不能將有限的資源從戰火中抽離,投入到以 AI 為核心的科技革命中,那麼西方世界乃至全球的經濟秩序都將面臨嚴峻的考驗。
中美之間的競爭,表面上是貿易,實質上是科技,而核心中的核心就是 AI。白宮這次的動作,預示著美國正在進行一場深刻的戰略轉向。我們《中國財經》將會持續關注這場轉變如何影響中國的應對,以及全球財經格局的洗牌。
謝謝大家。https://t.co/7dY8BZnB3C
Latest episode of Lifespan is out!
I'm so proud of the team who worked >300 hrs to make the most in-depth, 2-part episode covering:
1. The real facts about fasting
2. Why & how it slows aspects of aging
3. Easy tricks to implement the practice 💪
https://t.co/I7yLbd6N0d
Ever since Jensen Huang said $MRVL could become a trillion-dollar company, Marvell’s stock has already rallied 50%.
$MRVL has gone from a year-to-date low of around $70 to roughly $310 today, a gain of about 342.857%, adding approximately $210 billion in market value.
And next week, $MRVL is set to be added to the S&P 500.
That could drive even more upside for the stock. Once index-driven buying begins, $MRVL’s market cap could potentially reach $500 billion to $800 billion by year-end.
For value investors, this is a stock with serious long-term investment appeal.
I’ve been saying for a while that $MRVL is a strong long-term hold. The market is proving that call right.
Have some patience. One year from now, you’ll be glad you listened.
Two days ago, something happened that most of the world still doesn't know about.
A human being received the first therapy in history designed to reverse cellular aging inside their body. Not slow it down. Not manage the symptoms. Actually reset the biological clock at the cellular level.
@lifebiosciences — co-founded by Harvard geneticist @davidasinclair — announced on June 9 that the first patient was dosed in their Phase 1 clinical trial of ER-100.
Let me explain what this actually is, because the implications extend far beyond the eye they're treating.
Your DNA is mostly intact throughout your life. Think of it as hardware. What degrades is the epigenome — the layer of chemical instructions that tells your genes what to do and when. Over decades, those instructions get corrupted. Genes that should be active go silent. Genes that should be silent activate. Cells lose their identity and function. We call this aging.
ER-100 uses three transcription factors — OCT4, SOX2, and KLF4 — to perform a partial epigenetic reset. These are the same factors that can reprogram any adult cell back into a stem cell. But they're not going that far. They're doing just enough to make damaged cells function like younger versions of themselves — without losing their identity.
The delivery is elegant. A one-time injection into the eye. An oral pill acts as an on/off switch — doctors control exactly how long the reprogramming lasts. If anything goes wrong, they turn it off.
This didn't come from nowhere.
In 2020, Sinclair's team restored vision in old mice and mice with glaucoma using this approach. The paper made the cover of Nature. They moved to monkeys with optic nerve damage — and successfully restored function with no major safety issues. Now, two days ago: the first human.
The trial is targeting glaucoma and NAION — a sudden "stroke of the eye" that causes devastating vision loss. Current treatments for both can only slow progression. Nothing reverses the damage. This therapy aims to regenerate damaged optic nerve cells by making them young again.
As a cardiologist, here's why this keeps me up at night — in the best possible way.
The eye is a safe, measurable starting point. But the platform technology isn't limited to the eye. If partial epigenetic reprogramming works safely in optic nerve cells, the same approach could theoretically be adapted for the brain, the spinal cord, the heart, the liver, the kidneys — any organ where cells lose function with age.
We're not testing whether we can treat one disease. We're testing whether we can treat aging itself as a reversible biological process.
For my entire career, cardiology has been magnificent at managing the downstream consequences of aging: high blood pressure, plaque buildup, heart failure, arrhythmias, stiffened arteries. We prescribe statins, antihypertensives, anticoagulants, and devices to manage what time does to the cardiovascular system. We're getting better every year.
But what if the cells themselves could be reset? What if the cardiac muscle cells that lose contractile function with age could be epigenetically restored? What if the endothelial cells lining sixty thousand miles of blood vessels could be made to behave like they did decades earlier?
That's not in a trial yet. But the foundational technology just entered a human body for the first time. And the companies racing in this space — Life Biosciences, Altos Labs, NewLimit — are backed by billions of dollars and some of the best scientists alive.
Essential caveats — because I'm a physician, not a hype man.
This is Phase 1. Roughly 18 patients. The primary goal is safety: does this cause harm? Is it tolerable? Efficacy data will come later. Many promising therapies have failed in the gap between animal models and human reality. Gene therapy carries risks — immune reactions, off-target effects, unintended cellular behavior. The oral on/off switch is a critical safety feature, but this is still the earliest possible stage of testing.
No one is reversing aging tomorrow. No one is living to 200 next year.
But a line was crossed two days ago that cannot be uncrossed.
For the first time in human history, someone received a therapy specifically designed to make damaged cells young again inside a living person. From impossible in theory to proven in mice to tested in monkeys to injected into a human being — in six years.
I've spent twenty years treating the consequences of aging. I've held hearts that were failing because time had degraded the cells beyond recovery. I've watched patients lose vision, cognition, mobility, and independence — and told them there was nothing I could do to reverse what time had taken.
This week, the conversation began to change.
Gene editing to permanently lower cholesterol. Personalized mRNA vaccines to hunt cancer. GLP-1 drugs rewiring metabolism, reducing depression, and cutting cancer metastasis. And now — partial cellular reprogramming to reverse aging at the epigenetic level.
I said months ago on this platform that our children may live past 100 — not frail, but strong and vibrant.
The evidence keeps building that this isn't optimism.
It's a timeline.
After 25 years of brave & brilliant work by hundreds of scientists in my lab to understand then safely reverse aging for the first time, it was moving to witness the first human dose being delivered 🥹 https://t.co/veQsyUEORz