"Cruise ship confined spaces and HVAC systems facilitated 👉aerosol👈 transmission."
We will accept apologies now from the Droplet Dogma gang. And @WHO.
Soutce: Hantavirus outbreak on cruise ship: public health challenges. Biosafety and Health, Jun 2026,
🇦🇺Australia: Starting in mid-2026, a new national program will test wastewater across Australia to track diseases like COVID-19 and the flu. This will help health officials spot outbreaks early and stay ready for future threats.
Source: https://t.co/0WUKg7EiLs
🚨 Belgium is facing a major long-term sick crisis.
Latest Riziv figures (end 2025): 576,643 long-term sick in the private sector + self-employed.
Add civil servants (medical/illness pensions): estimates around 87,000 or more!
Total estimate: 650,000 – 670,000+ people out of the workforce for over a year(= 12-14%)
Tens of thousands of them are dealing with Long COVID (a significant and growing group, often misdiagnosed as burnout or other psychosocial issues).
Record numbers, still rising, driven by burnout, depression, musculoskeletal problems & post-COVID.
Among the highest in Europe.
Time for real solutions instead of just handing out benefits!!
#LongTermSick #LongCOVID #Belgium
Research progress on the association between viruses and cardiac diseases
🚨Your heart’s deadliest serial offender just got named: SarsCoV2
Interesting Chinees review just mapped how six viruses attack the heart.
One stands out!
Not because the authors scream “worst ever,” but because the science does.
→Direct heart-cell invasion + long-term damage + cumulative hits from regular reinfections = a unique threat!
Vaccines help. But the data is sobering.
Let’s break it down virus by virus(review):
1. SARSCoV2:
- Enters cardiomyocytes and pericytes via ACE2 receptor (membrane fusion/endocytosis).
- Direct infection triggers myocarditis, pericarditis, arrhythmias, heart failure (de novo or exacerbation), microclots, thromboembolic events and myocardial infarction,
- Long-term: persistent cardiac inflammation, dysautonomia and long-COVID cardiovascular symptoms,
- Possible viral persistence in tissue,
- Vaccination reduces myocarditis risk/CV complications compared with natural infection,
-Regular reinfections!
2.Influenza (A/B):
- Direct replication in cardiomyocytes, Purkinje cells and endothelial cells (independent of lung titers),
- Causes myocarditis, pericarditis, arrhythmias, heart-failure flares and myocardial infarction via both cytopathic effects and cytokine-driven systemic inflammation/endothelial dysfunction.
- Long-term: myocardial fibrosis and remodelling,
- Vaccines lower infection-related cardiovascular complications,
3. HIV:
- No productive cardiomyocyte infection,
- Indirect damage from chronic immune activation, endothelial dysfunction, metabolic changes and antiretroviral toxicity,
- Drives cardiomyopathy (myocarditis in advanced cases), heart failure, myocardial infarction and stroke,
- Long-term: premature cardiovascular aging, fibrosis and remodelling even on suppressive therapy,
4. CVB3 (Coxsackievirus B3):
- Enters via CAR receptor,
- Direct cytopathic necrosis, oedema and impaired contractility in cardiomyocytes,
- Rapid progression to myocarditis, dilated cardiomyopathy, arrhythmias (QTc prolongation) and heart failure,
- Chronic inflammatory cardiomyopathy is a hallmark sequela,
5. Human cytomegalovirus(HCMV):
- Lifelong latency with reactivation (especially in immunocompromised),
- Induces myocarditis, heart failure, transplant vasculopathy and atherosclerosis through sustained inflammation and vascular injury,
- Long-term myocardial remodelling,
6. Arthropod-borne viruses(Arboviruses = DENV, CHIKV, ZIKV):
- Direct cardiac invasion (some models) plus immune-mediated injury,
- Associated with myocarditis, arrhythmias, heart failure, shock and (CHIKV) dilated cardiomyopathy or long-term ischemic heart disease/cerebrovascular risk.
‼️So, this minireview examines six cardiotropic viruses side-by-side, yet SARS-CoV-2 receives the most space because of its proven direct cardiomyocyte invasion, explosive multi-pathway damage, and documented long-term cardiac persistence, risks that broader population data show accumulate relentlessly with every regular reinfection, creating an immediate and chronic cardiovascular burden on a global scale that no other virus in this review matches.
→Your heart’s deadliest serial offender just got named: SarsCoV2!
#PREVENTION #COVIDHeart #RepeatInfectionKills #AvoidSars2 #AvoidReinfections #YouOnlyHaveOneHeart
https://t.co/BPKRV7nwbA
Dear DfE,
The biggest cause of pupil absence and teacher absence, by a very long way, is illness.
Your own data shows this clearly.
If you want to improve school attendance, you have to improve health.
Start with HEPA air purifiers.
They cost less than £10 per pupil.
Please
results suggest that a positive test for COVID-19 (vs a negative test) was associated with increased rates of diagnosis of various infections in the 12 months following an acute SARS-CoV-2 infection. The effects of C-19 on the immune system https://t.co/7eDYIdic2I
We’re a primary care practice in Vermont. We implemented a practice-wide protocol screening every patient at every encounter for recent SARS-CoV-2 infection history. What we’re observing in our panel is not consistent with a psychosomatic framework.
We’re seeing measurable, objective increases in new-onset hypertension, acute cardiovascular events, new-onset allergic disease, and new-onset type 2 diabetes mellitus, all temporally correlated with infection history. These are not symptom reports. These are clinical findings.
There are plenty of world-class L0ngC0vid experts far smarter than I am.
But I bring something they don’t: I’m a CV surgeon and endovascular specialist. I’ve had hearts, lungs, arteries, and other organs literally in my hands. I’ve seen their insides, healthy and ravaged, with my own eyes.
That gives me one brutal, irreplaceable edge: I can take the science and translate it straight into the living, bleeding reality of human anatomy.
And what I see coming is ugly.
You, personally and as a society, are in for one hell of a shock.
Read this 🧵👇
If you keep minimising SARSCoV2, refuse to protect yourselves, and keep swallowing the lies of pseudo-experts chasing money or psychiatrists salivating over a fresh FND goldmine… you’re walking straight into disaster.
Tell every last one of them to https://t.co/rw58iRjUf4 off!
Then follow the hard, unfolding science.👇
Your organs don’t care about opinions. They only care about damage. And the damage is already stacking up, be it momentarily maybe still clinically "silent"! #AvoidSars2 #AvoidReinfections
@hannahspierMD With respect - you need to stay in your own lane here. The more you double down the more you reveal that you have no idea what you are talking about when it comes to complex chronic illness & post-acute infectious diseases.
The fact that you are trying to smugly claim you’ve 1/2
I’m proud to have our latest research published on how long #SARSCoV2 remains infectious while airborne! We studied the fundamental processes that drive the loss of viral infectivity in the aerosol phase. #COVIDisAirborne
Here’s a thread going over some of the findings.
The findings suggest that long-term exposure to common pollutants may negatively influence brain health even in lower-pollution environments
https://t.co/xx32H4Ar8P