@psychogramy@9mmsmg Here's one, and I'm not kidding:
"Priapism: Painful, prolonged erections lasting over four hours that can cause permanent tissue damage if untreated."
In other words, permanent errectile dissfunction.
@carrierichards@ErwanLeCorre Do it 5 times in a raw, few times a day, for few months, and you will notice a difference. If 5 is too much, start with 3, and do more when it gets easier. If you strugle with 1, do one few times a day. Find a workable way to start, what's feasible to you, use a stool if needed.
🇪🇺 Chat Control has passed 😔
They can and will now legally scan any person's messages, emails and photos you send without a warrant
The way they passed this law when the majority of the European Parliament was against it will shock you:
They waited until most EP members were on holiday so only a few were present and then created an "urgent" vote for it to pass it through
There's nothing democratic about any of it and big powerful forces are behind this that can manipulate the EU for whatever they want
Democracy in Europe died a bit today 😔
@JoeC4281@SamaHoole@DrJohnDenstedt I too was supposed to have stomach ulcers for the rest of my life, but ~25 years ago I took antibiotics for 2 weeks, prescribed 5 years after initial diagnosis, and had no more problems. You never know. Hopefully, we will find what causes too much uric acid.
These two paragraphs of my verdict are crucial for everyone to read and understand.
"Even if all of the statements made by Van Langenhove are based on scientific evidence and statistics, it makes no difference to the criminal intent. Van Langenhove is not charged with spreading false information. He is charged with presenting facts in a way that incites hatred against persons on the grounds of one or more of the protected criteria in the Anti-Racism Law.” 1⃣
"For Van Langenhove to have committed a crime, it is not necessary for him to have incited concrete acts of hate or violence. It suffices that others are incited to take on a general attitude of intolerance or disapproval regarding a group protected under the criteria of the Anti-Racism Law." 2⃣
This means you can go to jail for "inciting hatred" even if your statements were 100% factual (see 1⃣) and even if you did NOT incite concrete acts of hate (see 2⃣).
The benchmark of "inciting hatred" , a crime punishable by prison, is thus "saying something that has the potential of inciting someone to have a general attitude of disapproval regarding a protected group". This means literally any criticism of mass migration is now a punishable offence. If you cite a statistic, and someone could potentially think less of a protected group (like migrants) because of it, you can be jailed.
The craziest part is that there is no defence possible against this. I brought the scientific studies that I cited to court, but the judge didn't care 1⃣. I also proved that the hundreds of students present at the lecture included students of all different political affiliations, and everyone was able to voice their opinion or ask questions. The lecture went very calmly, so obviously nobody was incited to hatred. But this too did not matter 2⃣, because if the judge says he believes there is the possibility that someone COULD be incited to "a general attitude of disapproval", this is enough for the judge to send me to jail, even without any evidence.
I'm telling you this to warn you that by the time these hate speech laws have come into place, it's already too late. You will NEVER be able to beat these laws in court. You have to stop them before they are implemented. Let my fate be your warning.
🚨Током 16 минута тишине је дошло до инцидента у којем је возач аутомобила регистарских таблица BG1070SV најпре ошамарио једног студента, а потом колима ударио човека старог скоро 90 година који је стајао на раскрсници током одавања поште.
Nick is not too young to show soft plaque after seven years of having a cholesterol of 700 mg/dL.
His n=1 case report DOES tell us something incredibly important.
In 1953, it was shown in 300 autopsies of American soldiers who died in the Korean war with a mean age of 22 that 77% had plaque and 15% had luminal narrowing.
Nick is in his 30s so it is expected for him to have some soft plaque even without extremely elevated cholesterol.
Causation can either be necessary, sufficient, or necessary and sufficient.
Nick’s n=1 case shows all on its own that 700 mg/dL cholesterol and its associated high LDL-C and ApoB is not sufficient to cause atherosclerosis.
It may still be necessary to have high LDL-C or ApoB.
In fact, Nick could be the only person on the entire planet who is missing one other factor that, once combined with high LDL-C or high ApoB, is sufficient to cause plaque.
As such it is possible that Nick’s results do not apply to any other person on the planet, when looking at his case report alone.
But that’s totally different from saying we can’t learn a principle from his case report that we know does translate to everyone else.
The principle is that high cholesterol is not sufficient to cause heart disease.
The reason we don’t know if his report generalizes to other people is not because we don’t know if we can generalize that one principle. It’s because we don’t know how Nick is different from other people and we therefore don’t know if anyone else has the same mix of relevant factors.
In other words, it does matter that his n is 1 because for this reason we can’t make a probability statement about the likelihood that someone else can maintain this high cholesterol for the same period of time without showing plaque.
But that’s separate from the point that it only takes a single cause of A coexisting with lack of B to show that A is not sufficient to cause B.
That’s assuming you measured A and B correctly.
Now if we move beyond his case report we can go back to the research showing LDL requires damage to be taken up into plaque and facilitate disease progression.
Maybe Nick is lacking that damage.
Also, the characterization of the LMHR phenotype is suggestive of a generalizable principle connecting Nick with these other folks. It needs a lot more research.
But the point that 700 mg/dL cholesterol for seven years coexists with no plaque is a major demonstration of the insufficiency of high cholesterol or ApoB to cause plaque.
🚨New Paper: "Seven Years of 700 Cholesterol Without Coronary Atherosclerosis: A Lean Mass Hyper-Responder Case Report"
Link: https://t.co/5VnRpZlFdR
For the past 7 years, I’ve been running what is essentially a natural experiment in cholesterol and heart health.
During that time, I’ve largely lived with:
👉Total cholesterol around 700 mg/dl
👉LDL cholesterol between 500–600 mg/dL
I recently underwent advanced coronary CT angiography imaging with AI-guided analysis. This is not a CAC. It measures all plaque (soft + calcified), with expert interpretation and AI-guided analysis capable of quantifying plaque down to the cubic millimeter (mm3).
Now, to address the obvious question:
Am I too young for plaque?
In brief: No.
The clearest comparison is individuals with homozygous familial hypercholesterolemia, who often have similarly extreme LDL/ApoB levels and can develop advanced plaque as toddlers, and even heart attacks as early as age 8.
Also, nutrition influencers in their 30s have publicly shared quantified plaque scores from these same imaging technologies. In one recent case, a plant-based influencer in his thirties was found to have 61.3 mm³ of plaque despite having far lower lifetime LDL exposure. (He can identify himself if he so chooses.)
My case also isn’t a one-off.
There are many individuals like me, including older individuals with similar LDL-C and ApoB without any plaque.
The difference is that I’m an unusually well-characterized subject, with extensive metabolic data and health markers tracked over time. You can learn more at the newsletter or open-access paper, linked above.
The science of heart health is not settled. And cholesterol is not a simple story.
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