Top Tweets for #PolyCythemia
When More Is Not Better – Polycythemia - Yves Bikorimana
https://t.co/j4iV4FK50O
#Hematology #Hemostasis #HemostasisNews #HemostasisToday #Medicine #Health #Polycythemia
Next-Generation Treatments Transforming Polycythemia Vera Care - Abhishek Kumar
https://t.co/ZcZpjiVfVM
#Hematology #Hemostasis #HemostasisNews #HemostasisToday #Medicine #Health #MedX #MedNews #MedEd #Polycythemia
Blood Disorders: More Than Just a Diagnosis – Malaika Mehboob
https://t.co/2TZ21DP4Dd
#BleedingDisorders #ClottingFactors #Health #Hematology #Hemophilia #Hemostasis #HemostasisToday #Medicine #Platelets #Polycythemia #SCD #Thalassemia #MedEd #MedX
Understanding Polycythemia - William Aird (@WilliamAird4)
https://t.co/67KLBrKn7N
#Health #Hematology #Hemostasis #Polycythemia #HemostasisToday #PolycythemiaPatientPackage #Medicine #MedEd #MedX
7) There are also criteria for #myelofibrosis when it arises after essential #thrombocythemia #ET and #polycythemia vera #PV #PCV:
The reaction my oncologist/hematologist had when I jokingly asked her do I have time to get one last final tattoo before starting any treatment. 🤣
I thought I was going to have to administer CPR.
#GarbageBlood
#Polycythemia
Polycythemia in one page. Made simple.
Fellows Residents here you go.
Save, learn and share .
#MedTwitter
#polycythemia
Zhou et al. reveal that heterozygous JAK2V617F variant in megakaryocyte-erythroid progenitors drives essential #thrombocythemia via STAT1-IDO1-AhR-RUNX1, whereas the homozygous JAK2V617F variant promotes #polycythemia vera through the activation of STAT5. https://t.co/XIEmGhGr7T
Flopsy BunBun is sad about bruises from blood work last wk.
I'm cranky coz this 2ndary #Polycythemia is kicking my ass.
My red blood cells are over generating, I'm slow & stupid, fatigue, headache, dizzy, blurred vision, lacking oxygen, I'm red, itchy & getting nosebleeds. 🖤😣🖤
1/ Our new paper is out 📢 — on Pacak–Zhuang syndrome:A rare triad of #pheochromocytoma/#paraganglioma, #polycythemia, and #somatostatinoma caused by mosaic HIF2α (EPAS1) activating mutations. Studying it offers insights into mosaic disorders:
https://t.co/kW28gLXdXh
#Polycythemia due to demand to
👇
Produce more #RBCs red blood cells
👇
Can be a fuse of #iron #deficiency
⬆️HCT ⬇️MCV
Thanks for
Insight @WilliamAird4
1/3
TESTOSTERONE-INDUCED POLYCYTHEMIA AND ID
I posted a series of labs showing elevated Hct and low ferritin, c/s polycythemia and iron deficiency (ID).
One overarching dx is polycythemia from any cause complicated by ID from increased erythropoietic demand for iron.
⚠️ IMMEDIATE #MEDICAL #HELP NEEDED FROM DOCTORS FOR MISTER SHAMEYA ⚠️
Please repost or answer if you have the proper knowledge.
#polycythemia #oncology #hematology #Gaza
🩸 Comprehensive #Polycythemia Vera Management:
Phlebotomy
•🎯 Target Hct ≤45% (CYTO-PV trial: reduced thrombotic & CV events).
•🩸 Remove 300-500 mL/session, frequency tailored to maintain targets.
Cytoreductive Therapy
•👥 For high-risk patients (age >60 or prior thrombosis):
•Hydroxyurea: 🟢 First-line (practical: monitor for cytopenias).
•Interferon-alpha: ✅ Ideal for younger patients, pregnancy, or hydroxyurea intolerance.
•Ruxolitinib: 🔬 JAK2 inhibitor for hydroxyurea-refractory/intolerant cases.
Aspirin
•💊 Low-dose (81-100 mg daily) universally recommended unless contraindicated.
Iron Management
•🚫 Avoid routine iron supplements—iron deficiency helps maintain target Hct.
Cardiovascular Risk
•🫀 Strict control of BP, lipids, glucose; smoking cessation is critical.
Symptoms
•Pruritus: 🧴 SSRIs, antihistamines, or ruxolitinib.
•Splenomegaly: 🫛 Use cytoreduction or JAK inhibitors if symptomatic.
Monitoring
•📊 Regularly assess Hct, WBCs, and platelets.
•🔍 Watch for progression to post-PV myelofibrosis or acute leukemia.
Practical Tips
•High-risk: 🩸 Combine phlebotomy, aspirin, and cytoreduction.
•Low-risk: 🩸 Phlebotomy + aspirin (no cytoreduction unless needed).
•Pregnancy: 🤰 Use interferon-alpha; avoid phlebotomy >36 weeks.
Exam Hints
1️⃣ CYTO-PV trial: Emphasize Hct ≤45%.
2️⃣ Thrombosis prevention: Low-dose aspirin + phlebotomy.
3️⃣ Symptomatic pruritus: Think ruxolitinib if refractory.
4️⃣ Hydroxyurea toxicity: Cytopenias (monitor closely).
🔬 Latest updates: Follow NCCN 2024 & ELN guidelines for personalized management. 💉 #Hematology #MPN
Polycythemia, or erythrocytosis, is a blood disorder that occurs when the body has too many red blood cells. This causes the blood to thicken and become more difficult to flow, leading to serious health problems such as Blood clots and blood cancers.
#Polycythemia #erythrocytosis
Polycythemia Vera or Polycythemia—what’s the difference? 🤔
Our new article breaks it down so you can stay informed: https://t.co/d6DIgIenkM
#polycythemia #mpnsm #polycythemiavera #pvreporter
10/ 📢 STAY INFORMED: Polycythemia may not be as well-known, but understanding the symptoms and consulting a doctor for blood tests and diagnosis can help you catch it early!
Take care of your health! 💪
#HealthAwareness #Polycythemia #MedicalFacts
#Polycythemia
Polycythemia refers to an increase in red cell count, hematocrit, and usually hemoglobin. True polycythemia may be either primary or secondary.
Primary true polycythemia:
•Uncontrolled production of red cells by the bone marrow.
Clinical features:
•Look for plethoric features of the patient.
•Look for peripheral arterial pulsations.
•Note for limb swelling (venous thrombosis) or digital ulcerations (arterial thrombosis).
•Look for itching marks due to pruritus.
•Check the blood pressure for hypertension.
•Look for a swollen erythematous big toe (gout).
•Examine the abdomen for splenomegaly.
Question: What are the causes of secondary polycythemia?
Answer: Increased renal erythropoietin production either due to hypoxia or renal causes, e.g., renal causes or ectopic tumors.
Investigations:
•High red cell count, hemoglobin, hematocrit, whole blood viscosity, and uric acid.
#MEDHM @IhabFathiSulima #MedEd
#MedX
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