Top Tweets for #Proinflammatory
Activation of MyD88/IRAK1 axis and downstream #proinflammatory signaling in healthy adult and neonatal African American skin
Trubetskoy et al
https://t.co/QUTmPQCdpS
#dermatology #dermscience #derm #dermatologia #dermresearch #dermscience
#BE can occur in #postmenopausal in absence of #exogenousestrogen or #obesity.
The #livercirrhosis associated with impaired #estrogenmetabolism, #diabetes, characterized by #chronicinflammation, contribute to a #proinflammatory #microenvironment
https://t.co/pCEML4fbKK
🏆 Highly Cited!
Substrate stiffness modulates cardiac #fibroblast activation, #senescence, and #proinflammatory secretory phenotype
Marina B. Felisbino, et al.
💻https://t.co/X0hfHMD42M
#AJPHeartCirc #Hydrogel
New insights into #Hpylori’s virulence machinery🕵️🧪 Josenhans & coll. unravel new interactions between CagT4SS surface proteins and outer membrane proteins HopQ & HopZ—key for host interaction and #proinflammatory signaling.🦠 @LMU_Muenchen
👉 https://t.co/fRgdgASrSH
Article✍️Inhibition of #WAC alleviates the #chondrocyte #proinflammatory secretory phenotype and #cartilage #degradation via #H2BK120ub1 and #H3K27me3 #coregulation. From Dr. Li @SunYat-senUniversity;
https://t.co/qVExyinwOI.
The #methyltransferase #Set7 regulates #proinflammatory gene expression through #H3K4me1 in #diabetic #endothelial cells. Could Set7 be considered a target in diabetic #endothelial #dysfunction?🙏🫰🐁🔬
Inhibition of #Kdm7A and #UTX significantly reduces #leukocyte #adhesion, offering therapeutic potential in the inflammatory response? 🫰🐁🔬
#VEGF-A induces the phosphorylation and nuclear export of #HDAC7, which prevents the repression of VEGF-responsive genes and promotes endothelial cell #proliferation and #migration. Could HADAC7 be the "on-off" switch in #diabetic #wound healing? 🙏🙏🐁
@endothelialnews @The_MCSociety @IJMS_MDPI @NatRevCardiol @CNIC_CARDIO @JCVPOnline @DCAcademyDK @WashUCardiology @WashUDeptMed @hopkinsheart @IEEEembs @TheLancet @Hiraselab_KU @HaoYin20 @MetabolismAnd @NatureRevEndo @TheLancetEndo @FacMedicinaUC @CMPLPUCCHILE @LuisSobrevia2 @SocFisiolChile @physoc @isth @ATHjournal @society_eas 👇🧵👇🧵🧵🧵
In this review Tyler Bauer from the @BTBKGMD lab summarize advances in the understanding of the epigenetic regulation of cell phenotype in diabetic wounds
https://t.co/0KNNf1ntg4
💡#MostViewedPaper
📕Alveolar #Macrophages in Viral #Respiratory #Infections: Sentinels and Saboteurs of Lung Defense
🧑🏻🔬By Prof. Dr. Andreas Beineke, et al.
🔗https://t.co/5333yGIBuf
#innate #immune #lung #alveolar #macrophage #proinflammatory #reaction
The second member we'd like to introduce is Dr. Dake Qi, associate professor at @umanitoba College of Pharmacy. His research interests center around #proinflammatory factors and #insulinresistance, #diabetes, and other #metabolicdysfunctions @CHRIManitoba @UM_RadyFHS
Our first #ThrowbackThursday comes from @UVMDeptMed and @UVMLarnerMed! Bariatric surgery decreases the capacity of plasma from #obese asthmatic subjects to augment airway epithelial cell #proinflammatory cytokine production (Paola Peña-García et al.):
https://t.co/Tc2G0klaFw
Interesting study on hypoxia exposure prior to / after endotoxin administration and #proinflammatory cytokine response.
We recommend this article & podcast ➡️ Community-acquired bacterial coinfections & #COVID https://t.co/Mazk6uAVTZ
https://t.co/UtMC2VdKcV
📌Thrilled to share a new #review published in @Landmark_IMR Interleukin-31: The Inflammatory Cytokine Connecting Pruritus and Cancer
https://t.co/HxH04bNYNb
#proinflammatory #cytokine #Interleukin #cancer #CancerResearch #skin #atopicdermatitis #inflammation #CellBiology
CONCLUSION
In women with complications, mesh induces a #proinflammatory response that persists years after implantation. The increase in MMP-9 in mesh explants that were removed for exposure indicates #degradation; the positive association between #interleukin-10 and M2 #macrophages in mesh explants that are removed for #pain is consistent with #fibrosis.
Keywords: #cytokine, #inflammatory response, #macrophage #phenotype, #polypropylene #mesh
https://t.co/IucdTMt6Ot
#JournalofPharmaceuticalAnalysis study finds that #TraditionalChineseMedicine #PienTzeHuang ameliorates #sepsis by inhibiting the activation of #MAPK and #NFkB #signaling and reducing #proinflammatory #cytokine secretion: https://t.co/8rsO3htlnX
👉#Spatial #proteomics emphasized #macrophage & #exhausted CD8+T cells #hub in #immunotherapy responsive #kidneycancer patients with #neoantigen-specific #HLA!
👉Here, #preexhausted CD8+T cells established dense #interactions with #proinflammatory #macrophages & #Cancers!
6/10🧵
When considering #PhageTherapy, are there any direct effects of #phage on the human host? @jbombscientist &co show that therapeutic phages can be detected by human #RespiratoryTract epithelial cells & can elicit #proinflammatory responses. #PLOSBiology https://t.co/ANiM2olzFf
When considering #PhageTherapy, are there any direct effects of #phage on the human host? @jbombscientist &co show that therapeutic phages can be detected by human #RespiratoryTract epithelial cells & can elicit #proinflammatory responses. #PLOSBiology https://t.co/ANiM2ol1PH
When considering #PhageTherapy, are there any direct effects of #phage on the human host? @jbombscientist &co show that therapeutic phages can be detected by human #RespiratoryTract epithelial cells & can elicit #proinflammatory responses. #PLOSBiology https://t.co/ANiM2olzFf
#STAT3 variants can be loss or gain of function, causing very different diseases
This review from @JoeMackie6 et al. @Tangye_Lab explores how studying #IEI has informed our knowledge of this #proinflammatory transcription factor 👉https://t.co/Xj588iKHbl
#WorldPIWeek
#Autophagy maintains cellular homeostasis by selectively degrading cytoplasmic cargoes; defects can lead to #proinflammatory disease. This study shows that selective degradation of the #immunoproteasome regulates innate inflammatory signaling #PLOSBiology https://t.co/scmAC6RHbO
#Autophagy maintains cellular homeostasis by selectively degrading cytoplasmic cargoes; defects can lead to #proinflammatory disease. This study shows that selective degradation of the #immunoproteasome regulates innate inflammatory signaling #PLOSBiology https://t.co/scmAC6R9mg
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