IVERMECTIN: FULL DOSAGE SCHEDULE FOR CANCER & PREVENTION
1000s of people use Dr. William Makis MD’s IVERMECTIN dosing chart. Here’s a clear, categorized breakdown based on body weight (mg/kg per day).
LOW DOSE: ≤ 0.5 mg/kg/day
**Best for:**
- Cancers in remission
- Strong family history or genetic predisposition
- Prophylaxis (preventive)
**Side effects:** No long-term side effects reported.
**Example:** Dr. Tess Lawrie reported a Stage 3 ovarian cancer case treated with chemo + 12 mg ivermectin daily. Tumor marker CA125 dropped from 288 to 22 after 2 months and the tumor vanished.
MEDIUM DOSE: 1.0 mg/kg/day
**Best for:** Starting dose for **most cancers** (lung, pancreatic, renal cell, gastric, etc.).
**Side effects:** No long-term side effects reported.
**Example:** Dr. Shankara Chetty’s 70-year-old prostate cancer patient (PSA 89) took 45 mg/day (plus lactoferrin). After two months PSA fell to 10.9.
HIGH DOSE: 2.0 mg/kg/day
**Best for:** Very aggressive cancers (leukemia, pancreatic, brain cancers).
**Side effects:** No long-term side effects reported.
**Example:** Dr. Allan Landrito’s Stage 4 gallbladder cancer patient took 2 mg/kg daily for 14 months — cancer disappeared.
VERY HIGH DOSE: ≥ 2.5 mg/kg/day
**Best for:** Extensive metastatic disease, extremely poor prognosis, or certain brain cancers.
**Side effects:** Possible short-term & transient visual effects (usually resolve in a few days).
**Example:** Dr. Shankara Chetty treated a patient with 2.5 mg/kg/day — no side effects reported.
**Quick conversion example (for a 60 kg / 132 lb person):**
- Low: ≤30 mg/day
- Medium: 60 mg/day (≈5×12 mg tablets or 1 teaspoon liquid)
- High: 120 mg/day
- Very High: ≥150 mg/day
Many anecdotal reports exist of long-term daily use (months to over a year) with no serious toxicity, but individual responses vary.
Always work with a knowledgeable clinician, especially if you have pre-existing conditions (e.g., vision issues or glaucoma). This is for educational purposes only.
Share to spread awareness — information is power. 💊
🚨 Dr. William Makis MD – Cancer Protocol A powerful protocol using Ivermectin + Fenbendazole for remission, aggressive cancer, and poor prognosis.
📌 Ivermectin
– 0.5–2.5mg/kg/day based on cancer severity
– Proven results: prostate, ovarian, gallbladder, brain cancer
– No long-term side effects (even at high doses)
📌 Fenbendazole
– 222–1000mg/day (6 days/week)
– Add: CBD, Curcumin, Milk Thistle
– Supports liver/kidney health
🧪 Real case studies.
📷 Full dosage chart below
@Eppsorn Vi har fått bedre råd, må vite 🤪🤡. Jeg tror at ved å si at man sliter, så får mange kastet : «lånt over evne» , «feil prioriteringer» i trynet. Ved å erkjenne dårlig råd så blir man automatisk gjort skyldig i dårlige valg. Folket blir gaslightet.
@IngarSten@Goitericus For meg så går det på økte matvarepriser. Har kjøpt first price biff og kjøttdeig. Føler ikke at vi har vært så råflotte, men prisene har gått rett i været. På alt.
🚨RANDOMIZED TRIAL: High-Dose Vitamin C Cuts Death Risk from Stage IV Pancreatic Cancer by 54% and DOUBLES Survival Time
Adding 75g IV vitamin C to chemotherapy DOUBLED median survival compared with chemo alone — in one of the deadliest cancers known ⬇️
Patients were randomized 1:1 to receive either gemcitabine plus nab-paclitaxel alone (n=16) or the same chemotherapy regimen combined with pharmacologic IV vitamin C (75 g, three times weekly) (n=18).
The results were striking in patients with Stage IV pancreatic cancer who received high-dose IV vitamin C:
✅Median overall survival doubled
8.3 months (chemo alone) → 16 months (chemo + IV vitamin C)
✅54% reduction in risk of death
Hazard ratio: 0.46
✅Progression-free survival improved
3.9 months → 6.2 months
✅Higher objective response rate
23% → 38%
✅Lower rates of serious adverse events overall
✅Lower rates of severe hematologic toxicity (e.g., neutropenia)
✅Pharmacologic plasma vitamin C levels achieved (~500-fold increase)
Both groups received identical chemotherapy backbones, meaning the survival difference is attributable to the addition of vitamin C.
In a disease where median survival has historically hovered around 8–11 months, doubling survival warrants serious attention and larger confirmatory trials.
So I endured two years of daily attacks, smears and abuse 😃
and look at this:
"National Cancer Institute Launches Preclinical Study on Ivermectin as Potential Cancer Treatment"
Good to know I was two years ahead of my time! 😉
We're going to do HUMAN TRIALS in FLORIDA btw...
De vil ha oss til å tro at mektige politikere og medlemmer av kongefamilien ikke visste hva Epstein drev med.
Helt vanlige folk på internet og sosiale medium visste det, men folk med enorme nettverk og ressurser hadde ikke denne kunnskapen?
Spar meg.
RFK Jr: "People who take the flu shot are protected against that strain of flu, but they’re 4.4x more likely to get a non-flu infection… It injures your immune system so that you’re more likely to get a non-flu viral upper respiratory infection."
This is the final line! "If the gov't can make you inject something into your body that you don't want, then there is nothing the government cannot do to you. This is the final line." -@RobSchneider
Er dette tragikomisk?
Kommunene sier selv hva som er problemet:
De drukner i lover og regler.
Rektorer bruker mer tid på rapportering enn på elever.
Sykepleiere må bruke masse tid på ting som ikke har med å pleie pasienter.
Og kommunene mangler folk.
Likevel lager staten ca 1000 nye lover og forskrifter hvert år.
Kommunalministeren sa hun skulle se på det,
men antallet har vokst fra 13 000 til over 17 000 siden på få år!
Det virker som at regelverket løper helt løpsk… av seg selv?
🚨 DR WILLIAM MAKIS: “THE CANCER BREAKTHROUGH THEY DON’T WANT TO TALK ABOUT”
Oncologist Dr William Makis dropped a bombshell on The Shannon Joy Show — revealing what decades of cancer research have missed.
“Antiparasitics have a dozen mechanisms of action… ivermectin goes after cancer stem cells…
fenbendazole and mebendazole shut down glucose transporters — starving the tumor of fuel.”
According to Dr Makis, chemo alone kills only the rapidly dividing cells — leaving cancer stem cells alive to spread years later.
But add ivermectin, he says, and those hidden cells are targeted too — reversing resistance and triggering “tumor shrinkage oncologists have never seen.”
400+ studies now explore ivermectin’s potential role in cancer treatment — yet mainstream medicine remains silent.
Could cheap, repurposed antiparasitics threaten the trillion-dollar cancer industry?
Dr Makis thinks so.
And he’s not backing down.
@ShannonJoyRadio
How fenbendazole combat cancer cells.
Below is a concise overview of how fenbendazole combats cancer cells, grounded in available research.
Mechanisms of Fenbendazole Against Cancer Cells:
(1) Disruption of Microtubule Polymerization:
-Mechanism: Fenbendazole binds to β-tubulin, a protein component of microtubules, inhibiting their polymerization. Microtubules are essential for cell division (mitosis), intracellular transport, and maintaining cell structure.
-Impact: This disrupts the mitotic spindle, leading to mitotic arrest and apoptosis (programmed cell death) in rapidly dividing cancer cells. It mimics the action of microtubule-targeting chemotherapeutic agents like paclitaxel or vincristine.
(2) Induction of Apoptosis:
-Mechanism: Fenbendazole triggers apoptosis through multiple pathways:
p53 Activation: It upregulates the tumor suppressor protein p53, which promotes apoptosis in response to DNA damage or cellular stress.
-Mitochondrial Pathway: It increases pro-apoptotic proteins (e.g., Bax) and decreases anti-apoptotic proteins (e.g., Bcl-2), leading to mitochondrial membrane permeabilization and caspase activation.
Impact: This causes cancer cell death, particularly in cells with high proliferation rates.
(3) Inhibition of Glucose Metabolism:
-Mechanism: Fenbendazole reduces glucose uptake and inhibits glycolysis, a key energy source for cancer cells (the Warburg effect). It downregulates glucose transporters (e.g., GLUT1) and glycolytic enzymes like hexokinase II.
-Impact: By starving cancer cells of energy, fenbendazole impairs their growth and survival, especially in metabolically active tumors.
(4) Induction of Oxidative Stress:
-Mechanism: Fenbendazole increases reactive oxygen species (ROS) production in cancer cells by disrupting mitochondrial function or inhibiting antioxidant enzymes like glutathione peroxidase.
-Impact: Elevated ROS causes oxidative damage to DNA, proteins, and lipids, triggering apoptosis or necrosis. Cancer cells, with lower antioxidant capacity than normal cells, are particularly vulnerable.
(5) Inhibition of Cancer Stem Cells (CSCs):
-Mechanism: Fenbendazole targets CSCs, which are responsible for tumor initiation, metastasis, and therapy resistance. It inhibits stemness-related pathways (e.g., Wnt/β-catenin, Notch) and reduces CSC markers.
-Impact: This prevents tumor recurrence and metastasis by eliminating the self-renewing CSC population.
(6) Suppression of Tumor Angiogenesis and Metastasis:
-Mechanism: Fenbendazole inhibits vascular endothelial growth factor (VEGF) expression and other angiogenesis-related pathways, reducing blood vessel formation in tumors. It also downregulates matrix metalloproteinases (MMPs) involved in extracellular matrix degradation.
-Impact: This limits tumor nutrient supply and prevents cancer cell invasion and metastasis.
(7) Enhancement of Immune Response:
-Mechanism: Fenbendazole may modulate the tumor microenvironment by reducing immunosuppressive cells (e.g., regulatory T cells) and enhancing cytotoxic T-cell activity. It also induces immunogenic cell death (ICD) in some models, releasing damage-associated molecular patterns (DAMPs) that stimulate immune recognition.
-Impact: This boosts the body’s ability to target cancer cells, potentially enhancing immunotherapy efficacy.
(8) Synergy with Other Therapies:
-Mechanism: Fenbendazole enhances the efficacy of chemotherapy (e.g., gemcitabine, taxanes) and radiotherapy by sensitizing cancer cells to these treatments. It may inhibit multidrug resistance proteins (e.g., P-glycoprotein), allowing better drug retention in cancer cells.
-Impact: Combination therapies can reduce required doses of toxic chemotherapeutics, improving outcomes.
Repost and retweet so that patients can be aware of this information.
#Fenbendazole
#Cancercure
På samme pressekonferanse som Trump ba gravide være forsiktig med å ta paracet, ble det også pratet om denne behandlingen. Ikke en norsk avis nevnte dette. De snyter altså folk for gode nyheter for å kjøre paracetnyheten (som forøvrig er gammelt nytt fra før Trump ble president)
Pediatric neurologist Dr. Richard Frye says it’s been known for more than 10 years that generic drug leucovorin could be a treatment for autism
Thank you @SecKennedy@DrMakaryFDA for finally bringing attention to this 🙏
@ThrillaRilla369 Find out if its biological. Gluten can make you depressed, (even if you dont have celiac deseas ) lack og omega 3. Find out if you have pyroluria, if you do, sink and b6 in high doses cam change your life.