@nevin_simon @shereenmoloney1 @bramernic Tell me you have no idea what you're talking about wit hout telling me you have no idea what you're talking about..
@SwaledaleMutton@BreezerGalway@LaymansScience He's obviously wildly wrong. But even if he wasn't, that would only be ~7,000 fewer susceptible hosts (assuming they're still alive). And one hell of away to acquire immunity to SARS-CoV-2...
@SwaledaleMutton@ScepticalAussie For all the predictions they make, this really is in it's infancy.
Couldn't find this one, but I'm guessing it'll be similar to their DENV one:
https://t.co/uUZuVqihNC
@andrew_croxford Just thinking, hypothetically could you have tested for anti-spike IgG shortly after you first dose, earlier than you'd expect class switching to occur? Not sure if there's an IgM/IgG ratio to distinguish primary/secondary exposure for COVID
@SmallRedOne Yeah, a variant only has to be better than the other progeny of that cell and/or just luckier.
I do wonder whether there is reduced tolerance in Delta to further AA substitutions because of loss of receptor avidity though. Wouldn't like to bet on it
@livelovenofear@TakethatCt Incorrect (see below).
SARS-CoV-2's genome is ssRNA (for all all intensive purposes mRNA). It does not only encode the spike protein, and it replicates.
The vaccines are incredibly safe.
@SwaledaleMutton Wish I'd known about this instead of endlessly trawling through old entomology books looking for something that vaguely looked like what I could see under the microscope.
Though I suspect they would've eventually politely asked me to please stop asking them what insect this is
@InCytometry The discovery of prions was also awarded a nobel. Have they considered prions as a potential treatment? After all, they won a nobel prize, that's how much they rate prions.