Top Tweets for #LongC0VID

over 2 years ago

One of the most significant scientific research papers in history. The first to show the "uncanny similarity" of gene sequences in SARS-CoV-2 to HIV genes, evidence it was created in a lab. Prashant Pradhan et al.

janiesaysyay's tweet photo. One of the most significant scientific research papers in history.

The first to show the "uncanny similarity" of gene sequences in SARS-CoV-2 to HIV genes, evidence it was created in a lab.

Prashant Pradhan et al. https://t.co/tYB0AUVfp7

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@KUPERWASSERLAB @weldeiry @RetsefL @RobertKennedyJr @jsm2334 #IFeelLove #FlyingV #DontLeaveMeThisWay #Lowdown #LookingForMisterGoodbar #Stay #HIV #AIDS #C0VID #LongC0VID

1 day ago

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WereOnOurOwn @letsblamerussia

about 2 months ago

🔥COVID can switch on the same extrafollicular DN2 B-cell program that fuels lupus and flood the blood with autoantibodies.....😳 ➡️ #LongC0vid ➡️ #AvoidSars2 ➡️ #AvoidReinfections😷

Eric Topol

@EricTopol

about 2 months ago

Self-directed antibodies are found in people with #LongCovid. A key source of these was identified, and these autoantibodies and cell origin are also seen in lupus. @ImmunityCP https://t.co/ncwEG6VKNr

EricTopol's tweet photo. Self-directed antibodies are found in people with #LongCovid. A key source of these was identified, and these autoantibodies and cell origin are also seen in lupus. @ImmunityCP
https://t.co/ncwEG6VKNr https://t.co/rgcj6SMIO7

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Udbhavi @bhavibee

2 months ago

Last July, I took a break from journalism and decided to pursue a Master's degree. Over the past year, I've written academic essays and data-driven policy papers but nothing for the newspaper. Took me long enough. :) #LongC0VID #HealthPolicy @DeccanHerald https://t.co/LaOq6oTxkv

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2 months ago

@caitoz #LongC0VID

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Robin. ANTI-GENOCIDE. Voting NO to genocide. @robinsoped101

2 months ago

2026.... and #LongC0VID is big news! 😳 Sadly many of us already knew this back in 2020, thanks to @fitterhappierAJ MSM are now catching up, ok, better late than never!😷

Bloomberg

@business

2 months ago

The immune systems of people with long Covid may remain locked in a prolonged battle with remnants of the coronavirus, producing inflammation while failing to effectively eliminate its source, new research suggests. https://t.co/2WCbrAZ9EF

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2 months ago

The MAiD program in Canada is being marketed to the disabled and will be available to be marketed to the mentally ill in 2027. I know of many people with #LongC0VID and other PAISs like #ME who are also choosing euthenasia mainly due to lack of research, lack of medical care, inability to support themselves (can no longer work), medical abuse.

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2 months ago

Read👇, please read..... let it sink in....and than look and listen around! 🧠😬😷 #LongC0VID

PsyPost.org

@PsyPost

3 months ago

A systematic review of 49 imaging studies highlights how COVID-19 affects the human brain. Researchers found widespread structural and functional changes in areas responsible for memory, emotion, and attention, providing insight into enduring… https://t.co/MfYUJiTq72

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3 months ago

Long-term angiogenic and thromboinflammatory signatures in post-COVID-19 syndrome 🚨3.5 years after severe COVID: VEGF-D skyrockets, clotting stays disrupted, and two-thirds still have fatigue + brain fog. The vascular wound clearly didn’t resolve! #LongC0VID ➡️Another study, now from Mexico( small cohort, n=32), showing persistent vascular/thromboinflammatory abnormalities, now documented at approximately 3.5 years post-infection (hospitalized 2020, follow-up 2023–2024). ➡️Key findings: - Small cohort of 32 adults (median age 50, 75% male) hospitalized with severe/critical COVID-19 in 2020 at Mexico’s National Institute of Respiratory Diseases, - Paired plasma samples from acute hospitalization vs. ~3.5-year follow-up (2023–2024), - Persistent symptoms with high prevalently: fatigue 65.6%, dyspnoea 62.5%, concentration difficulties 65.6%, - Angiogenic markers: VEGF-D rose sharply (median 10.4 → 75.2 pg/mL, p<0.001), angiopoietin-1 trended upward. Soluble P-selectin fell, - Thromboinflammatory changes: alpha-2-macroglobulin increased (38.7 → 86.8 mg/dL, p<0.001). Prothrombin time remained abnormal in a subset, - Strong correlations linked VEGF-D/VEGF-R2 to coagulation parameters and dyspnoea. ‼️So, according to this study, severe COVID-19 leaves a long-lasting vascular scar. Nearly four years later, survivors still show active angiogenic dysregulation and coagulation imbalance that track with ongoing fatigue, breathlessness, and cognitive problems. The infection’s endothelial and thromboinflammatory damage does not simply resolve. ‼️Note, when compared with the science at hand today, we need to add this: Endothelial/thromboinflammatory changes persist months–years, indeed and worse after severe illness but careful, this also can happen after mild cases. Reinfections can further accumulate damage, with subsequent CV risk increases. It’s not all bad news, trajectories vary, patients can improve, but some retain clear lasting effects. #AvoidSars2 #AvoidReinfections https://t.co/uauU7wkOAu

HarrySpoelstra's tweet photo. Long-term angiogenic and thromboinflammatory signatures in post-COVID-19 syndrome

🚨3.5 years after severe COVID: VEGF-D skyrockets, clotting stays disrupted, and two-thirds still have fatigue + brain fog. The vascular wound clearly didn’t resolve! #LongC0VID

➡️Another study, now from Mexico( small cohort, n=32), showing persistent vascular/thromboinflammatory abnormalities, now documented at approximately 3.5 years post-infection (hospitalized 2020, follow-up 2023–2024).

➡️Key findings:
- Small cohort of 32 adults (median age 50, 75% male) hospitalized with severe/critical COVID-19 in 2020 at Mexico’s National Institute of Respiratory Diseases,

- Paired plasma samples from acute hospitalization vs. ~3.5-year follow-up (2023–2024),

- Persistent symptoms with high prevalently: fatigue 65.6%, dyspnoea 62.5%, concentration difficulties 65.6%,

- Angiogenic markers: VEGF-D rose sharply (median 10.4 → 75.2 pg/mL, p<0.001), angiopoietin-1 trended upward. Soluble P-selectin fell,

- Thromboinflammatory changes: alpha-2-macroglobulin increased (38.7 → 86.8 mg/dL, p<0.001). Prothrombin time remained abnormal in a subset,

- Strong correlations linked VEGF-D/VEGF-R2 to coagulation parameters and dyspnoea.

‼️So, according to this study, severe COVID-19 leaves a long-lasting vascular scar. Nearly four years later, survivors still show active angiogenic dysregulation and coagulation imbalance that track with ongoing fatigue, breathlessness, and cognitive problems. The infection’s endothelial and thromboinflammatory damage does not simply resolve.

‼️Note, when compared with the science at hand today, we need to add this:
Endothelial/thromboinflammatory changes persist months–years, indeed and worse after severe illness but careful, this also can happen after mild cases. Reinfections can further accumulate damage, with subsequent CV risk increases. It’s not all bad news, trajectories vary, patients can improve, but some retain clear lasting effects.
#AvoidSars2 #AvoidReinfections
https://t.co/uauU7wkOAu

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5 months ago

😡“Children and young people (CYP) with PCC persisting for 3.5-years post-infection experience multiple symptoms of wide-ranging severity and disruption to daily life, education and social participation.” #Tiredness #LongC0vid #AvoidReinfections https://t.co/IrZ58wcHrF

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7 months ago

🚨Pre-existing endocrine, metabolic, or gastrointestinal conditions, NOT ACUTE COVID-19 SEVERITY, dramatically determine the risk of severe, persistent neurological damage long after infection. #LongC0VID #AvoidSars2 https://t.co/dBMCVB2ujW

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7 months ago

Overview and Pathophysiology of Long COVID 🚨200–400 MILLION people worldwide are crippled by Long COVID. That's not a 'mild' virus aftermath, it's multi-organ destruction that persists for years. Wake up. #LongC0vid ➡️Authored by @elisaperego78 , a Long COVID patient-researcher and advocate (with lived experience of chronic illness), it brings authenticity and depth rarely found in traditional academic reviews, blending rigorous synthesis with real-world urgency.💪👏 ➡️Summary: 1. Long COVID affects an estimated 200–409 million people globally, with pooled prevalence around 36% across studies. Risks persist across all ages, even in mild/asymptomatic or vaccinated/Omicron cases, though attenuated by vaccination, 2. It is a heterogeneous, multi-system condition involving dozens of symptoms (e.g, fatigue, brain fog, dyspnea, pain) that evolve over time, often relapsing, with potential for subclinical damage, disability, and increased mortality, 3. Major pathophysiological mechanisms include viral persistence in tissues, immune dysregulation (e.g, lymphopenia, T-cell exhaustion, autoantibodies, complement issues, mast cell activation), autoimmunity, endothelial dysfunction, micro/macro-thrombosis (including fibrinolysis-resistant microclots), chronic inflammation, microbiome dysbiosis, and reactivation of latent pathogens, 4. Organ-specific involvement is widespread: cardiovascular/endothelial (e.g, vasculopathy, accelerated aging, perfusion defects), heart (myocarditis, arrhythmias, ischemia), lungs (fibrosis, thrombosis, perfusion abnormalities), CNS (neuroinflammation, Gray matter loss, BBB disruption), PNS (neuropathy, dysautonomia/POTS-like), GI (dysbiosis, barrier impairment), hepatobiliary/pancreas (injury, new-onset diabetes), kidney (progression to CKD, thrombotic microangiopathy), 5. Evidence draws from imaging (e.g, CMR showing up to 78% cardiac involvement post-mild infection), histology/autopsy (viral presence, thrombi, NETs), and large meta-analyses (e.g, 97 million people showing elevated autoimmune disease risk), 6. Challenges include heterogeneous case definitions (WHO, NICE, etc.), limited biomarker access, surveillance gaps post-2022, and reinfection contributions. ➡️‼️In short, this isn't just another review, it's a patient-powered wake-up call exposing Long COVID as one of the most complex, widespread, and under-addressed biological crises of our era. ‼️So, Long COVID represents a profound, enduring public health crisis driven by persistent viral and immune-mediated multi-organ destruction, with no resolution in sight without urgent, scaled-up research and intervention. #WAKEUP #AvoidSars2 #AvoidReinfections

HarrySpoelstra's tweet photo. Overview and Pathophysiology of Long COVID

🚨200–400 MILLION people worldwide are crippled by Long COVID. That's not a 'mild' virus aftermath, it's multi-organ destruction that persists for years. Wake up. #LongC0vid

➡️Authored by @elisaperego78 , a Long COVID patient-researcher and advocate (with lived experience of chronic illness), it brings authenticity and depth rarely found in traditional academic reviews, blending rigorous synthesis with real-world urgency.💪👏

➡️Summary:
1. Long COVID affects an estimated 200–409 million people globally, with pooled prevalence around 36% across studies. Risks persist across all ages, even in mild/asymptomatic or vaccinated/Omicron cases, though attenuated by vaccination,

2. It is a heterogeneous, multi-system condition involving dozens of symptoms (e.g, fatigue, brain fog, dyspnea, pain) that evolve over time, often relapsing, with potential for subclinical damage, disability, and increased mortality,

3. Major pathophysiological mechanisms include viral persistence in tissues, immune dysregulation (e.g, lymphopenia, T-cell exhaustion, autoantibodies, complement issues, mast cell activation), autoimmunity, endothelial dysfunction, micro/macro-thrombosis (including fibrinolysis-resistant microclots), chronic inflammation, microbiome dysbiosis, and reactivation of latent pathogens,

4. Organ-specific involvement is widespread: cardiovascular/endothelial (e.g, vasculopathy, accelerated aging, perfusion defects), heart (myocarditis, arrhythmias, ischemia), lungs (fibrosis, thrombosis, perfusion abnormalities), CNS (neuroinflammation, Gray matter loss, BBB disruption), PNS (neuropathy, dysautonomia/POTS-like), GI (dysbiosis, barrier impairment), hepatobiliary/pancreas (injury, new-onset diabetes), kidney (progression to CKD, thrombotic microangiopathy),

5. Evidence draws from imaging (e.g, CMR showing up to 78% cardiac involvement post-mild infection), histology/autopsy (viral presence, thrombi, NETs), and large meta-analyses (e.g, 97 million people showing elevated autoimmune disease risk),

6. Challenges include heterogeneous case definitions (WHO, NICE, etc.), limited biomarker access, surveillance gaps post-2022, and reinfection contributions.

➡️‼️In short, this isn't just another review, it's a patient-powered wake-up call exposing Long COVID as one of the most complex, widespread, and under-addressed biological crises of our era.

‼️So, Long COVID represents a profound, enduring public health crisis driven by persistent viral and immune-mediated multi-organ destruction, with no resolution in sight without urgent, scaled-up research and intervention. #WAKEUP #AvoidSars2 #AvoidReinfections

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9 months ago

Fascinating study explanation 🧵👇 "The virus may die. But its fragments can still act. Sometimes fast. Sometimes slow." ➡️ Sit down, read and 🤔! #LongC0vid

Zdenek Vrozina

@ZdenekVrozina

9 months ago

What we once described as toxin-like persistence is now better understood, hypothetically, as a peptide-driven, shape-dependent membrane vulnerability - with nanostructures acting as storage, not effectors. Yes - hypothesis - but now with a mechanism.🧵

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Long Covid Advocacy 💙

@LongCovidAdvoc

over 1 year ago

📒A must read by @JessieLChaffee Haunting, vivid & poignant. It weaves in the: ☀️symbolism of Bradbury's 'All Summer in a Day' 📚the setting of Greenwich Village & the prison turned library ✊activism 🕳️quiet violence of absence we experience from #LongC0vid 👉 in @lithub

LongCovidAdvoc's tweet photo. 📒A must read by @JessieLChaffee

Haunting, vivid & poignant.
It weaves in the:
☀️symbolism of Bradbury's 'All Summer in a Day'
📚the setting of Greenwich Village & the prison turned library
✊activism
🕳️quiet violence of absence we experience from #LongC0vid

👉 in @lithub https://t.co/S08vb8SK2g

Jessie Chaffee

@JessieLChaffee

over 1 year ago

Very grateful to have my essay about living with #longcovid, and the many missings it has wrought, up on @lithub. 🧵 https://t.co/hA82lQ2hpj… via @lithub

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Long Covid Advocacy 💙

@LongCovidAdvoc

over 1 year ago

@AshleyDalton_MP There's no treatment for #LongC0vid Prevalence rates are not going down The only way is prevention Listen to CATA 💙🙏

David Osborn @SafeDavid3

over 1 year ago

💥 💥💥💥💥💥 CATA has written to the Minister, respectfully advising her that, as per her predecessor (Gwynne), she’s been badly advised. One can only speculate whether by DHSC, UK-HSA or the NHS/IPC “droplet, not airborne” dogmatists. https://t.co/uGPDdpPPQZ 💥💥💥💥💥💥 1/11

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PwME 4 bioMEdical research @ValeBodi

over 1 year ago

@ssayssayssay don't leave me this way > #C0VID #LongC0VID #HIV #AIDS https://t.co/sOlYEWzYMX

PolyBio

@polybioRF

over 1 year ago

8/ Drawing on what scientists have learned from other viral infections such as #HIV, hepatitis C, and #cancer clinical trials, the report outlines several promising approaches to treat long COVID patients with persistent SARS-CoV-2:

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almost 2 years ago

The FUNCAP free iph app Version 1.1 now available on App Store FUNctional CAPacity - a clinical & research questionnaire for #MECFS & #LongC0VID Score your own capacity Create a PDF report to save or share Optionally include heart and movement metrics https://t.co/j9jhTJ7ESM

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Protect Our Province BC @Protect_BC

almost 2 years ago

“bizarre complaints that nobody can fix where no actual treatment was given. The patients are demanding treatments for conditions that nobody can give them the diagnosis for.” #LongC0VID

Paul Winston MD - Restoring Movement is the goal.

@drpaulwinston

almost 2 years ago

Don’t worry I’m also a big fan of yours, but doctors have no protection right now. Complaints to hospitals and the college are skyrocketing because people are so angry about lack of access to care. Dr. are going to work scared because the next patient is going to report them to the college for spurious reasons. Hundreds of hours at hospitals taken up by bizarre complaints that nobody can fix where no actual treatment was given. The patients are demanding treatments for conditions that nobody can give them the diagnosis for. Imagine spending 1000 hours because people don’t want to give you surgery.

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