🤍

[🤍 @hellolily0]

@kennyhealth444 Happy birthday!💕

[🤍 @hellolily0]

@bryan_johnson @_katetolo You guys should look into progesterone therapy for endo. Abnormal tissue growth is caused by high estrogen, it isn’t a mystery. Wishing her health and healing. It really seems like you like to embarrass her :(

[the heart @Fredward3948576]

It has been a privilege and an honor to giggle on the phone with you

[Alannah @alannah00]

Cancer cells are normal cells. There’s nothing uniquely evil about them. They are just adapting to oxygen interruption.
Justice for innocent cancer cells🛡️

[Master Metabolism @lowmegatron]

CO₂ gas absorbed through the skin kills cancer cells and shrinks tumors.

The research was carried out in mice with three types of human tumors: breast cancer, osteosarcoma, and malignant fibrous histiocytoma, also known as undifferentiated pleomorphic sarcoma.

The study found that CO₂ treatment caused significant cancer cell death when it was applied for 10 minutes or longer. Tumor size was also significantly reduced when treatment lasted more than 5 minutes.

CO₂ was applied by putting the mice, minus the head, in a CO₂-filled bag. In earlier research, the researchers found that applying CO₂ through the skin via a hydrogel reduced the size of several tumor types. It also caused cancer cells to die through a natural cell-death process called apoptosis.

The researchers tried different ways of application and, overall, the best treatment conditions were:

— Applying CO₂ for more than 10 minutes
— Treating twice per week
— Leaving 3 or 4 days between treatments
— Applying the treatment directly over the tumor site

“The results suggest that this novel transcutaneous CO₂ application might be useful to treat primary tumors, while mitigating some side effects, and therefore could be safe for clinical trials.”

Ref: Optimization of antitumor treatment conditions for transcutaneous CO₂ application: An in vivo study

Be very careful not to submerge your head in CO₂. This can be fatal. There is also one case where someone accidentally filled up the room with CO₂ as the inflow tube leaked. [PMID: 34394794]

[🤍 @hellolily0]

@gentlegossip Do it! ❤️

[Iroquois Pliskin ☦︎ @pliskin1roquois]

https://t.co/CpMZzK5EnU

[🤍 @hellolily0]

@BioavailableNd 👑👑👑

[Metabolic M @Metabolicmonstr]

When proteins are under the influence of CO2 they form molecular bonds, called Carbamates, which changes the protein's function.
If CO2 doesn't isn't available, less benign things will stick to the amino group, like PUFAs or free radicals.
In the literature this is mostly researched for hemoglobin, but it happens with every neurotransmitter, hormone... every protein in the body.

"Neurotransmitter aralkylamines, such as phenethylamine, tyramine, dopamine, tryptamine, serotonin, and histamine, absorb aerial CO2..."
(PMID: 40993071)

There's even a study showing that carbamates are a major metabolite in (bull) sperm. And bulls that were more fertile had higher levels of carbamates.
(PMID: 31533629)

[🤍 @hellolily0]

@TakeThiamine

[🤍 @hellolily0]

I'm learning that the secret to a healthy, energetic life is to develop no self defense mechanisms. just feel everything. Children with high energy naturally have no self-defense mechanisms & emotional armor. as we age, we start bracing ourselves, but self-protection always comes at an energetic cost.

It's better to be open & fully take the pain, lest we calcify and close ourselves off from life.

high atp = relaxed
low atp = stiff, tense. don't waste your precious energy guarding yourself. rigor mortis of the soul

[Ling’s Cell Physiology @aihtheory]

“The organism is always trying to come back to order.”

[🤍 @hellolily0]

@halsonissen He actually gets that a lot haha

[Ruth @Know_Yourself_H]

"If you grind up a computer and dissolve it in acid, you can find out exactly what substances it was made of, but you won't learn from that information how the computer worked." Ray on the genetic paradigm of cancer.

[Master Metabolism @lowmegatron]

Healing depends on thyroid hormone.

In people with cancer of the voice box, treating radiation-induced hypothyroidism lowers the need for follow-up surgery by about 70%.

University of Michigan researchers found that low thyroid hormone levels dramatically increase the risk of wound-healing complications after total laryngectomy in patients with laryngeal (voice box) cancer who previously received radiation.

In the retrospective study of 182 patients, hypothyroid patients had nearly 4 times higher risk of fistula formation, with risk rising as hypothyroidism worsened.

Fistulas occur in 40–50% of these surgeries and prolong hospital stays while delaying speech and swallowing.

“What they found was that hypothyroid patients, or those with low thyroid function, had nearly four times the risk of fistula formation than those who had normal thyroid function. As the level of hypothyroidism increased, so did the corresponding risk of fistula formation and the need for another operation.

This study is the first to show the role of hypothyroidism as a predictor for these types of wound complications.

"This was our big aha moment," says senior author and otolaryngologist Matthew Spector, M.D. "The applicability is pretty obvious. You can give a thyroid hormone supplement to patients and it may decrease their rate of fistula and improve their wound healing in the postoperative period. And that adds less hospitalization, better utilization of hospital costs. Everything would get better from this."

A follow-up trial found that fistula formation halved and the need for further surgery dropped by about 70%.

Given that they used a sup-optimal solution (levothyroxine), it’s likely these figures could improve significantly when patients are given appropriate doses of T3 alongside levothyroxine.

“Postoperative intravenous levothyroxine supplementation reduced rates of acute hypothyroidism, fistula formation, and fistula requiring reoperation in patients undergoing salvage total laryngectomy without adverse effects. Intravenous levothyroxine is a viable strategy to reduce wound complications in this high-risk patient population.”

“... the hypothyroid patient commonly suffered from one or many of such symptoms as weakness, itching, constipation, somnolence, muscular pains, elevated blood fat levels, susceptibility to infections, poor healing of wounds, premature atherosclerosis, and gangrene.”

— Broda Barnes

Ref:
Treating hypothyroidism to stop a stubborn surgical complication
Hypothyroidism and Wound Healing After Salvage Laryngectomy
Phase II Clinical Trial of Intravenous Levothyroxine to Mitigate Pharyngocutaneous Fistula in Euthyroid Patients Undergoing Salvage Laryngectomy

[🤍 @hellolily0]

@alannah00 Omg❤️❤️ily

[Metabolic M @Metabolicmonstr]

@hellolily0 https://t.co/FhrTD9QONx

[🤍 @hellolily0]

@MoreOrLessAMess This is so insane. She’s trusting her maternal instinct & made her daughter feel safe. Very bizarre and predatory behavior to want to separate parents from their kids. Never leave your child alone with a medical provider under any circumstance

[🤍 @hellolily0]

@pliskin1roquois Wow incredible! Wonder if he was sedated on 1000mg

[🤍 @hellolily0]

Vitamin D and progesterone are functionally interdependent. A deficiency in either can reduce the benefits of the other:

“Vitamin D3 increased the expression of membrane progesterone receptor α on CD4+ T-cell. Vitamin D3 increased the expression of membrane progesterone receptor β on CD4+ T-cell. Vitamin D3 level may affect the optimum progesterone immune regulatory activity” (PMID: 30959372)

“Recently, we discovered that the vitamin D receptor (VDR) is induced in T cells by progesterone. This finding connects the function of progesterone to that of vit. D and suggests that the two steroid hormones cooperate with each other for sequential and effective regulation of the immune system” (PMID: 25826095).

"Progesterone is a novel inducer of vit.D receptor (VDR) in T cells and makes T cells highly sensitive to calcitriol. At the molecular level, the induction by progesterone is mediated by two progesterone receptor binding elements in the intron region after the first non-coding exon of the human VDR gene. Increased expression of VDR by progesterone allows highly sensitive regulation of T cells by vit.D even when vit.D levels are suboptimal" (PMID: 25548222).