🚀 Our new paper is out in Nature Communications!
We uncovered a mechanistic switch to boost myelination: deleting Gtf2i in myelinating glia enhances Sox10/Mbp activity → thicker myelin, faster conduction, improved motor coordination.
🔗 https://t.co/ZSzns8K2vB
Excited to share my Ph.D. paper, now published at @NatureComms!
We uncovered a transcriptional role for Tfii-i in oligodendrocytes, its loss resulting in enhanced myelin thickness and signal conduction.
https://t.co/8Xwq7mIBhu
A coffee with a friend @barak_boaz58635 led to a great collaboration! ☕️🤝 We're excited to share our latest publication in @CellReports :
Cell-of-origin-specific behavioral deficits in oligodendrocyte-derived glioblastoma
https://t.co/LrPX9ecATX
@TelAvivUni@of_sagol
A beautifully written BBC piece on Williams syndrome, highlighting our lab’s work alongside others on the neural roots of sociability and developmental challenges. Grateful to see the science so clearly communicated.
https://t.co/KP1r8VCVfy
#WilliamsSyndrome, #myelin, #Social
🚨📢 Excited to announce our new paper in Science Advances! We’ve uncovered how Shank3 mutations affect myelination in an autism mouse model and human iPSC-derived OPCs. 🧠💡 Check it out!
https://t.co/0uVHAKHVJS
A neurobiological study reveals Gtf2i's pivotal roles in brain development and mediating neuronal mitochondrial function, validated by human brain samples derived from individuals with Williams syndrome.
@barakboaz@GiladLevy7
https://t.co/RpKjnKIHVn
I am looking for some advice/input/help with calcium imaging signals we record from mouse brain primary oligodendrocyte precursor cells. Anyone? Any good papers? Thanks!
#oligodendrocyte, #calciumimaging,
Happy to share our latest research article from the lab, focused on brain development abnormalities related to Williams syndrome, in collaboration with @YanivAssaf and @nshomron