PhD-student and lic. psych. investigating social learning and decision-making in addiction using behavioral experiments, clin. trials, and comp. modeling
Intressant disputation vid @karolinskainst den 13 juni!
@SimonJangard försvarar sin avhandling "Prosocial Learning and Decision-Making in Alcohol Use Disorder" kl. 13:00 i Franklinsalen, Tomtebodavägen 6a, Solna.
Huvudhandledare: @EmotionLabKI
Läs mer: https://t.co/VMe3wabch6
Excited to share our new paper on social reward learning in alcohol use disorder (AUD) in Translational Psychiatry! https://t.co/qbLh1PJgRA w/ @B_Lindstroem, Lotfi Khemiri, Nitya Jayaram-Lindström, & @EmotionLabKI.
Across two separate samples, we examined whether individuals with AUD show deficits in prosocial learning—learning to maximize rewards for others. Surprisingly, reward learning was intact in AUD and unrelated to reduced prosocial behavior.
Using computational modeling, we found comparable learning mechanisms in AUD and healthy controls, suggesting that social deficits in AUD are not driven by impaired reward learning.
These findings highlight the need to explore alternative mechanisms behind social dysfunction in AUD.
Evolutionary explanations for morality and committed relationships are a huge advance.
Are they useful for psychiatry?
I share my hopes and hesitations in this 30 min. video.
Thanks to @RealAdamHunt@RiadhAbed1@ISEMPH@HumBehEvoSoc
https://t.co/uKTaRmlnmu
Moral learning and decision-making across the lifespan https://t.co/JGnswEnuQ0 Excited to share our new preprint with @Connected_Mindz & @JC_Dreher. We review evidence from infancy to old age in light of 4 key aspects of morality within a value-based decision-making framework.
@Oliver_S_Curry@simianghost@PeterMonnerjahn@jchalupa_@DavidDeutschOxf@OxfordPopper Hmm, pragmatically speaking yes (and I agree with you here). But I think P. would argue that *all* theories are simplifications (quantification, causal structure, models etc.) to help us understand the *real* world, and in being so, they could never be true in an ultimate sense.
@DevinGoure@Athens_Stranger @JamesXrequiem Come on guys, isn't the debate of different ideas what X is all about these days, i.e. a live stream on your different intepretations of Nietzsche would sure be something valueable!
@elonmusk@imPenny2x Very surprising to me if true! I would anticipate that a regular training schedule would be more than essential to simultaneously steer all your companies. What’s your secret? :)
@Miriam_Sebold Glad to hear! Yes, that would be an interesting project! In fact, it is not only fairness but also altruism and trust: https://t.co/mCXTLhk2FI :)
@Miriam_Sebold Congrats, very nice findings! Would you anticipate that this stronger response to social exclusion in AD is related to previous findings on stronger reactions to unfairness in AD? (e.g. studies by Brevers et al: https://t.co/bD4lSI358J)
@OdorJohan @H_I_now@granvillejmath@SimonCervenka Tack Johan! Ja det var ett spännande projekt, inte minst på grund av att motivera och resonera kring ”noll-fynd”.
Excited to share a new paper on Striatal Dopamine D2 receptor availability as a predictor in the development of alcohol use disorder (AUD) in Addiction: https://t.co/6Rxnw66ktM w/ N. J-Lindström, @H_I_now, @granvillejmath, P. P-Sigray, J. Franck, J. Borg, and @SimonCervenka
Low striatal dopamine D2 receptor availability may not be a strong predictor in the development of alcohol use disorder
@simonjangard
https://t.co/BMzheTSvqH
The results are consistent with previous cross-sectional studies demonstrating a small effect of D2R on alcohol use indicating that prior D2R is not clinically useful to identify social drinkers at risk of later development of AUD.
In assessing PET data (n=67) in healthy individuals and subsequent alcohol use and related factors at a 8-16 year follow-up, we demonstrate no support for a large effect between D2R availability and later alcohol use nor for the majority of the alcohol-related factors.