@Dr_GuyRozen Thanks Guy. I wonder though, if there would still be an increased risk placing a second lead at more experienced centers or more experienced operators?
@narrowQRS@EM_RESUS@PPodrid@ecgrhythms Josh what would you do with slower atypical flutters? Say 150-200? Do you skip anticoagulant and ablate w/o tee then?
@AveragingBogey I had a patient w AV block from sarcoidosis. No antegrade conduction but slow retrograde conduction. During dual chamber pacing he’d spontaneously go into slow PMTs.
@javadm20@narrowQRS@rdschaller@SergioPinski@SchakrabartiEP If no effusion/he’s stable don’t do anything until noac is held for a few days. Get tte right away and ct if possible to delineate lead location better. If truly emergent could give kcentra/andaxanet.
@EJSMD Some very directed training: vasc surg mostly, plastics (a little) and I would’ve love to spend more time in the OR watching open hearts (not sure I would’ve needed to actually do much but learning anatomy and observing)
@StevenZweibel I’ve seen it- similar presentation (swelling/discharge on both sides). Skin testing using Abbott’s kit was negative for all components. We had used a tyrx pouch during both implants initially, so ended up doing an implant w/o the pouch and were successful.
@narrowQRS@Dr_Nazarian_EP@jeffrey_vinocur@5Stabbies That’s interesting Josh. What I don’t understand is why every low amplitude atrial EGM actually starts before the V? If it starts earlier why is it blanked ?