Save the date!
Happy to announce, the joint GfE/DSDB/SEBD Meeting „Development&Regeneration“ is coming to Potsdam with many exciting speakers and keynotes (Hans Clevers, Isabel Fariñas, Thomas Bosch, Frank Bradke):
https://t.co/fTUr5RsDx2
@GfEeV@IntSocDevBiol@SEBiolDev
Top-notch conference on basic and clinical aspects of vascular anomalies in February 2025 in Berlin. Just two more days to submit your abstract. Don't miss this opportunity to showcase your latest work: https://t.co/8Fji2UBU6l | @BIH_Innovation | @MDC_Berlin | @VasBioBerlin
Our latest out today @Nature!
We revisit a century-old theory and show that metabolic gradients work hand-in-hand with genetic&signalling instructions to shape the embryo. Glucose isn't just an energy source, it acts as a major conductor of the body plan🍬
https://t.co/mfx6Hk7nma
📢@MRSantosLab - LINE1 RNA prevents developmental reversion of human embryonic stem cells to the 8 cell-like state! + new insights on PRC2, nucleolus and chr. 19. Revised and expanded now @Dev_Cell, free access until Dec 4th (earlier version on bioRxiv):
https://t.co/tdVo5Gj2Xo
This Monday, FNRS presented Drs. @MiikkaVikkula & @LaurenceBoon1 with the Gagna & Van Heck International Prize for their contribution to the understanding of vascular anomalies, which has led to medical progress on an international scale. #sciencenews https://t.co/jN9o9Fblxe
Last day of this year's Kloster Seeon Angioscience meeting. Thanks to @PotenteLab & @cleaverlab for putting this so spectacularly together and fostering a great community spirit. Everybody else, please mark your calendar: The 14th KS meeting will be held on Sept. 20-23, 2026.
VitelloTag, a new approach developed at the MBL, resolves a major bottleneck to using the gene-editing tool CRISPR-Cas9 in many research organisms. https://t.co/PmypMOC0Mq
Why are so many inherited traits not explained by genome sequence variation? In my PhD work with @wbderry we describe a new mechanism of inheritance in animals that may account for this ‘missing heritability' 🧵https://t.co/K0tfW4PIoi
Our work on a therapeutic approach in the vascular disease CCM has just been published. Targeting the polycomb repressive complex 1 suppresses lesion formation in a murine CCM model: DOI: 10.1161/CIRCULATIONAHA.123.067438
@vascularbiology@EVB_Org@dzhk_germany@circulation